about
Mechanism and consequence of the autoactivation of p38α mitogen-activated protein kinase promoted by TAB1Constitutive glycogen synthase kinase-3alpha/beta activity protects against chronic beta-adrenergic remodelling of the heart.Targeting of mannan-binding lectin-associated serine protease-2 confers protection from myocardial and gastrointestinal ischemia/reperfusion injury.Myocardial stress remodelling after regional infarction is independent of glycogen synthase kinase-3 inactivation.Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension.Cardiac myosin-binding protein C: a potential early biomarker of myocardial injury.Patent foramen ovale and scuba diving: a practical guide for physicians on when to refer for screeningAdvancements in pressure-volume catheter technology - stress remodelling after infarction.A chemical genetic approach reveals that p38alpha MAPK activation by diphosphorylation aggravates myocardial infarction and is prevented by the direct binding of SB203580.Moving in extreme environments: inert gas narcosis and underwater activities.Loss of Protein Kinase Novel 1 (PKN1) is associated with mild systolic and diastolic contractile dysfunction, increased phospholamban Thr17 phosphorylation, and exacerbated ischaemia-reperfusion injury.A Novel α-Calcitonin Gene-Related Peptide Analogue Protects Against End-Organ Damage in Experimental Hypertension, Cardiac Hypertrophy, and Heart Failure.Epistatic rescue of Nkx2.5 adult cardiac conduction disease phenotypes by prospero-related homeobox protein 1 and HDAC3.Cardiac hypertrophy in mice expressing unphosphorylatable phospholemman.Non-Invasive whole-body detection of complement activation using radionuclide imaging in a mouse model of myocardial ischaemia-reperfusion injury.Overexpression of the transcription factor Hand1 causes predisposition towards arrhythmia in mice.Cardioprotection by an anti-MASP-2 antibody in a murine model of myocardial infarction.Pravastatin therapy during preeclampsia prevents long-term adverse health effects in mice.Author Correction: Non-Invasive whole-body detection of complement activation using radionuclide imaging in a mouse model of myocardial ischaemia-reperfusion injury.Thymosin β4 facilitates epicardial neovascularization of the injured adult heartThe TAB1-p38α complex aggravates myocardial injury and can be targeted by small moleculesTissue acidosis does not mediate the hypoxia selectivity of [Cu][Cu(ATSM)] in the isolated perfused rat heartDetection of anthracycline-induced cardiotoxicity using perfusion-corrected Tc sestamibi SPECTPost-infarction remodeling is independent of mitogen-activated protein kinase kinase 3 (MKK3)Aged-senescent cells contribute to impaired heart regeneration.Response to role of hyperleptinemia in the regulation of blood pressure and cardiac functionTrapped platelets activated in ischemia initiate ventricular fibrillationThe POU4F2/Brn-3b transcription factor is required for the hypertrophic response to angiotensin II in the heart
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P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
James E Clark
@en
James E Clark
@nl
type
label
James E Clark
@en
James E Clark
@nl
prefLabel
James E Clark
@en
James E Clark
@nl
P31
P496
0000-0001-7574-2162