HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
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HIV Genome-Wide Protein Associations: a Review of 30 Years of ResearchRemodeling of the Host Cell Plasma Membrane by HIV-1 Nef and Vpu: A Strategy to Ensure Viral Fitness and PersistenceFusion of Enveloped Viruses in Endosomes.Differential Control of BST2 Restriction and Plasmacytoid Dendritic Cell Antiviral Response by Antagonists Encoded by HIV-1 Group M and O Strains.SERINC5 protein inhibits HIV-1 fusion pore formation by promoting functional inactivation of envelope glycoproteins.Exposing HIV's weaknessesCyclin F/FBXO1 Interacts with HIV-1 Viral Infectivity Factor (Vif) and Restricts Progeny Virion Infectivity by Ubiquitination and Proteasomal Degradation of Vif Protein through SCFcyclin F E3 Ligase MachineryHIV-1 Nef Signaling in Intestinal Mucosa Epithelium Suggests the Existence of an Active Inter-kingdom Crosstalk Mediated by Exosomes.Mechanisms of Virologic Control and Clinical Characteristics of HIV+ Elite/Viremic ControllersSulforaphane Inhibits HIV Infection of Macrophages through Nrf2.They Might Be Giants: Does Syncytium Formation Sink or Spread HIV Infection?Tuning of AKT-pathway by Nef and its blockade by protease inhibitors results in limited recovery in latently HIV infected T-cell line.The Evolutionary Histories of Antiretroviral Proteins SERINC3 and SERINC5 Do Not Support an Evolutionary Arms Race in Primates.Activation of the DNA Damage Response Is a Conserved Function of HIV-1 and HIV-2 Vpr That Is Independent of SLX4 RecruitmentChanges in the Plasticity of HIV-1 Nef RNA during the Evolution of the North American Epidemic.A Highly Conserved Residue in HIV-1 Nef Alpha Helix 2 Modulates Protein Expression.The Antagonism of HIV-1 Nef to SERINC5 Particle Infectivity Restriction Involves the Counteraction of Virion-Associated Pools of the Restriction Factor.Functional Interplay Between Murine Leukemia Virus Glycogag, Serinc5, and Surface Glycoprotein Governs Virus Entry, with Opposite Effects on Gammaretroviral and Ebolavirus Glycoproteins.TLR7/8 agonist induces a post-entry SAMHD1-independent block to HIV-1 infection of monocytes.S2 from equine infectious anemia virus is an infectivity factor which counteracts the retroviral inhibitors SERINC5 and SERINC3.Hili Inhibits HIV Replication in Activated T Cells.Modeling the evolution of SIV sooty mangabey progenitor virus towards HIV-2 using humanized mice.Cellular Antiviral Factors that Target Particle Infectivity of HIV-1Cell-based Fluorescence Complementation Reveals a Role for HIV-1 Nef Protein Dimerization in AP-2 Adaptor Recruitment and CD4 Co-receptor Down-regulation.Effect of HIV-1 Env on SERINC5 AntagonismLipopolysaccharide suppresses human immunodeficiency virus 1 reverse transcription in macrophages.Amino acids at positions 3, 168, and 169 are associated with the ability of Nef proteins from HIV-1 CRF01_AE to downmodulate CD4.HIV-1 Nef: Taking Control of Protein Trafficking.Lipopeptides: a novel antigen repertoire presented by major histocompatibility complex class I molecules.The V3 Loop of HIV-1 Env Determines Viral Susceptibility to IFITM3 Impairment of Viral Infectivity.Role of Innate Genes in HIV Replication.Endocytic sorting motif interactions involved in Nef-mediated downmodulation of CD4 and CD3.Primate lentiviruses use at least three alternative strategies to suppress NF-κB-mediated immune activation.HIV-1 Nef-induced cardiotoxicity through dysregulation of autophagy.The host-cell restriction factor SERINC5 restricts HIV-1 infectivity without altering the lipid composition and organization of viral particles.Identification of SERINC5-001 as the Predominant Spliced Isoform for HIV-1 Restriction.BCA2/Rabring7 Interferes with HIV-1 Proviral Transcription by Enhancing the SUMOylation of IκBα.Enhancing Virion Tethering by BST2 Sensitizes Productively and Latently HIV-infected T cells to ADCC Mediated by Broadly Neutralizing Antibodies.HIV-1 Nef sequesters MHC-I intracellularly by targeting early stages of endocytosis and recycling.The Potency of Nef-Mediated SERINC5 Antagonism Correlates with the Prevalence of Primate Lentiviruses in the Wild.
P2860
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P2860
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
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2015 nî lūn-bûn
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2015 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
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2015 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@ast
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en-gb
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@nl
type
label
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@ast
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en-gb
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@nl
prefLabel
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@ast
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en-gb
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@nl
P2093
P2860
P50
P921
P3181
P356
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P1476
HIV-1 Nef promotes infection by excluding SERINC5 from virion incorporation
@en
P2093
Anetta Nowosielska
Annachiara Rosa
Massimo Pizzato
Roberto Bertorelli
Sean M McCauley
Serena Ziglio
Shih Lin Goh
Veronica De Sanctis
P2860
P2888
P304
P3181
P356
10.1038/NATURE15399
P407
P577
2015-09-30T00:00:00Z
P6179
1011008448