CUL9 mediates the functions of the 3M complex and ubiquitylates survivin to maintain genome integrity.
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Survivin - The inconvenient IAPAn XRCC4 splice mutation associated with severe short stature, gonadal failure, and early-onset metabolic syndrome.The Proapoptotic F-box Protein Fbxl7 Regulates Mitochondrial Function by Mediating the Ubiquitylation and Proteasomal Degradation of SurvivinThe GALNT9, BNC1 and CCDC8 genes are frequently epigenetically dysregulated in breast tumours that metastasise to the brainAurora kinase A regulates Survivin stability through targeting FBXL7 in gastric cancer drug resistance and prognosis.microRNA-143/145 loss induces Ras signaling to promote aggressive Pten-deficient basal-like breast cancer.Cytoplasmic E3 ubiquitin ligase CUL9 controls cell proliferation, senescence, apoptosis and genome integrity through p53.Chromosomal instability induced by increased BIRC5/Survivin levels affects tumorigenicity of glioma cells.NEDD8:AcM-UBE2M binds CUL9:RBX1 ubiquitin ligase complexAcM-UBE2M transfers NEDD8 to CUL9:RBX1CUL9:RBX1 ubiquitinates BIRC5NEDD8-CUL9:RBX1 binds CUL7:CCDC8:OBSL1Chromatin-Bound Cullin-Ring Ligases: Regulatory Roles in DNA Replication and Potential Targeting for Cancer Therapy.A novel mutation in a Japanese patient with 3M syndrome
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CUL9 mediates the functions of the 3M complex and ubiquitylates survivin to maintain genome integrity.
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2014 nî lūn-bûn
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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2014 թվականի մայիսին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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Angelique W Whitehurst
Kathryn M Cappell
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10.1016/J.MOLCEL.2014.03.046
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P50
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2014-05-01T00:00:00Z