about
Multiscale modelling of vascular tumour growth in 3D: the roles of domain size and boundary conditionsCapillary Filling at the Microscale: Control of Fluid Front Using GeometryMathematical models of the VEGF receptor and its role in cancer therapy.Towards whole-organ modelling of tumour growth.Hybrid approaches for multiple-species stochastic reaction-diffusion modelsModelling aspects of cancer dynamics: a review.Accelerated geroncogenesis in hereditary breast-ovarian cancer syndromeGermline BRCA1 mutation reprograms breast epithelial cell metabolism towards mitochondrial-dependent biosynthesis: evidence for metformin-based "starvation" strategies in BRCA1 carriers.Mesoscopic and continuum modelling of angiogenesisSuppression of endogenous lipogenesis induces reversion of the malignant phenotype and normalized differentiation in breast cancer.Metabostemness: a new cancer hallmark.Gerometabolites: the pseudohypoxic aging side of cancer oncometabolites.Front microrheology of the non-Newtonian behaviour of blood: scaling theory of erythrocyte aggregation by aging.Oncometabolic Nuclear Reprogramming of Cancer Stemness.Robustness of differentiation cascades with symmetric stem cell division3D hybrid modelling of vascular network formation.Stochastic models of receptor oligomerization by bivalent ligand.Stochastic modelling of the eradication of the HIV-1 infection by stimulation of latently infected cells in patients under highly active anti-retroviral therapy.Stochastic multi-scale models of competition within heterogeneous cellular populations: Simulation methods and mean-field analysis.Stochastic modelling of viral blips in HIV-1-infected patients: effects of inhomogeneous density fluctuations.Senescence-Inflammatory Regulation of Reparative Cellular Reprogramming in Aging and Cancer.Quiescence: a mechanism for escaping the effects of drug on cell populations.Metabostemness: Metaboloepigenetic reprogramming of cancer stem-cell functions.Breast cancer dormancy can be maintained by small numbers of micrometastases.Comment on "Morphogenetic action through flux-limited spreading" by Verbeni, Sánchez, Mollica, Siegl-Cachedenier, Carleton, Guerrero, Ruiz i Altaba, and Soler.The effects of intrinsic noise on the behaviour of bistable cell regulatory systems under quasi-steady state conditions.Mathematical models of the fate of lymphoma B cells after antigen receptor ligation with specific antibodies.Coarse-graining and hybrid methods for efficient simulation of stochastic multi-scale models of tumour growth.A mathematical model of the effects of hypoxia on the cell-cycle of normal and cancer cells.Noise-induced bistability in the fate of cancer phenotypic quasispecies: a bit-strings approach.Evolutionary escape on complex genotype-phenotype networks.Surviving evolutionary escape on complex genotype-phenotype networks.From invasion to latency: intracellular noise and cell motility as key controls of the competition between resource-limited cellular populations.Stochastic quasi-steady state approximations for asymptotic solutions of the chemical master equation.Stability analysis of multi-compartment models for cell production systems.Epigenetic regulation of cell fate reprogramming in aging and disease: A predictive computational model.Angiogenesis and vascular remodelling in normal and cancerous tissues.Modelling cell growth and its modulation of the G1/S transition.A mathematical model of Doxorubicin treatment efficacy for non-Hodgkin's lymphoma: investigation of the current protocol through theoretical modelling results.Minimum Action Path Theory Reveals the Details of Stochastic Transitions Out of Oscillatory States
P50
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P50
description
onderzoeker
@nl
researcher
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ricercatore
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հետազոտող
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name
Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
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type
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Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
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Tomas Alarcon
@es
Tomas Alarcon
@nl
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