Zhangfei: a second cellular protein interacts with herpes simplex virus accessory factor HCF in a manner similar to Luman and VP16.
about
Zhangfei is a potent and specific inhibitor of the host cell factor-binding transcription factor LumanHCF-1 functions as a coactivator for the zinc finger protein Krox20Identification of bZIP interaction partners of viral proteins HBZ, MEQ, BZLF1, and K-bZIP using coiled-coil arraysCooperative interaction of Zhangfei and ATF4 in transactivation of the cyclic AMP response elementLuman/CREB3 induces transcription of the endoplasmic reticulum (ER) stress response protein Herp through an ER stress response elementHost cell factor-1 interacts with and antagonizes transactivation by the cell cycle regulatory factor Miz-1A novel protein, Luman/CREB3 recruitment factor, inhibits Luman activation of the unfolded protein responseTranscriptional corepressor SMILE recruits SIRT1 to inhibit nuclear receptor estrogen receptor-related receptor gamma transactivationThe evolutionarily conserved longevity determinants HCF-1 and SIR-2.1/SIRT1 collaborate to regulate DAF-16/FOXOAn activation domain in the C-terminal subunit of HCF-1 is important for transactivation by VP16 and LZIP.A novel function for human factor C1 (HCF-1), a host protein required for herpes simplex virus infection, in pre-mRNA splicingInteraction of HCF-1 with a cellular nuclear export factorPDCD2 is a negative regulator of HCF-1 (C1)Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleControl of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities.The neuronal host cell factor-binding protein Zhangfei inhibits herpes simplex virus replication.Site-specific proteolysis of the transcriptional coactivator HCF-1 can regulate its interaction with protein cofactors.Tegument protein control of latent herpesvirus establishment and animation.Zhangfei/CREB-ZF - a potential regulator of the unfolded protein responseHerpes simplex virus type 1 and bovine herpesvirus 1 latency.Phosphorylation of the VP16 transcriptional activator protein during herpes simplex virus infection and mutational analysis of putative phosphorylation sites.Molecular cloning of Drosophila HCF reveals proteolytic processing and self-association of the encoded protein.Curcumin differentially regulates endoplasmic reticulum stress through transcriptional corepressor SMILE (small heterodimer partner-interacting leucine zipper protein)-mediated inhibition of CREBH (cAMP responsive element-binding protein H)The effect of Zhangfei/CREBZF on cell growth, differentiation, apoptosis, migration, and the unfolded protein response in several canine osteosarcoma cell lines.Inhibition of pyrimidine synthesis reverses viral virulence factor-mediated block of mRNA nuclear export.Herpes simplex virus infections are arrested in Oct-1-deficient cellsThe transcription factor CREBZF is a novel positive regulator of p53.Molecular characterization of SMILE as a novel corepressor of nuclear receptorsCREBZF expression and hormonal regulation in the mouse uterus.Ursodeoxycholic acid inhibits liver X receptor α-mediated hepatic lipogenesis via induction of the nuclear corepressor SMILE.A transcriptional regulatory role of the THAP11-HCF-1 complex in colon cancer cell function.A protein sequestering system reveals control of cellular programs by the transcriptional coactivator HCF-1.Effects of cyclic AMP response element binding protein-Zhangfei (CREBZF) on the unfolded protein response and cell growth are exerted through the tumor suppressor p53Mechanism for the induction of cell death in ONS-76 medulloblastoma cells by Zhangfei/CREB-ZF.CREBZF, a novel Smad8-binding protein.The effect of Zhangfei on the unfolded protein response and growth of cells derived from canine and human osteosarcomas.Loss of HCF-1-chromatin association precedes temperature-induced growth arrest of tsBN67 cells.Stabilization but not the transcriptional activity of herpes simplex virus VP16-induced complexes is evolutionarily conserved among HCF family members.Combinatorial transcription of herpes simplex virus and varicella zoster virus immediate early genes is strictly determined by the cellular coactivator HCF-1.Inhibition of LZIP-mediated transcription through direct interaction with a novel host cell factor-like protein.
P2860
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P248
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P2860
Zhangfei: a second cellular protein interacts with herpes simplex virus accessory factor HCF in a manner similar to Luman and VP16.
description
2000 nî lūn-bûn
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2000 թուականի Յունիսին հրատարակուած գիտական յօդուած
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2000 թվականի հունիսին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年论文
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Zhangfei: a second cellular pr ...... nner similar to Luman and VP16
@nl
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@ast
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en-gb
type
label
Zhangfei: a second cellular pr ...... nner similar to Luman and VP16
@nl
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@ast
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en-gb
prefLabel
Zhangfei: a second cellular pr ...... nner similar to Luman and VP16
@nl
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@ast
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en-gb
P2860
P921
P356
P1476
Zhangfei: a second cellular pr ...... ner similar to Luman and VP16.
@en
P2860
P304
P356
10.1093/NAR/28.12.2446
P407
P577
2000-06-01T00:00:00Z