Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis. - Test2 - elhamkhani - TriplyDB

Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

http://www.wikidata.org/entity/Q24816875

about

An Epstein-Barr virus anti-apoptotic protein constitutively expressed in transformed cells and implicated in burkitt lymphomagenesis: the Wp/BHRF1 link Vaccinia virus anti-apoptotic F1L is a novel Bcl-2-like domain-swapped dimer that binds a highly selective subset of BH3-containing death ligands Extracellular vesicles during Herpes Simplex Virus type 1 infection: an inquire Potential cellular functions of Epstein-Barr Nuclear Antigen 1 (EBNA1) of Epstein-Barr Virus Epstein-Barr virus and host cell methylation: regulation of latency, replication and virus reactivation Structural and Biochemical Bases for the Inhibition of Autophagy and Apoptosis by Viral BCL-2 of Murine γ-Herpesvirus 68 Molecular basis of the regulation of Beclin 1-dependent autophagy by the gamma-herpesvirus 68 Bcl-2 homolog M11 Structural Basis for Apoptosis Inhibition by Epstein-Barr Virus BHRF1 Structural Insight into BH3 Domain Binding of Vaccinia Virus Antiapoptotic F1L A Computationally Designed Inhibitor of an Epstein-Barr Viral Bcl-2 Protein Induces Apoptosis in Infected Cells Structural basis of Deerpox virus-mediated inhibition of apoptosis ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus Resveratrol prevents EBV transformation and inhibits the outgrowth of EBV-immortalized human B cells BH3 domains define selective inhibitory interactions with BHRF-1 and KSHV BCL-2 EBV, HHV8 and HIV in B cell non Hodgkin lymphoma in Kampala, Uganda.Human microRNAs target a functionally distinct population of genes with AT-rich 3' UTRs.Molecular signature of Epstein Barr virus-positive Burkitt lymphoma and post-transplant lymphoproliferative disorder suggest different roles for Epstein Barr virus.Epstein-Barr virus and multiple sclerosis: potential opportunities for immunotherapy AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.Childhood cancers: what is a possible role of infectious agents?Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection Micro RNAs of Epstein-Barr virus promote cell cycle progression and prevent apoptosis of primary human B cells.Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression Conserved microRNA targeting in Drosophila is as widespread in coding regions as in 3'UTRs.Cellular microRNAs 200b and 429 regulate the Epstein-Barr virus switch between latency and lytic replication.The EBV immunoevasins vIL-10 and BNLF2a protect newly infected B cells from immune recognition and elimination Multiple functions of BCL-2 family proteins.Identification and characterization of microRNA in the dairy goat (Capra hircus) mammary gland by Solexa deep-sequencing technology.Quantitative studies of Epstein-Barr virus-encoded microRNAs provide novel insights into their regulation.MicroRNA-155 regulates human angiotensin II type 1 receptor expression in fibroblasts.VR09 cell line: an EBV-positive lymphoblastoid cell line with in vivo characteristics of diffuse large B cell lymphoma of activated B-cell type The effect of Epstein-Barr virus Latent Membrane Protein 2 expression on the kinetics of early B cell infection Burkitt's lymphoma: the Rosetta Stone deciphering Epstein-Barr virus biology.Unexpected patterns of Epstein-Barr virus transcription revealed by a high throughput PCR array for absolute quantification of viral mRNA.Autophagy in herpesvirus immune control and immune escape.Tegument protein control of latent herpesvirus establishment and animation.Interferon regulatory factor 4 (IRF-4) targets IRF-5 to regulate Epstein-Barr virus transformation.The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latency Transcriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.

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Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

http://www.wikidata.org/entity/Q24816875

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2005 nî lūn-bûn

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2005年の論文

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Markus Altmann

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Wolfgang Hammerschmidt

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P2860

NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells

Viruses and Apoptosis

Strategies and mechanisms for host and pathogen survival in acute and persistent viral infections

The EBV nuclear antigen 1 (EBNA1) enhances B cell immortalization several thousandfold

Viral homologs of BCL-2: role of apoptosis in the regulation of virus infection

Gene disruption in Escherichia coli: TcR and KmR cassettes with the option of Flp-catalyzed excision of the antibiotic-resistance determinant

Liquid-crystalline, phage-like packing of encapsidated DNA in herpes simplex virus

A model for persistent infection with Epstein-Barr virus: the stealth virus of human B cells.

Epstein-Barr virus-mediated B-cell proliferation is dependent upon latent membrane protein 1, which simulates an activated CD40 receptor

BHRF1 of Epstein-Barr virus, which is homologous to human proto-oncogene bcl2, is not essential for transformation of B cells or for virus replication in vitro.

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16277553

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apoptotic process

Epstein–Barr virus

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1283332

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