Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Histidine-Rich Oligopeptides To Lessen Copper-Mediated Amyloid-β Toxicity.Orchestration of dynamic copper navigation - new and missing pieces.

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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http://www.wikidata.org/entity/Q58863948

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im September 2015 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2015

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Neurotoxicity of Alzheimer's disease Aβ peptides is induced by small changes in the Aβ42 to Aβ40 ratio

Regulation of brain iron and copper homeostasis by brain barrier systems: implication in neurodegenerative diseases

In vitro peroxidase oxidation induces stable dimers of beta-amyloid (1-42) through dityrosine bridge formation

Modulation of tau phosphorylation by environmental copper.

Neurotoxic, redox-competent Alzheimer's beta-amyloid is released from lipid membrane by methionine oxidation.

Binding of zinc(II) and copper(II) to the full-length Alzheimer's amyloid-beta peptide.

The ZIP family of metal transporters.

Treatment with a copper-zinc chelator markedly and rapidly inhibits beta-amyloid accumulation in Alzheimer's disease transgenic mice.

Reevaluation of copper(I) affinity for amyloid-β peptides by competition with ferrozine--an unusual copper(I) indicator.

The amyloid-beta peptide of Alzheimer's disease binds Cu(I) in a linear bis-his coordination environment: insight into a possible neuroprotective mechanism for the amyloid-beta peptide

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092503

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scholarly article

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10.1063/1.4921314

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http://www.w3.org/2001/XMLSchema#string

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Patrick Gamez

Ana B Caballero

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2015-09-01T00:00:00Z

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neurotoxicity

Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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Copper in Alzheimer’s disease: Implications in amyloid aggregation and neurotoxicity

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P1476

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