HPV-18 E6 mutants reveal p53 modulation of viral DNA amplification in organotypic cultures.
about
Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6The Role of the DNA Damage Response throughout the Papillomavirus Life CycleStructure based identification and characterization of flavonoids that disrupt human papillomavirus-16 E6 functionViral DNA Replication Orientation and hnRNPs Regulate Transcription of the Human Papillomavirus 18 Late Promoter.CCCTC-binding factor recruitment to the early region of the human papillomavirus 18 genome regulates viral oncogene expression.Serine/Arginine-Rich Splicing Factor 3 and Heterogeneous Nuclear Ribonucleoprotein A1 Regulate Alternative RNA Splicing and Gene Expression of Human Papillomavirus 18 through Two Functionally Distinguishable cis ElementsThe viral E8^E2C repressor limits productive replication of human papillomavirus 16HPV E6 oncoprotein as a potential therapeutic target in HPV related cancers.Signaling pathways in HPV-associated cancers and therapeutic implications.Model systems to study the life cycle of human papillomaviruses and HPV-associated cancers.HPV 16 E5 oncoprotein is expressed in early stage carcinogenesis and can be a target of immunotherapy.The human papillomavirus E7 oncoprotein as a regulator of transcription.Host cell restriction factors that limit transcription and replication of human papillomavirus.The role of protein kinase A regulation of the E6 PDZ-binding domain during the differentiation-dependent life cycle of human papillomavirus type 18.Mechanisms by which HPV Induces a Replication Competent Environment in Differentiating Keratinocytes.Human papillomavirus type 18 E5 oncogene supports cell cycle progression and impairs epithelial differentiation by modulating growth factor receptor signalling during the virus life cycle.Evaluation of ODE-Bn-PMEG, an acyclic nucleoside phosphonate prodrug, as an antiviral against productive HPV infection in 3D organotypic epithelial cultures.STAT3 activation by E6 is essential for the differentiation-dependent HPV18 life cycle.The DNA damage response activates HPV16 late gene expression at the level of RNA processing.
P2860
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P2860
HPV-18 E6 mutants reveal p53 modulation of viral DNA amplification in organotypic cultures.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
HPV-18 E6 mutants reveal p53 m ...... ation in organotypic cultures.
@en
type
label
HPV-18 E6 mutants reveal p53 m ...... ation in organotypic cultures.
@en
prefLabel
HPV-18 E6 mutants reveal p53 m ...... ation in organotypic cultures.
@en
P2093
P2860
P356
P1476
HPV-18 E6 mutants reveal p53 m ...... ation in organotypic cultures.
@en
P2093
Eun-Young Kho
Hsu-Kun Wang
Louise T Chow
N Sanjib Banerjee
Thomas R Broker
P2860
P304
P356
10.1073/PNAS.1304855110
P407
P577
2013-04-09T00:00:00Z