Structure, function and regulation of CSB: a multi-talented gymnast - Test2 - elhamkhani - TriplyDB

Structure, function and regulation of CSB: a multi-talented gymnast

Structure, function and regulation of CSB: a multi-talented gymnast

http://www.wikidata.org/entity/Q26995830

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Cell-autonomous progeroid changes in conditional mouse models for repair endonuclease XPG deficiency Transcriptional and Posttranslational Regulation of Nucleotide Excision Repair: The Guardian of the Genome against Ultraviolet Radiation A novel ERCC6 splicing variant associated with a mild Cockayne syndrome phenotype NAP1L1 accelerates activation and decreases pausing to enhance nucleosome remodeling by CSB CSB interacts with SNM1A and promotes DNA interstrand crosslink processing Elements That Regulate the DNA Damage Response of Proteins Defective in Cockayne Syndrome Regulation of the Rhp26ERCC6/CSB chromatin remodeler by a novel conserved leucine latch motif The sequence-specific transcription factor c-Jun targets Cockayne syndrome protein B to regulate transcription and chromatin structure.Body mass index is associated with gene methylation in estrogen receptor-positive breast tumors.The transcription-coupled repair protein ERCC6/CSB also protects against repeat expansion in a mouse model of the fragile X premutation.The Repeat Expansion Diseases: The dark side of DNA repair.The Cellular Response to Oxidatively Induced DNA Damage and Polymorphism of Some DNA Repair Genes Associated with Clinicopathological Features of Bladder Cancer.The CSB chromatin remodeler and CTCF architectural protein cooperate in response to oxidative stress.DNA excision repair at telomeres.The C-terminal Region and SUMOylation of Cockayne Syndrome Group B Protein Play Critical Roles in Transcription-coupled Nucleotide Excision Repair.Mfd as a central partner of transcription coupled repair.The role of Cockayne Syndrome Protein B in transcription regulation Overexpression of parkin rescues the defective mitochondrial phenotype and the increased apoptosis of Cockayne Syndrome A cells.Chromatin Architectural Changes during Cellular Senescence and Aging.ROS-induced R loops trigger a transcription-coupled but BRCA1/2-independent homologous recombination pathway through CSB Novel frame shift mutation in ERCC6 leads to a severe form of Cockayne syndrome with postnatal growth failure and early death: A case report and brief literature review Polymorphism of DNA repair genes OGG1, XRCC1, XPD and ERCC6 in bladder cancer in Belarus First molecular study in Lebanese patients with Cockayne syndrome and report of a novel mutation in ERCC8 gene

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Structure, function and regulation of CSB: a multi-talented gymnast

http://www.wikidata.org/entity/Q26995830

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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Structure, function and regulation of CSB: a multi-talented gymnast

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Structure, function and regulation of CSB: a multi-talented gymnast

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Structure, function and regulation of CSB: a multi-talented gymnast

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J.MAD.2013.02.004

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Mechanisms of Ageing and Development

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Structure, function and regulation of CSB: a multi-talented gymnast

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Hua-Ying Fan

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Robert J. Lake

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P2860

Reconstitution of a core chromatin remodeling complex from SWI/SNF subunits

The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair

Tumor suppressor HIC1 directly regulates SIRT1 to modulate p53-dependent DNA-damage responses

Functional consequences of mutations in the conserved SF2 motifs and post-translational phosphorylation of the CSB protein

Cockayne syndrome A and B proteins differentially regulate recruitment of chromatin remodeling and repair factors to stalled RNA polymerase II in vivo

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

HARP is an ATP-driven annealing helicase

Human transcription-repair coupling factor CSB/ERCC6 is a DNA-stimulated ATPase but is not a helicase and does not disrupt the ternary transcription complex of stalled RNA polymerase II

Cooperation of the Cockayne syndrome group B protein and poly(ADP-ribose) polymerase 1 in the response to oxidative stress.

Cockayne syndrome protein B interacts with and is phosphorylated by c-Abl tyrosine kinase

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202-11

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scholarly article

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10.1016/J.MAD.2013.02.004

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2013-01-01T00:00:00Z

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