Interacting regions in Stat3 and c-Jun that participate in cooperative transcriptional activation
about
The coiled-coil domain of Stat3 is essential for its SH2 domain-mediated receptor binding and subsequent activation induced by epidermal growth factor and interleukin-6STAT-1 and c-Fos interaction in nitric oxide synthase-2 gene activationTranscriptional synergy mediated by SAF-1 and AP-1: critical role of N-terminal polyalanine and two zinc finger domains of SAF-1Cross-talk between KLF4 and STAT3 regulates axon regenerationPrinciples of interleukin (IL)-6-type cytokine signalling and its regulationSignal transducers and activators of transcription 3 (STAT3) inhibits transcription of the inducible nitric oxide synthase gene by interacting with nuclear factor kappaBGRIM-19, a death-regulatory gene product, suppresses Stat3 activity via functional interactionSTAT3-dependent enhanceosome assembly and disassembly: synergy with GR for full transcriptional increase of the alpha 2-macroglobulin geneReciprocal inhibition between MyoD and STAT3 in the regulation of growth and differentiation of myoblastsEnhanced antiviral and antiproliferative properties of a STAT1 mutant unable to interact with the protein kinase PKRSTAT3 nuclear import is independent of tyrosine phosphorylation and mediated by importin-alpha3.Structure, function, and regulation of STAT proteinsA novel nuclear zinc finger protein EZI enhances nuclear retention and transactivation of STAT3BATF-JUN is critical for IRF4-mediated transcription in T cellsSTAT4 serine phosphorylation is critical for IL-12-induced IFN-gamma production but not for cell proliferationStat3 isoforms, alpha and beta, demonstrate distinct intracellular dynamics with prolonged nuclear retention of Stat3beta mapping to its unique C-terminal end.Identification of interacting transcription factors regulating tissue gene expression in humanSTAT1β is not dominant negative and is capable of contributing to gamma interferon-dependent innate immunity.Progestins induce transcriptional activation of signal transducer and activator of transcription 3 (Stat3) via a Jak- and Src-dependent mechanism in breast cancer cellsNegative Regulation of Hepatic Inflammation by the Soluble Resistance-Related Calcium-Binding Protein via Signal Transducer and Activator of Transcription 3.The role of STATs in transcriptional control and their impact on cellular function.Jak-Stat signal transduction pathway through the eyes of cytokine class II receptor complexes.Modulation of STAT signaling by STAT-interacting proteins.Protein kinase C epsilon regulation of translocator protein (18 kDa) Tspo gene expression is mediated through a MAPK pathway targeting STAT3 and c-Jun transcription factors.Increased DNA binding activity of NF-kappaB, STAT-3, SMAD3 and AP-1 in acutely damaged liverStat3 directly controls the expression of Tbx5, Nkx2.5, and GATA4 and is essential for cardiomyocyte differentiation of P19CL6 cells.Characterization of a dominant-active STAT that promotes tumorigenesis in Drosophila.Activation of STAT proteins and growth control.STAT3 activation by type I interferons is dependent on specific tyrosines located in the cytoplasmic domain of interferon receptor chain 2c. Activation of multiple STATS proceeds through the redundant usage of two tyrosine residues.Ghrelin induces leptin resistance by activation of suppressor of cytokine signaling 3 expression in male rats: implications in satiety regulationComplex interactions of transcription factors in mediating cytokine biology in T cells.STAT proteins: novel molecular targets for cancer drug discovery.A signal transducer and activator of transcription 3·Nuclear Factor κB (Stat3·NFκB) complex is necessary for the expression of fascin in metastatic breast cancer cells in response to interleukin (IL)-6 and tumor necrosis factor (TNF)-αSynergistic interaction between leptin and cholecystokinin in the rat nodose ganglia is mediated by PI3K and STAT3 signaling pathways: implications for leptin as a regulator of short term satiety.MUC1-C oncoprotein promotes STAT3 activation in an autoinductive regulatory loop.Cell-type and transcription factor specific enrichment of transcriptional cofactor motifs in ENCODE ChIP-seq dataRoles and regulation of stat family transcription factors in human breast cancer.Breast cancer cells condition lymphatic endothelial cells within pre-metastatic niches to promote metastasisVagal control of satiety and hormonal regulation of appetiteSignal transducers and activators of transcription 3 (STAT3) directly regulates cytokine-induced fascin expression and is required for breast cancer cell migration.
P2860
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P2860
Interacting regions in Stat3 and c-Jun that participate in cooperative transcriptional activation
description
1999 nî lūn-bûn
@nan
1999 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Interacting regions in Stat3 a ...... ive transcriptional activation
@ast
Interacting regions in Stat3 a ...... ive transcriptional activation
@en
Interacting regions in Stat3 a ...... ive transcriptional activation
@nl
type
label
Interacting regions in Stat3 a ...... ive transcriptional activation
@ast
Interacting regions in Stat3 a ...... ive transcriptional activation
@en
Interacting regions in Stat3 a ...... ive transcriptional activation
@nl
prefLabel
Interacting regions in Stat3 a ...... ive transcriptional activation
@ast
Interacting regions in Stat3 a ...... ive transcriptional activation
@en
Interacting regions in Stat3 a ...... ive transcriptional activation
@nl
P2093
P2860
P3181
P356
P1476
Interacting regions in Stat3 a ...... ive transcriptional activation
@en
P2093
C M Horvath
J E Darnell
M H Wrzeszczynska
P2860
P304
P3181
P356
10.1128/MCB.19.10.7138
P407
P577
1999-10-01T00:00:00Z