about
A DNA damage and stress inducible G protein-coupled receptor blocks cells in G2/MTumor microenvironment and neurofibromatosis type I: connecting the GAPsMice lacking the orphan G protein-coupled receptor G2A develop a late-onset autoimmune syndromeGenome-wide siRNA-based functional genomics of pigmentation identifies novel genes and pathways that impact melanogenesis in human cells.Identification of hair shaft progenitors that create a niche for hair pigmentation.Cells of origin in the embryonic nerve roots for NF1-associated plexiform neurofibromaDirect genetic demonstration of G alpha 13 coupling to the orphan G protein-coupled receptor G2A leading to RhoA-dependent actin rearrangement.Skin-derived precursors as a source of progenitors for cutaneous nerve regeneration.Cell of origin and microenvironment contribution for NF1-associated dermal neurofibromasBET bromodomain inhibition triggers apoptosis of NF1-associated malignant peripheral nerve sheath tumors through Bim inductionThe role of nerve microenvironment for neurofibroma development.Tumor segmentation of whole-body magnetic resonance imaging in neurofibromatosis type 1 patients: tumor burden correlates.Updated nomenclature for human and mouse neurofibromatosis type 1 genesClinical Value of Multiparametric Whole-Body Magnetic Resonance Imaging over Whole-Spine Magnetic Resonance Imaging in Patients with Neurofibromatosis Type I.Cutaneous involvement of pre-existing Rosai-Dorfman disease via post-herpetic isotopic response.Arsenical keratosis secondary to Fowler solution.Contributions of inflammation and tumor microenvironment to neurofibroma tumorigenesis.2016 Children's Tumor Foundation conference on neurofibromatosis type 1, neurofibromatosis type 2, and schwannomatosis.The biology of cutaneous neurofibromas: Consensus recommendations for setting research prioritiesSpatiotemporal loss of NF1 in Schwann cell lineage leads to different types of cutaneous neurofibroma susceptible to modification by the Hippo pathwayNF1 heterozygosity fosters de novo tumorigenesis but impairs malignant transformationCutaneous neurofibromas in the genomics era: current understanding and open questionsAn exophytic, bleeding nodule on the left malar regionCreating a comprehensive research strategy for cutaneous neurofibromasDiffusion-weighted imaging and diffusion tensor imaging as adjuncts to conventional MRI for the diagnosis and management of peripheral nerve sheath tumors: current perspectives and future directionsDouble-Stranded RNA Sensing Determines Epithelial Cell IdentityRobust surgical approach for cutaneous neurofibroma in neurofibromatosis type 1Overcoming BET Inhibitor Resistance in Malignant Peripheral Nerve Sheath TumorsHeterozygous Tumor Suppressor Microenvironment in Cancer DevelopmentTranslating current basic research into future therapies for neurofibromatosis type 1
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description
researcher ORCID ID = 0000-0003-2817-5382
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wetenschapper
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name
Lu Q Le
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Lu Q Le
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Lu Q Le
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type
label
Lu Q Le
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Lu Q Le
@en
Lu Q Le
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prefLabel
Lu Q Le
@ast
Lu Q Le
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Lu Q Le
@nl
P31
P496
0000-0003-2817-5382