about
RAS signalling through PI3-Kinase controls cell migration via modulation of Reelin expressionThe GATA2 transcriptional network is requisite for RAS oncogene-driven non-small cell lung cancer.Determination of synthetic lethal interactions in KRAS oncogene-dependent cancer cells reveals novel therapeutic targeting strategies.Identification of TIGAR in the equilibrative nucleoside transporter 2-mediated response to fludarabine in chronic lymphocytic leukemia cells.Oncogenic RAS Signaling Promotes Tumor Immunoresistance by Stabilizing PD-L1 mRNA.Development of combination therapies to maximize the impact of KRAS-G12C inhibitors in lung cancerSHOC2 phosphatase-dependent RAF dimerization mediates resistance to MEK inhibition in RAS-mutant cancersRAC1P29S Induces a Mesenchymal Phenotypic Switch via Serum Response Factor to Promote Melanoma Development and Therapy Resistance
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Miriam Molina-Arcas
@ast
Miriam Molina-Arcas
@en
Miriam Molina-Arcas
@es
Miriam Molina-Arcas
@nl
type
label
Miriam Molina-Arcas
@ast
Miriam Molina-Arcas
@en
Miriam Molina-Arcas
@es
Miriam Molina-Arcas
@nl
prefLabel
Miriam Molina-Arcas
@ast
Miriam Molina-Arcas
@en
Miriam Molina-Arcas
@es
Miriam Molina-Arcas
@nl
P106
P31
P496
0000-0002-8176-8948