about
Role of mitochondria in apoptotic and necroptotic cell death in the developing brainA dual role for AMP-activated protein kinase (AMPK) during neonatal hypoxic-ischaemic brain injury in miceInvestigating the regulation of brain-specific kinases 1 and 2 by phosphorylationMind bomb-2 is an E3 ligase that ubiquitinates the N-methyl-D-aspartate receptor NR2B subunit in a phosphorylation-dependent manner.The regulation and function of mammalian AMPK-related kinases.Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) signaling and cell death in the immature central nervous system after hypoxia-ischemia and inflammation.Effect of Trp53 gene deficiency on brain injury after neonatal hypoxia-ischemiaMitochondria and perinatal brain injury.Mitochondria: hub of injury responses in the developing brain.Muscarinic receptor activation of AMP-activated protein kinase inhibits orexigenic neuropeptide mRNA expression.Mitochondria, Bioenergetics and Excitotoxicity: New Therapeutic Targets in Perinatal Brain InjuryCell Death in the Developing Brain after Hypoxia-IschemiaMolecular characterization of the AMP-activated protein kinase and its role in cellular metabolism.Oxidative stress and endoplasmic reticulum (ER) stress in the development of neonatal hypoxic-ischaemic brain injury.Spatial and temporal regulation of RACK1 function and N-methyl-D-aspartate receptor activity through WD40 motif-mediated dimerization.γδT cells but not αβT cells contribute to sepsis-induced white matter injury and motor abnormalities in mice.Mitochondrial dynamics, mitophagy and biogenesis in neonatal hypoxic-ischaemic brain injury.Magnesium induces preconditioning of the neonatal brain via profound mitochondrial protection.Voxel-wise comparisons of cellular microstructure and diffusion-MRI in mouse hippocampus using 3D Bridging of Optically-clear histology with Neuroimaging Data (3D-BOND).AMP-activated protein kinase (AMPK) is a tau kinase, activated in response to amyloid β-peptide exposure.Neuroprotective exendin-4 enhances hypothermia therapy in a model of hypoxic-ischaemic encephalopathyPost-mortem Characterisation of a Case With an ACTG1 Variant, Agenesis of the Corpus Callosum and Neuronal Heterotopia.Interneuron Development Is Disrupted in Preterm Brains With Diffuse White Matter Injury: Observations in Mouse and HumanLipopolysaccharide-induced alteration of mitochondrial morphology induces a metabolic shift in microglia modulating the inflammatory response in vitro and in vivo
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description
researcher
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wetenschapper
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name
C Thornton
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C Thornton
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type
label
C Thornton
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C Thornton
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prefLabel
C Thornton
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C Thornton
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0000-0001-7676-3272