TRPA1 is essential for the vascular response to environmental cold exposure. - Wikidata - awgldk - TriplyDB

TRPA1 is essential for the vascular response to environmental cold exposure.

TRPA1 is essential for the vascular response to environmental cold exposure.

http://www.wikidata.org/entity/Q43092169

about

Hypoxia-induced sensitisation of TRPA1 in painful dysesthesia evoked by transient hindlimb ischemia/reperfusion in mice.Acute cardiopulmonary toxicity of inhaled aldehydes: role of TRPA1 TRPV1 dysfunction in cystinosis patients harboring the homozygous 57 kb deletion TRPA1 and TRPV1 contribute to propofol-mediated antagonism of U46619-induced constriction in murine coronary arteries.Environmental cold exposure increases blood flow and affects pain sensitivity in the knee joints of CFA-induced arthritic mice in a TRPA1-dependent manner.TRPA1 activation leads to neurogenic vasodilatation: involvement of reactive oxygen nitrogen species in addition to CGRP and NO Capsaicin-Sensitive Sensory Nerves Mediate the Cellular and Microvascular Effects of H2S via TRPA1 Receptor Activation and Neuropeptide Release.Development of an in vivo target-engagement biomarker for TRPA1 antagonists in humans.The sympathetic nervous system is controlled by transient receptor potential vanilloid 1 in the regulation of body temperature.Synergistic Effect of Ferulic Acid and Z-Ligustilide, Major Components of A. sinensis, on Regulating Cold-Sensing Protein TRPM8 and TPRA1 In Vitro.Transient receptor potential canonical 5 (TRPC5) protects against pain and vascular inflammation in arthritis and joint inflammation.Sensory Neuron-Specific Deletion of TRPA1 Results in Mechanical Cutaneous Sensory Deficits.Molecular sensors and modulators of thermoreception.Effects of β-estradiol on cold-sensitive receptor channel TRPM8 in ovariectomized rats.Cardamonin, a Novel Antagonist of hTRPA1 Cation Channel, Reveals Therapeutic Mechanism of Pathological Pain.Comprehensive thermal preference phenotyping in mice using a novel automated circular gradient assay.Calmodulin is responsible for Ca2+-dependent regulation of TRPA1 Channels A Novel α-Calcitonin Gene-Related Peptide Analogue Protects Against End-Organ Damage in Experimental Hypertension, Cardiac Hypertrophy, and Heart Failure.TRPA1 channels: molecular sentinels of cellular stress and tissue damage.Breast cancer induced nociceptor aberrant growth and collateral sensory axonal branching.Relevance of TRPA1 and TRPM8 channels as vascular sensors of cold in the cutaneous microvasculature.Ca2+-permeable mechanosensitive channels MCA1 and MCA2 mediate cold-induced cytosolic Ca2+ increase and cold tolerance in Arabidopsis.A historical perspective on the role of sensory nerves in neurogenic inflammation.Association of Raynaud's phenomenon with a polymorphism in the NOS1 gene.

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TRPA1 is essential for the vascular response to environmental cold exposure.

http://www.wikidata.org/entity/Q43092169

description

2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

@wuu

2014年学术文章

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2014年学术文章

@zh-cn

2014年学术文章

@zh-hans

2014年学术文章

@zh-my

2014年学术文章

@zh-sg

2014年學術文章

@yue

2014年學術文章

@zh-hant

name

TRPA1 is essential for the vascular response to environmental cold exposure.

@en

TRPA1 is essential for the vascular response to environmental cold exposure.

@nl

type

label

TRPA1 is essential for the vascular response to environmental cold exposure.

@en

TRPA1 is essential for the vascular response to environmental cold exposure.

@nl

prefLabel

TRPA1 is essential for the vascular response to environmental cold exposure.

@en

TRPA1 is essential for the vascular response to environmental cold exposure.

@nl

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Nature Communications

P1476

TRPA1 is essential for the vascular response to environmental cold exposure.

@en

P2093

Clive Gentry

http://www.w3.org/2001/XMLSchema#string

Jennifer V Bodkin

http://www.w3.org/2001/XMLSchema#string

Khadija M Alawi

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Rabea Graepel

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Richard Heads

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Salil Srivastava

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Xenia Kodji

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P2860

TRPA1 mediates formalin-induced pain

Pharmacological characterization and CNS effects of a novel highly selective alpha2C-adrenoceptor antagonist JP-1302.

Pharmacological profile of BIBN4096BS, the first selective small molecule CGRP antagonist

TRPA1 contributes to cold, mechanical, and chemical nociception but is not essential for hair-cell transduction

TRPA1 mediates the inflammatory actions of environmental irritants and proalgesic agents

The menthol receptor TRPM8 is the principal detector of environmental cold

ANKTM1, a TRP-like channel expressed in nociceptive neurons, is activated by cold temperatures

Impaired nociception and pain sensation in mice lacking the capsaicin receptor

TRPA1 receptor modulation attenuates bladder overactivity induced by spinal cord injury

TRPA1 contributes to cold hypersensitivity

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5732

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scholarly article

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10.1038/NCOMMS6732

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P478

5

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Aisah A. Aubdool

Elizabeth S Fernandes

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2014-12-11T00:00:00Z

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269574833

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25501034

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4284811

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