Sendai virus infection induces efficient adaptive immunity independently of type I interferons
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Cloned Defective Interfering Influenza RNA and a Possible Pan-Specific Treatment of Respiratory Virus DiseasesA Protective Role for Dengue Virus-Specific CD8+ T CellsDisease-promoting effects of type I interferons in viral, bacterial, and coinfectionsMapping and Role of the CD8+ T Cell Response During Primary Zika Virus Infection in MiceA novel small molecule inhibitor of influenza A viruses that targets polymerase function and indirectly induces interferonE3 Ubiquitin Ligase NEDD4 Promotes Influenza Virus Infection by Decreasing Levels of the Antiviral Protein IFITM3Alpha/beta interferon receptor signaling amplifies early proinflammatory cytokine production in the lung during respiratory syncytial virus infection.Granulocyte colony-stimulating factor protects mice during respiratory virus infections.Toll-like receptor-independent triggering of dendritic cell maturation by viruses.Chemoproteomics reveals Toll-like receptor fatty acylation.Sendai virus pathogenesis in mice is prevented by Ifit2 and exacerbated by interferon.Novel mode of ISG15-mediated protection against influenza A virus and Sendai virus in mice.Defective viral genomes arising in vivo provide critical danger signals for the triggering of lung antiviral immunityIL-27 Limits Type 2 Immunopathology Following Parainfluenza Virus Infection.The Ebola virus VP35 protein binds viral immunostimulatory and host RNAs identified through deep sequencingType I interferons are essential in controlling neurotropic coronavirus infection irrespective of functional CD8 T cells.The Impact of the Interferon/TNF-Related Apoptosis-Inducing Ligand Signaling Axis on Disease Progression in Respiratory Viral Infection and Beyond.Highly immunostimulatory RNA derived from a Sendai virus defective viral genomeType I Interferon-Mediated Induction of Antiviral Genes and Proteins Fails to Protect Cells from the Cytopathic Effects of Sendai Virus Infection.Antagonism of the interferon-induced OAS-RNase L pathway by murine coronavirus ns2 protein is required for virus replication and liver pathology.Concomitant type I IFN receptor-triggering of T cells and of DC is required to promote maximal modified vaccinia virus Ankara-induced T-cell expansion.
P2860
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P2860
Sendai virus infection induces efficient adaptive immunity independently of type I interferons
description
2006 nî lūn-bûn
@nan
2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
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name
Sendai virus infection induces ...... endently of type I interferons
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Sendai virus infection induces ...... endently of type I interferons
@en
Sendai virus infection induces ...... endently of type I interferons
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type
label
Sendai virus infection induces ...... endently of type I interferons
@ast
Sendai virus infection induces ...... endently of type I interferons
@en
Sendai virus infection induces ...... endently of type I interferons
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prefLabel
Sendai virus infection induces ...... endently of type I interferons
@ast
Sendai virus infection induces ...... endently of type I interferons
@en
Sendai virus infection induces ...... endently of type I interferons
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P2093
P2860
P1433
P1476
Sendai virus infection induces ...... endently of type I interferons
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P2093
Jacob S Yount
Tamar Hermesh
Thomas M Moran
P2860
P304
P356
10.1128/JVI.80.9.4538-4545.2006
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P577
2006-05-01T00:00:00Z