Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement - Test2 - elhamkhani - TriplyDB

Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement

Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement

http://www.wikidata.org/entity/Q24594632

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Nanoparticle-encapsulated chlorhexidine against oral bacterial biofilms The human microbiome: our second genome Porphyromonas gingivalis as a potential community activist for disease Periodontal Disease-Induced Atherosclerosis and Oxidative Stress Complement Involvement in Periodontitis: Molecular Mechanisms and Rational Therapeutic Approaches Periodontal Diseases: Bug Induced, Host Promoted Historical and contemporary hypotheses on the development of oral diseases: are we there yet?Innocent until proven guilty: mechanisms and roles of Streptococcus-Candida interactions in oral health and disease The bacteriome-mycobiome interaction and antifungal host defense Diabetes mellitus related bone metabolism and periodontal disease The oral microbiome and the immunobiology of periodontal disease and caries Complement and dysbiosis in periodontal disease Filifactor alocis: The Newly Discovered Kid on the Block with Special Talents Periodontitis: from microbial immune subversion to systemic inflammation Porphyromonas gingivalis: an invasive and evasive opportunistic oral pathogen Aggressive Periodontitis: microbes and host response, who to blame?The Role of Oral Pathobionts in Dysbiosis during Periodontitis Development The biogeography of polymicrobial infection Neutrophil homeostasis and inflammation: novel paradigms from studying periodontitis The enduring importance of animal models in understanding periodontal disease Extract from Rumex acetosa L. for prophylaxis of periodontitis: inhibition of bacterial in vitro adhesion and of gingipains of Porphyromonas gingivalis by epicatechin-3-O-(4β→8)-epicatechin-3-O-gallate (procyanidin-B2-Di-gallate)Identification of Small-Molecule Inhibitors against Meso-2, 6-Diaminopimelate Dehydrogenase from Porphyromonas gingivalis Immunomicrobial pathogenesis of periodontitis: keystones, pathobionts, and host response The Microbiome and Complement Activation: A Mechanistic Model for Preterm Birth.The link between periodontal disease and rheumatoid arthritis: an updated review.Periodontal dysbiosis linked to periodontitis is associated with cardiometabolic adaptation to high-fat diet in mice.Doppler flowmetry as a tool of predictive, preventive and personalised dentistry.The leukocyte integrin antagonist Del-1 inhibits IL-17-mediated inflammatory bone loss LL-37 opsonizes and inhibits biofilm formation of Aggregatibacter actinomycetemcomitans at subbactericidal concentrations.Genetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis.Defective neutrophil recruitment in leukocyte adhesion deficiency type I disease causes local IL-17-driven inflammatory bone loss.Biodiversity and functional genomics in the human microbiome.Integrated metagenomic data analysis demonstrates that a loss of diversity in oral microbiota is associated with periodontitis.Disruption of heterotypic community development by Porphyromonas gingivalis with small molecule inhibitors.Bacterial invasion and persistence: critical events in the pathogenesis of periodontitis?Oral administration of Lactobacillus gasseri SBT2055 is effective in preventing Porphyromonas gingivalis-accelerated periodontal disease.Promotion of endodontic lesions in rats by a novel extraradicular biofilm model using obturation materials.Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis.Periodontal infection, impaired fasting glucose and impaired glucose tolerance: results from the Continuous National Health and Nutrition Examination Survey 2009-2010.Lung Microbiota and Its Impact on the Mucosal Immune Phenotype.

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P2860

Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement

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description

2011 nî lūn-bûn

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2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Low-abundance biofilm species ...... nsal microbiota and complement

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Low-abundance biofilm species ...... nsal microbiota and complement

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Low-abundance biofilm species ...... nsal microbiota and complement

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J.CHOM.2011.10.006

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Cell Host & Microbe

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Low-abundance biofilm species ...... nsal microbiota and complement

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Ahmed Hashim

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Asil Alsam

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Keith L Kirkwood

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Mark A Payne

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Megan L McIntosh

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Mehmet A Eskan

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Ravi Jotwani

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Richard P Darveau

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Shuang Liang

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P2860

Determination of the genome sequence of Porphyromonas gingivalis strain ATCC 33277 and genomic comparison with strain W83 revealed extensive genome rearrangements in P. gingivalis

The C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone loss

The skin microbiome

Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?

Salmonella enterica serovar typhimurium exploits inflammation to compete with the intestinal microbiota

Relative contribution of LFA-1 and Mac-1 to neutrophil adhesion and migration

Complement: a key system for immune surveillance and homeostasis

Microbial manipulation of receptor crosstalk in innate immunity

Complement regulators and inhibitory proteins.

Genetic control of susceptibility to Porphyromonas gingivalis-induced alveolar bone loss in mice

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497-506

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scholarly article

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441242

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10.1016/J.CHOM.2011.10.006

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John D. Lambris

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2011-10-27T00:00:00Z

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51755824

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22036469

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