Transforming growth factor beta -inducible independent binding of SMAD to the Smad7 promoter
about
Transcription activation of FLRG and follistatin by activin A, through Smad proteins, participates in a negative feedback loop to modulate activin A functionSmad7 antagonizes transforming growth factor beta signaling in the nucleus by interfering with functional Smad-DNA complex formationTransforming growth factor-beta inhibits pulmonary surfactant protein B gene transcription through SMAD3 interactions with NKX2.1 and HNF-3 transcription factorsStoichiometry of active smad-transcription factor complexes on DNAActivation of Smad transcriptional activity by protein inhibitor of activated STAT3 (PIAS3)GATA- and Smad1-dependent enhancers in the Smad7 gene differentially interpret bone morphogenetic protein concentrationsInterfering with TGFbeta-induced Smad3 nuclear accumulation differentially affects TGFbeta-dependent gene expressionSynergism between transcription factors TFE3 and Smad3 in transforming growth factor-beta-induced transcription of the Smad7 geneFLRG, an activin-binding protein, is a new target of TGFbeta transcription activation through Smad proteinsInhibition of BMP2-induced, TAK1 kinase-mediated neurite outgrowth by Smad6 and Smad7Roles for the MH2 domain of Smad7 in the specific inhibition of transforming growth factor-beta superfamily signalingTGF-beta inhibits muscle differentiation through functional repression of myogenic transcription factors by Smad3Smad4 dependency defines two classes of transforming growth factor {beta} (TGF-{beta}) target genes and distinguishes TGF-{beta}-induced epithelial-mesenchymal transition from its antiproliferative and migratory responses.Opposite Smad and chicken ovalbumin upstream promoter transcription factor inputs in the regulation of the collagen VII gene promoter by transforming growth factor-beta.Pin1 promotes transforming growth factor-beta-induced migration and invasion.Graded Nodal/Activin signaling titrates conversion of quantitative phospho-Smad2 levels into qualitative embryonic stem cell fate decisions.Defective repression of c-myc in breast cancer cells: A loss at the core of the transforming growth factor beta growth arrest program.TSC-22 promotes transforming growth factor β-mediated cardiac myofibroblast differentiation by antagonizing Smad7 activity.Fibroblast growth factor 9 (FGF9)-pituitary homeobox 2 (PITX2) pathway mediates transforming growth factor β (TGFβ) signaling to regulate cell proliferation in palatal mesenchyme during mouse palatogenesis.Repression of Smad transcriptional activity by PIASy, an inhibitor of activated STAT.Structural basis for the Smad5 MH1 domain to recognize different DNA sequences.The N domain of Smad7 is essential for specific inhibition of transforming growth factor-beta signaling.Transforming growth factor (TGF)-beta1-producing regulatory T cells induce Smad-mediated interleukin 10 secretion that facilitates coordinated immunoregulatory activity and amelioration of TGF-beta1-mediated fibrosis.Smad4-Irf6 genetic interaction and TGFβ-mediated IRF6 signaling cascade are crucial for palatal fusion in mice.Valproic acid suppresses collagen by selective regulation of Smads in conjunctival fibrosis.A dynein motor attachment complex regulates TGFß/Smad3 signaling.TGF-β signalling and reactive oxygen species drive fibrosis and matrix remodelling in myxomatous mitral valves.Smad2 is essential for maintenance of the human and mouse primed pluripotent stem cell state.Chromatin immunoprecipitation on microarray analysis of Smad2/3 binding sites reveals roles of ETS1 and TFAP2A in transforming growth factor beta signaling.Structure-aided prediction of mammalian transcription factor complexes in conserved non-coding elements.MicroRNA-182 targets SMAD7 to potentiate TGFβ-induced epithelial-mesenchymal transition and metastasis of cancer cells.Smad7: not only a regulator, but also a cross-talk mediator of TGF-β signalling.SMAD7: a timer of tumor progression targeting TGF-β signaling.Regulation of TGF-β Family Signaling by Inhibitory Smads.Decapentaplegic-responsive silencers contain overlapping mad-binding sites.Smad proteins are targets of transforming growth factor beta1 in immortalised gonadotrophin-releasing hormone releasing neurones.Repression of endogenous Smad7 by Ski.Evidence for a role of the JNK cascade in Smad7-mediated apoptosis.Inactivation of TGFβ receptors in stem cells drives cutaneous squamous cell carcinomaRepression of Smad4 transcriptional activity by SUMO modification.
P2860
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P2860
Transforming growth factor beta -inducible independent binding of SMAD to the Smad7 promoter
description
2000 nî lūn-bûn
@nan
2000 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@ast
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en-gb
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@nl
type
label
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@ast
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en-gb
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@nl
prefLabel
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@ast
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en-gb
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@nl
P2093
P2860
P921
P3181
P356
P1476
Transforming growth factor bet ...... of SMAD to the Smad7 promoter
@en
P2093
P2860
P304
P3181
P356
10.1073/PNAS.090099297
P407
P577
2000-06-06T00:00:00Z