Potential role for luman, the cellular homologue of herpes simplex virus VP16 (alpha gene trans-inducing factor), in herpesvirus latency
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Zhangfei: a second cellular protein interacts with herpes simplex virus accessory factor HCF in a manner similar to Luman and VP16.CREB-H: a novel mammalian transcription factor belonging to the CREB/ATF family and functioning via the box-B element with a liver-specific expressionZhangfei is a potent and specific inhibitor of the host cell factor-binding transcription factor LumanCooperative interaction of Zhangfei and ATF4 in transactivation of the cyclic AMP response elementLuman/CREB3 induces transcription of the endoplasmic reticulum (ER) stress response protein Herp through an ER stress response elementA novel protein, Luman/CREB3 recruitment factor, inhibits Luman activation of the unfolded protein responseA novel isoform of human LZIP negatively regulates the transactivation of the glucocorticoid receptorLuman, the cellular counterpart of herpes simplex virus VP16, is processed by regulated intramembrane proteolysisN-terminal transcriptional activation domain of LZIP comprises two LxxLL motifs and the host cell factor-1 binding motifComputational prediction of membrane-tethered transcription factorsThe liver-enriched transcription factor CREB-H is a growth suppressor protein underexpressed in hepatocellular carcinomaInteraction of HCF-1 with a cellular nuclear export factorThe luminal domain of ATF6 senses endoplasmic reticulum (ER) stress and causes translocation of ATF6 from the ER to the GolgiDe novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Control of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities.Gene array analysis reveals changes in peripheral nervous system gene expression following stimuli that result in reactivation of latent herpes simplex virus type 1: induction of transcription factor Bcl-3.Transcriptional profiling of chondrodysplasia growth plate cartilage reveals adaptive ER-stress networks that allow survival but disrupt hypertrophyEntry of herpes simplex virus type 1 (HSV-1) into the distal axons of trigeminal neurons favors the onset of nonproductive, silent infectionThe neuronal host cell factor-binding protein Zhangfei inhibits herpes simplex virus replication.Sensing nerve injury at the axonal ER: activated Luman/CREB3 serves as a novel axonally synthesized retrograde regeneration signal.Tegument protein control of latent herpesvirus establishment and animation.Zhangfei/CREB-ZF - a potential regulator of the unfolded protein responseHerpes simplex virus type 1 and bovine herpesvirus 1 latency.Knockdown of CREB3/Luman by shRNA in Mouse Granulosa Cells Results in Decreased Estradiol and Progesterone Synthesis and Promotes Cell Proliferation.Association of the cellular coactivator HCF-1 with the Golgi apparatus in sensory neurons.The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection.Recruitment of the transcriptional coactivator HCF-1 to viral immediate-early promoters during initiation of reactivation from latency of herpes simplex virus type 1.Expression pattern implicates a potential role for luman recruitment factor in the process of implantation in uteri and development of preimplantation embryos in mice.The signalling from endoplasmic reticulum-resident bZIP transcription factors involved in diverse cellular physiology.Roles of regulated intramembrane proteolysis in virus infection and antiviral immunity.Physiological functions of endoplasmic reticulum stress transducer OASIS in central nervous system.JAB1/CSN5 inhibits the activity of Luman/CREB3 by promoting its degradation.Gene expression profiling in the HSV-1 latently infected mouse trigeminal ganglia following hyperthermic stress.Transcriptional regulation of secretory capacity by bZip transcription factors.Loss of HCF-1-chromatin association precedes temperature-induced growth arrest of tsBN67 cells.Stabilization but not the transcriptional activity of herpes simplex virus VP16-induced complexes is evolutionarily conserved among HCF family members.Novel Brn3a cis-acting sequences mediate transcription of human trkA in neurons.Chlamydia pneumoniae inclusion membrane protein Cpn0147 interacts with host protein CREB3Cloning and characterization of rat Luman/CREB3, a transcription factor highly expressed in nervous system tissue.
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P2860
Potential role for luman, the cellular homologue of herpes simplex virus VP16 (alpha gene trans-inducing factor), in herpesvirus latency
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2000 nî lūn-bûn
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
@en-gb
Potential role for luman, the ...... actor), in herpesvirus latency
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Potential role for luman, the ...... actor), in herpesvirus latency
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10.1128/JVI.74.2.934-943.2000
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2000-01-01T00:00:00Z