Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A
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Regulation of TopBP1 oligomerization by Akt/PKB for cell survivalAdapting the Stress Response: Viral Subversion of the mTOR Signaling PathwaymTOR Regulation of Lymphoid Cells in Immunity to PathogensRegulation of the Wnt/β-Catenin Signaling Pathway by Human Papillomavirus E6 and E7 OncoproteinsNanomedicine strategies for sustained, controlled, and targeted treatment of cancer stem cells of the digestive systemComputer-aided targeting of the PI3K/Akt/mTOR pathway: toxicity reduction and therapeutic opportunitiesThe Human Papillomavirus 16 E7 Oncoprotein Attenuates AKT Signaling To Promote Internal Ribosome Entry Site-Dependent Translation and Expression of c-MYC.Improved ATM kinase inhibitor KU-60019 radiosensitizes glioma cells, compromises insulin, AKT and ERK prosurvival signaling, and inhibits migration and invasionPhysiological regulation of Akt activity and stability.Retinoblastoma-independent antiproliferative activity of novel intracellular antibodies against the E7 oncoprotein in HPV 16-positive cellsAvian reovirus p17 and σA act cooperatively to downregulate Akt by suppressing mTORC2 and CDK2/cyclin A2 and upregulating proteasome PSMB6.Differential regulation of cutaneous oncoprotein HPVE6 by wtp53, mutant p53R248W and ΔNp63α is HPV type dependent.The battle over mTOR: an emerging theatre in host-pathogen immunityPIK3CA-mediated PI3-kinase signalling is essential for HPV-induced transformation in vitroE6 and E7 from human papillomavirus type 16 cooperate to target the PDZ protein Na/H exchange regulatory factor 1.The Protein Phosphatase 2A regulatory subunit Twins stabilizes Plk4 to induce centriole amplification.Modulation of apoptosis by human papillomavirus (HPV) oncoproteins.The role of the PI3K/Akt/mTOR signalling pathway in human cancers induced by infection with human papillomaviruses.Cellular entry of human papillomavirus type 16 involves activation of the phosphatidylinositol 3-kinase/Akt/mTOR pathway and inhibition of autophagy.Increased phosphorylation of p70 S6 kinase is associated with HPV16 infection in cervical cancer and esophageal cancer.HPV-18 E6 mutants reveal p53 modulation of viral DNA amplification in organotypic cultures.PP2A-dependent disruption of centrosome replication and cytoskeleton organization in Drosophila by SV40 small tumor antigenCellular transformation by human papillomaviruses: lessons learned by comparing high- and low-risk virusesThe TORrid affairs of viruses: effects of mammalian DNA viruses on the PI3K-Akt-mTOR signalling pathway.Phosphorylation of the human papillomavirus type 16 E1--E4 protein at T57 by ERK triggers a structural change that enhances keratin binding and protein stabilityThe papillomavirus E7 proteins.The dual pathway inhibitor rigosertib is effective in direct patient tumor xenografts of head and neck squamous cell carcinomas.Phosphoproteome Analysis Reveals the Molecular Mechanisms Underlying Deoxynivalenol-Induced Intestinal Toxicity in IPEC-J2 CellsModulation of autophagy-like processes by tumor viruses.Glycogen synthase kinase 3 beta: can it be a target for oral cancer.The critical protein interactions and structures that elicit growth deregulation in cancer and viral replication.Show me your license, please: deregulation of centriole duplication mechanisms that promote amplification.Make yourself at home: viral hijacking of the PI3K/Akt signaling pathway.Inhibition of PP1 phosphatase activity by HBx: a mechanism for the activation of hepatitis B virus transcription.Signaling pathways in HPV-associated cancers and therapeutic implications.Metabolic reprogramming: a hallmark of viral oncogenesis.Cutaneous human papillomaviruses down-regulate AKT1, whereas AKT2 up-regulation and activation associates with tumors.Overexpressed-eIF3I interacted and activated oncogenic Akt1 is a theranostic target in human hepatocellular carcinoma.The human papillomavirus type 16 E6 oncoprotein activates mTORC1 signaling and increases protein synthesis.The human papillomavirus (HPV) E6* proteins from high-risk, mucosal HPVs can direct degradation of cellular proteins in the absence of full-length E6 protein.
P2860
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P2860
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A
description
2005 nî lūn-bûn
@nan
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Activation of the protein kina ...... nvolving interaction with PP2A
@ast
Activation of the protein kina ...... nvolving interaction with PP2A
@en
Activation of the protein kina ...... nvolving interaction with PP2A
@en-gb
Activation of the protein kina ...... nvolving interaction with PP2A
@nl
type
label
Activation of the protein kina ...... nvolving interaction with PP2A
@ast
Activation of the protein kina ...... nvolving interaction with PP2A
@en
Activation of the protein kina ...... nvolving interaction with PP2A
@en-gb
Activation of the protein kina ...... nvolving interaction with PP2A
@nl
prefLabel
Activation of the protein kina ...... nvolving interaction with PP2A
@ast
Activation of the protein kina ...... nvolving interaction with PP2A
@en
Activation of the protein kina ...... nvolving interaction with PP2A
@en-gb
Activation of the protein kina ...... nvolving interaction with PP2A
@nl
P2093
P2860
P3181
P356
P1433
P1476
Activation of the protein kina ...... nvolving interaction with PP2A
@en
P2093
Lawrence Banks
Paola Massimi
Stephen M Dilworth
P2860
P2888
P304
P3181
P356
10.1038/SJ.ONC.1208935
P407
P577
2005-11-24T00:00:00Z
P5875
P6179
1046942463