Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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P1476

Loss of Smad7 Promotes Inflammation in Rheumatoid Arthritis

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P2093

Gengmin Zhou

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Hui-Yao Lan

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Jiyang Lv

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Meiying Wang

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Qing-Wen Wang

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Qingxia Qin

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Rong Mu

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Xiaolin Sun

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Yueming Cai

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P2860

Involvement of the circadian rhythm and inflammatory cytokines in the pathogenesis of rheumatoid arthritis

IL-17 family: cytokines, receptors and signaling

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis

Interleukin-17 family members and inflammation

Cytokines in the pathogenesis of rheumatoid arthritis

T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma

Ultrasound-microbubble-mediated gene transfer of inducible Smad7 blocks transforming growth factor-beta signaling and fibrosis in rat remnant kidney.

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2537

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scholarly article

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10.3389/FIMMU.2018.02537

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English

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P577

2018-01-01T00:00:00Z

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30450102

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P932

6224447

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