NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells - Test2 - elhamkhani - TriplyDB

NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells

NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells

http://www.wikidata.org/entity/Q22254088

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Interferon regulatory factor 4 is involved in Epstein-Barr virus-mediated transformation of human B lymphocytes The oncogenic protein kinase Tpl-2/Cot contributes to Epstein-Barr virus-encoded latent infection membrane protein 1-induced NF-kappaB signaling downstream of TRAF2 The viral etiology of AIDS-associated malignancies Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.Epstein-Barr virus latent genes To be, or not to be: NF-kappaB is the answer--role of Rel/NF-kappaB in the regulation of apoptosis Host-pathogen interactions: subversion and utilization of the NF-kappa B pathway during infection.Cellular disorganization and extensive apoptosis in the developing heart of mice that lack cardiac muscle alpha-actin: apparent cause of perinatal death Lupeol is one of active components in the extract of Chrysanthemum indicum Linne that inhibits LMP1-induced NF-κB activation Genetic diversity of EBV-encoded LMP1 in the Swiss HIV Cohort Study and implication for NF-Κb activation c-Rel deficiency increases caspase-4 expression and leads to ER stress and necrosis in EBV-transformed cells Identification and Function of MicroRNAs Encoded by Herpesviruses Inhibition of the LKB1-AMPK pathway by the Epstein-Barr virus-encoded LMP1 promotes proliferation and transformation of human nasopharyngeal epithelial cells Epstein-Barr virus: 40 years on EBV latent membrane protein 1 activates Akt, NFkappaB, and Stat3 in B cell lymphomas.Yeast genetic selections to optimize RNA decoys for transcription factor NF-kappa B Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection Association between NFKB1 -94ins/del ATTG Promoter Polymorphism and Cancer Susceptibility: An Updated Meta-Analysis.RNA Viruses: ROS-Mediated Cell Death.Reactive oxygen signaling and MAPK activation distinguish Epstein-Barr Virus (EBV)-positive versus EBV-negative Burkitt's lymphoma.Dual expression of Epstein-Barr virus, latent membrane protein-1 and human papillomavirus-16 E6 transform primary mouse embryonic fibroblasts through NF-κB signaling An Epstein-Barr virus isolated from a lymphoblastoid cell line has a 16-kilobase-pair deletion which includes gp350 and the Epstein-Barr virus nuclear antigen 3A Epstein-Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growth Latent membrane protein 1 is dispensable for Epstein-Barr virus replication in human embryonic kidney 293 cells.Activation of NF-κB via endosomal Toll-like receptor 7 (TLR7) or TLR9 suppresses murine herpesvirus 68 reactivation.Control of oncogenesis and cancer therapy resistance by the transcription factor NF-kappaB.Epstein-Barr virus EBNA2 blocks Nur77- mediated apoptosis The NF-κB genomic landscape in lymphoblastoid B cells.Molecular virology of Epstein-Barr virus.Epstein-Barr latent membrane protein 1 transformation site 2 activates NF-kappaB in the absence of NF-kappaB essential modifier residues 133-224 or 373-419.SplicerEX: a tool for the automated detection and classification of mRNA changes from conventional and splice-sensitive microarray expression data.The Epstein-Barr virus latent membrane protein 1 putative Janus kinase 3 (JAK3) binding domain does not mediate JAK3 association or activation in B-lymphoma or lymphoblastoid cell lines Toll-like receptor 7 stimulates the expression of Epstein-Barr virus latent membrane protein 1 Virus and cell RNAs expressed during Epstein-Barr virus replication EBNA3A association with RBP-Jkappa down-regulates c-myc and Epstein-Barr virus-transformed lymphoblast growth.Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines.RNAs induced by Epstein-Barr virus nuclear antigen 2 in lymphoblastoid cell lines.IL-1 receptor-associated kinase 1 is critical for latent membrane protein 1-induced p65/RelA serine 536 phosphorylation and NF-kappaB activation.Epstein-Barr virus latent membrane protein 1 activation of NF-kappaB through IRAK1 and TRAF6.CD20 antibody primes B lymphocytes for type I interferon production.

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NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells

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2000 nî lūn-bûn

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2000 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2000 թվականի մայիսին հրատարակված գիտական հոդված

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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Proceedings of the National Academy of Sciences of the United States of America

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NF-kappa B inhibition causes s ...... ansformed lymphoblastoid cells

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D M Davidson

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E D Cahir-McFarland

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J Duong

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S L Schauer

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P2860

Molecular characterization of mitochondrial apoptosis-inducing factor

The amino-terminal domains of Epstein-Barr virus nuclear proteins 3A, 3B, and 3C interact with RBPJ(kappa)

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

Coupling of a signal response domain in I kappa B alpha to multiple pathways for NF-kappa B activation

TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways

The TNF receptor 1-associated protein TRADD signals cell death and NF-kappa B activation

Epstein-Barr virus-encoded latent membrane protein 1 activates the JNK pathway through its extreme C terminus via a mechanism involving TRADD and TRAF2

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis

The Epstein-Barr virus oncoprotein latent membrane protein 1 engages the tumor necrosis factor receptor-associated proteins TRADD and receptor-interacting protein (RIP) but does not induce apoptosis or require RIP for NF-kappaB activation

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6055-60

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10.1073/PNAS.100119497

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11

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97

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Elliott D. Kieff

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2000-05-23T00:00:00Z

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12505752

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10811897

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apoptotic process

negative regulation of apoptotic process

Nuclear factor kappa B subunit 1

Epstein–Barr virus

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18557

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