Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury - Wikidata - awgldk - TriplyDB

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

http://www.wikidata.org/entity/Q35012392

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Mitochondria-Targeted Antioxidants: Future Perspectives in Kidney Ischemia Reperfusion Injury Mitochondria: a new therapeutic target in chronic kidney disease Manganese neurotoxicity and the role of reactive oxygen species Darkness at the end of the tunnel: poststenotic kidney injury How the Innate Immune System Senses Trouble and Causes Trouble Mitochondrial oxidative stress in aging and healthspan Targeting Mitochondria and Reactive Oxygen Species-Driven Pathogenesis in Diabetic Nephropathy Targeting mitochondrial cardiolipin and the cytochrome c/cardiolipin complex to promote electron transport and optimize mitochondrial ATP synthesis First-in-class cardiolipin-protective compound as a therapeutic agent to restore mitochondrial bioenergetics Serendipity and the discovery of novel compounds that restore mitochondrial plasticity Disruption of cytochrome c heme coordination is responsible for mitochondrial injury during ischemia The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin Improved mitochondrial function underlies the protective effect of pirfenidone against tubulointerstitial fibrosis in 5/6 nephrectomized rats Role of reduced manganese superoxide dismutase in ischemia-reperfusion injury: a possible trigger for autophagy and mitochondrial biogenesis?Cellular and Molecular Mechanisms of AKI.Targeting Endogenous Repair Pathways after AKI.Formoterol restores mitochondrial and renal function after ischemia-reperfusion injury Mitochondrial dysfunction in inherited renal disease and acute kidney injury Reduction of early reperfusion injury with the mitochondria-targeting peptide bendavia Dietary supplementation with essential amino acids boosts the beneficial effects of rosuvastatin on mouse kidney In vivo multiphoton imaging of mitochondrial structure and function during acute kidney injury.Renal cortical hexokinase and pentose phosphate pathway activation through the EGFR/Akt signaling pathway in endotoxin-induced acute kidney injury.Mitochondrial protection restores renal function in swine atherosclerotic renovascular disease.Spironolactone rescues Dot1a-Af9-mediated repression of endothelin-1 and improves kidney injury in streptozotocin-induced diabetic rats Pharmacological targeting of GSK3β confers protection against podocytopathy and proteinuria by desensitizing mitochondrial permeability transition.Mitochondrial impairment in the five-sixth nephrectomy model of chronic renal failure: proteomic approach.Targeting CD36-mediated inflammation reduces acute brain injury in transient, but not permanent, ischemic stroke.Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathy Mitochondrial function and disturbances in the septic kidney Partial Support Ventilation and Mitochondrial-Targeted Antioxidants Protect against Ventilator-Induced Decreases in Diaphragm Muscle Protein Synthesis.A Mitochondrion-Targeted Antioxidant Ameliorates Isoflurane-Induced Cognitive Deficits in Aging Mice Folate receptor-targeted antioxidant therapy ameliorates renal ischemia-reperfusion injury.Meclizine Preconditioning Protects the Kidney Against Ischemia-Reperfusion Injury.Stimulation of carnitine palmitoyltransferase 1 improves renal function and attenuates tissue damage after ischemia/reperfusion.Reduction of ischemia/reperfusion injury with bendavia, a mitochondria-targeting cytoprotective Peptide.Endothelial pentraxin 3 contributes to murine ischemic acute kidney injury.Percutaneous revascularization for ischemic nephropathy: the past, present, and future.Chronic Therapy With Elamipretide (MTP-131), a Novel Mitochondria-Targeting Peptide, Improves Left Ventricular and Mitochondrial Function in Dogs With Advanced Heart Failure.Tolerance of the human kidney to isolated controlled ischemia.Mitochondria-targeted heme oxygenase-1 decreases oxidative stress in renal epithelial cells

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Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

http://www.wikidata.org/entity/Q35012392

description

2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

@ast

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

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type

label

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

@ast

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

@en

prefLabel

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

@ast

Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

@en

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Journal of the American Society of Nephrology

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Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury

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Clara Y Tow

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Dunli Wu

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Feng-Ying Cheng

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Hazel H Szeto

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Michael Ganger

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Shaoyi Liu

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Shaun F Darrah

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Surya V Seshan

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Yi Soong

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P2860

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

Energetic determinants of tyrosine phosphorylation of focal adhesion proteins during hypoxia/reoxygenation of kidney proximal tubules

Cyclosporin and organ specific toxicity: clinical aspects, pharmacogenetics and perspectives.

Perspectives in cell cycle regulation: lessons from an anoxic vertebrate.

Anaerobic and aerobic pathways for salvage of proximal tubules from hypoxia-induced mitochondrial injury.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Microvascular endothelial injury and dysfunction during ischemic acute renal failure.

Mechanisms of renal cell repair and regeneration after acute renal failure.

New concepts in reactive oxygen species and cardiovascular reperfusion physiology.

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1041-1052

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10.1681/ASN.2010080808

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6

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22

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51102141

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21546574

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