about
Neuropeptides and their receptors: innovative science providing novel therapeutic targetsCalcitonin gene-related peptide: physiology and pathophysiologyAn ongoing role of α-calcitonin gene-related peptide as part of a protective network against hypertension, vascular hypertrophy, and oxidative stressEvidence for the pathophysiological relevance of TRPA1 receptors in the cardiovascular system in vivo.4-oxo-2-nonenal (4-ONE): evidence of transient receptor potential ankyrin 1-dependent and -independent nociceptive and vasoactive responses in vivo.The ability of neuropeptide Y to mediate responses in the murine cutaneous microvasculature: an analysis of the contribution of Y1 and Y2 receptors.Alarin is a vasoactive peptide.Environmental cold exposure increases blood flow and affects pain sensitivity in the knee joints of CFA-induced arthritic mice in a TRPA1-dependent manner.TRPA1 activation leads to neurogenic vasodilatation: involvement of reactive oxygen nitrogen species in addition to CGRP and NOThe sympathetic nervous system is controlled by transient receptor potential vanilloid 1 in the regulation of body temperature.Transient receptor potential canonical 5 (TRPC5) protects against pain and vascular inflammation in arthritis and joint inflammation.TRPA1 is essential for the vascular response to environmental cold exposure.Basal and activity-induced release of substance P from primary afferent fibres in NK1 receptor knockout mice: evidence for negative feedback.Tumour necrosis factor alpha mediates transient receptor potential vanilloid 1-dependent bilateral thermal hyperalgesia with distinct peripheral roles of interleukin-1beta, protein kinase C and cyclooxygenase-2 signalling.Hydrogen peroxide is a novel mediator of inflammatory hyperalgesia, acting via transient receptor potential vanilloid 1-dependent and independent mechanisms.Targeted disruption of the galanin gene attenuates inflammatory responses in murine skin.The role of perivascular adipose tissue-derived sensory nerves in influencing vascular regulation.Superoxide generation and leukocyte accumulation: key elements in the mediation of leukotriene B₄-induced itch by transient receptor potential ankyrin 1 and transient receptor potential vanilloid 1.Galanin-like peptides exert potent vasoactive functions in vivo.TRPV1 deletion enhances local inflammation and accelerates the onset of systemic inflammatory response syndrome.Anandamide produced by Ca(2+)-insensitive enzymes induces excitation in primary sensory neurons.Evidence that the modulatory effect of galanin on inflammatory edema formation is mediated by the galanin receptor 3 in the murine microvasculature.Correspondence: Challenging a proposed role for TRPC5 in aortic baroreceptor pressure-sensing.Association of Raynaud's phenomenon with a polymorphism in the NOS1 gene.
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description
Professor of Pharmacology at King's College London
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fysioloog
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name
Susan D Brain
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Susan D Brain
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Susan D Brain
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Susan D. Brain
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type
label
Susan D Brain
@ast
Susan D Brain
@es
Susan D Brain
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Susan D. Brain
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altLabel
Susan Brain
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prefLabel
Susan D Brain
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Susan D Brain
@es
Susan D Brain
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Susan D. Brain
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P244
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P1153
7005340169
P21
P214
P244
P31
P496
0000-0002-9684-8342
P735
P7859
lccn-n98803212