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Dietary Pectin Increases Intestinal Crypt Stem Cell Survival following Radiation InjuryNovel changes in NF-{kappa}B activity during progression and regression phases of hyperplasia: role of MEK, ERK, and p38.DCLK1 regulates pluripotency and angiogenic factors via microRNA-dependent mechanisms in pancreatic cancer.DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transitionFluvastatin interferes with hepatitis C virus replication via microtubule bundling and a doublecortin-like kinase-mediated mechanism.Differential effects of β-catenin and NF-κB interplay in the regulation of cell proliferation, inflammation and tumorigenesis in response to bacterial infectionDoublecortin-like kinase 1 is elevated serologically in pancreatic ductal adenocarcinoma and widely expressed on circulating tumor cellsDCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinomaAblation of Doublecortin-Like Kinase 1 in the Colonic Epithelium Exacerbates Dextran Sulfate Sodium-Induced Colitis.Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury.Inflammatory and oncogenic roles of a tumor stem cell marker doublecortin-like kinase (DCLK1) in virus-induced chronic liver diseasesEvidence of functional cross talk between the Notch and NF-κB pathways in nonneoplastic hyperproliferating colonic epithelium.Utility of a bacterial infection model to study epithelial-mesenchymal transition, mesenchymal-epithelial transition or tumorigenesis.Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells.Inhibition of Notch signaling reduces the number of surviving Dclk1+ reserve crypt epithelial stem cells following radiation injury.Small molecule kinase inhibitor LRRK2-IN-1 demonstrates potent activity against colorectal and pancreatic cancer through inhibition of doublecortin-like kinase 1.XMD8-92 inhibits pancreatic tumor xenograft growth via a DCLK1-dependent mechanism.Correction: Fluvastatin Interferes with Hepatitis C Virus Replication via Microtubule Bundling and a Doublecortin-like Kinase-Mediated MechanismDCLK1-isoform2 alternative splice variant promotes pancreatic tumor immunosuppressive M2-macrophage polarization
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-7587-9553
@en
name
Parthasarathy Chandrakesan
@ast
Parthasarathy Chandrakesan
@en
Parthasarathy Chandrakesan
@es
Parthasarathy Chandrakesan
@nl
type
label
Parthasarathy Chandrakesan
@ast
Parthasarathy Chandrakesan
@en
Parthasarathy Chandrakesan
@es
Parthasarathy Chandrakesan
@nl
prefLabel
Parthasarathy Chandrakesan
@ast
Parthasarathy Chandrakesan
@en
Parthasarathy Chandrakesan
@es
Parthasarathy Chandrakesan
@nl
P106
P31
P496
0000-0002-7587-9553