Combined Therapy with SS31 and Mitochondria Mitigates Myocardial Ischemia-Reperfusion Injury in Rats

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Combined Therapy with SS31 and Mitochondria Mitigates Myocardial Ischemia-Reperfusion Injury in Rats

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im September 2018 veröffentlichter wissenschaftlicher Artikel

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scientific article published on 15 September 2018

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2018

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name

Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Item

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Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Combined Therapy with SS31 and ...... mia-Reperfusion Injury in Rats

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Christopher Glenn Wallace

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Fan-Yen Lee

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Han-Tan Chai

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Hon-Kan Yip

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Hung-I Lu

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Jiunn-Jye Sheu

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Pei-Hsun Sung

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Pei-Lin Shao

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Sarah Chua

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Sheng-Ying Chung

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Rationale for establishing regional ST-elevation myocardial infarction receiving center (SRC) networks

The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin

Macrophage roles following myocardial infarction

Cardioprotective Effects of Mitochondria-Targeted Peptide SBT-20 in two Different Models of Rat Ischemia/Reperfusion.

Innate immune response after acute myocardial infarction and pharmacomodulatory action of tacrolimus in reducing infarct size and preserving myocardial integrity.

The inflammatory response in myocardial infarction.

Inhibition of dipeptidyl peptidase-IV enzyme activity protects against myocardial ischemia-reperfusion injury in rats.

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

Sirtuin 3-dependent mitochondrial dynamic improvements protect against acute kidney injury.

Adipose-derived mesenchymal stem cells embedded in platelet-rich fibrin scaffolds promote angiogenesis, preserve heart function, and reduce left ventricular remodeling in rat acute myocardial infarction.

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2782

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scholarly article

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10.3390/IJMS19092782

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English

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Kuan-Hung Chen

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2018-09-15T00:00:00Z

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30223594

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6164143

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