Lynx1 Prevents Long-Term Potentiation Blockade and Reduction of Neuromodulator Expression Caused by Aβ1-42 and JNK Activation

about

Lynx1 Prevents Long-Term Potentiation Blockade and Reduction of Neuromodulator Expression Caused by Aβ1-42 and JNK Activation

Item

http://www.wikidata.org/entity/Q58568989

description

wetenschappelijk artikel

@nl

name

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@en

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@nl

type

Item

label

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@en

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@nl

prefLabel

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@en

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@nl

P1476

Lynx1 Prevents Long-Term Poten ...... d by Aβ1-42 and JNK Activation

@en

P2093

E N Lyukmanova

http://www.w3.org/2001/XMLSchema#string

M A Shulepko

http://www.w3.org/2001/XMLSchema#string

M L Bychkov

http://www.w3.org/2001/XMLSchema#string

M P Kirpichnikov

http://www.w3.org/2001/XMLSchema#string

N A Vasilyeva

http://www.w3.org/2001/XMLSchema#string

P M Balaban

http://www.w3.org/2001/XMLSchema#string

P2860

Long-Term Potentiation and Memory

NMR Structure and Action on Nicotinic Acetylcholine Receptors of Water-soluble Domain of Human LYNX1

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

Nicotinic acetylcholine receptor signalling: roles in Alzheimer's disease and amyloid neuroprotection.

Abeta toxicity in Alzheimer's disease: globular oligomers (ADDLs) as new vaccine and drug targets.

Human-specific endogenous retroviral insert serves as an enhancer for the schizophrenia-linked gene PRODH.

Lynx1 and Aβ1-42 bind competitively to multiple nicotinic acetylcholine receptor subtypes.

Nicotinic regulation of CREB activation in hippocampal neurons by glutamatergic and nonglutamatergic pathways.

Inhibition of phosphorylation of JNK suppresses Aβ-induced ER stress and upregulates prosurvival mitochondrial proteins in rat hippocampus.

lynx1, an endogenous toxin-like modulator of nicotinic acetylcholine receptors in the mammalian CNS.

P304

57-61

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P698

30397527

http://www.w3.org/2001/XMLSchema#string

P932

6209405

http://www.w3.org/2001/XMLSchema#string