The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair
about
Functional consequences of mutations in the conserved SF2 motifs and post-translational phosphorylation of the CSB proteinCockayne syndrome group B protein has novel strand annealing and exchange activities.Structure, function and regulation of CSB: a multi-talented gymnastCockayne syndrome B protein stimulates apurinic endonuclease 1 activity and protects against agents that introduce base excision repair intermediatesIdentification of two missense mutations of ERCC6 in three Chinese sisters with Cockayne syndrome by whole exome sequencingDifferential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells.Molecular characterization of an acidic region deletion mutant of Cockayne syndrome group B protein.Cockayne syndrome group B cellular and biochemical functions.Tethering of the conserved piggyBac transposase fusion protein CSB-PGBD3 to chromosomal AP-1 proteins regulates expression of nearby genes in humans.Elements That Regulate the DNA Damage Response of Proteins Defective in Cockayne SyndromeRegulation of the Rhp26ERCC6/CSB chromatin remodeler by a novel conserved leucine latch motifLack of recognition by global-genome nucleotide excision repair accounts for the high mutagenicity and persistence of aristolactam-DNA adducts.Cockayne syndrome group B protein is engaged in processing of DNA adducts of lipid peroxidation product trans-4-hydroxy-2-nonenalA variant of the Cockayne syndrome B gene ERCC6 confers risk of lung cancer.Transcription-coupled nucleotide excision repair in mammalian cells: molecular mechanisms and biological effects.Nucleic acid binding activity of human Cockayne syndrome B protein and identification of Ca(2+) as a novel metal cofactorUV-induced association of the CSB remodeling protein with chromatin requires ATP-dependent relief of N-terminal autorepression.In UV-irradiated Saccharomyces cerevisiae, overexpression of Swi2/Snf2 family member Rad26 increases transcription-coupled repair and repair of the non-transcribed strand.The Cockayne syndrome group B protein is a functional dimer.ROS-induced R loops trigger a transcription-coupled but BRCA1/2-independent homologous recombination pathway through CSB
P2860
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P2860
The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair
description
1999 nî lūn-bûn
@nan
1999 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
The ATPase domain but not the ...... ct is essential for DNA repair
@ast
The ATPase domain but not the ...... ct is essential for DNA repair
@en
The ATPase domain but not the ...... ct is essential for DNA repair
@en-gb
The ATPase domain but not the ...... ct is essential for DNA repair
@nl
type
label
The ATPase domain but not the ...... ct is essential for DNA repair
@ast
The ATPase domain but not the ...... ct is essential for DNA repair
@en
The ATPase domain but not the ...... ct is essential for DNA repair
@en-gb
The ATPase domain but not the ...... ct is essential for DNA repair
@nl
prefLabel
The ATPase domain but not the ...... ct is essential for DNA repair
@ast
The ATPase domain but not the ...... ct is essential for DNA repair
@en
The ATPase domain but not the ...... ct is essential for DNA repair
@en-gb
The ATPase domain but not the ...... ct is essential for DNA repair
@nl
P2093
P2860
P356
P1476
The ATPase domain but not the ...... ct is essential for DNA repair
@en
P2093
P2860
P304
P356
10.1091/MBC.10.11.3583
P407
P577
1999-11-01T00:00:00Z