Functional cross-antagonism between transcription factors FLI-1 and EKLF - Test2 - elhamkhani - TriplyDB

Functional cross-antagonism between transcription factors FLI-1 and EKLF

Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Mammalian Krüppel-like factors in health and diseases Genomic landscape of megakaryopoiesis and platelet function defects Hematopoiesis: an evolving paradigm for stem cell biology Overexpression of Fli-1 in astrocytoma is associated with poor prognosis.Transcriptional control of megakaryocyte development.Characterization of megakaryocyte GATA1-interacting proteins: the corepressor ETO2 and GATA1 interact to regulate terminal megakaryocyte maturation Megakaryopoiesis Thrombocytopenia in mice lacking the carboxy-terminal regulatory domain of the Ets transcription factor Fli1.Unique in vitro and in vivo thrombopoietic activities of ingenol 3,20 dibenzoate, a Ca(++)-independent protein kinase C isoform agonist.Congenital amegakaryocytic thrombocytopenia iPS cells exhibit defective MPL-mediated signaling.Distinct, strict requirements for Gfi-1b in adult bone marrow red cell and platelet generation.Identification of nuclear import and export signals within Fli-1: roles of the nuclear import signals in Fli-1-dependent activation of megakaryocyte-specific promoters.Direct competition between DNA binding factors highlights the role of Krüppel-like Factor 1 in the erythroid/megakaryocyte switch Oncogenic Fli-1 is a potential prognostic marker for the progression of epithelial ovarian cancer.Krüppel-like factors compete for promoters and enhancers to fine-tune transcription.A large gene network in immature erythroid cells is controlled by the myeloid and B cell transcriptional regulator PU.1.Hierarchical differentiation of myeloid progenitors is encoded in the transcription factor network Spi-1, Fli-1 and Fli-3 (miR-17-92) oncogenes contribute to a single oncogenic network controlling cell proliferation in friend erythroleukemia.Dynamic transcription factor activity profiles reveal key regulatory interactions during megakaryocytic and erythroid differentiation Eafs control erythroid cell fate by regulating c-myb expression through Wnt signaling.Transcription factor networks in erythroid cell and megakaryocyte development The multifunctional role of EKLF/KLF1 during erythropoiesis.Prospective isolation of human erythroid lineage-committed progenitors.Important genes in the pathogenesis of 5q- syndrome and their connection with ribosomal stress and the innate immune system pathway.Novel roles for KLF1 in erythropoiesis revealed by mRNA-seq.Reciprocal regulation of alternative lineages by Rgs18 and its transcriptional repressor Gfi1b α-1,6-Fucosyltransferase (FUT8) inhibits hemoglobin production during differentiation of murine and K562 human erythroleukemia cells.Spi-1 and Fli-1 directly activate common target genes involved in ribosome biogenesis in Friend erythroleukemic cells.Gfi1 integrates progenitor versus granulocytic transcriptional programming.The role of PU.1 and GATA-1 transcription factors during normal and leukemogenic hematopoiesis.Emerging properties of animal gene regulatory networks.Ex vivo production of platelets from stem cells.Gene Ontology-driven transcriptional analysis of CD34+ cell-initiated megakaryocytic cultures identifies new transcriptional regulators of megakaryopoiesis.Decoding early myelopoiesis from dynamics of core endogenous network.NLS-tagging: an alternative strategy to tag nuclear proteins.Dual actions of Meis1 inhibit erythroid progenitor development and sustain general hematopoietic cell proliferation.Differentiation-dependent interactions between RUNX-1 and FLI-1 during megakaryocyte development.Ets-1 regulates plasma cell differentiation by interfering with the activity of the transcription factor Blimp-1.Novel role for EKLF in megakaryocyte lineage commitment.TEL/ETV6 accelerates erythroid differentiation and inhibits megakaryocytic maturation in a human leukemia cell line UT-7/GM.

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

http://www.wikidata.org/entity/Q24541389

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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MCB.23.4.1390-1402.2003

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Molecular and Cellular Biology

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Functional cross-antagonism between transcription factors FLI-1 and EKLF

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Alexandre Doubeikovski

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Colette Gonnet

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François Morle

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Joëlle Starck

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Nathalie Cohet

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Sandrine Sarrazin

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Zina Doubeikovskaia

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P2860

mHDA1/HDAC5 histone deacetylase interacts with and represses MEF2A transcriptional activity

Hemorrhage, impaired hematopoiesis, and lethality in mouse embryos carrying a targeted disruption of the Fli1 transcription factor

Fli-1, an Ets-related transcription factor, regulates erythropoietin-induced erythroid proliferation and differentiation: evidence for direct transcriptional repression of the Rb gene during differentiation.

Erythroid Krüppel-like factor (EKLF) is active in primitive and definitive erythroid cells and is required for the function of 5'HS3 of the beta-globin locus control region

Regulation of transcription factor YY1 by acetylation and deacetylation

Spi-1/PU.1 is a positive regulator of the Fli-1 gene involved in inhibition of erythroid differentiation in friend erythroleukemic cell lines

Direct interaction of hematopoietic transcription factors PU.1 and GATA-1: functional antagonism in erythroid cells

A novel, erythroid cell-specific murine transcription factor that binds to the CACCC element and is related to the Krüppel family of nuclear proteins

Negative cross-talk between hematopoietic regulators: GATA proteins repress PU.1

Physical and functional interactions between the transcription factor PU.1 and the coactivator CBP

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1390-1402

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10.1128/MCB.23.4.1390-1402.2003

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4

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23

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Martine Duterque-Coquillaud

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2003-02-01T00:00:00Z

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12556498

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141137

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