Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
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Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complementThe C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone lossNucleoside-diphosphate-kinase: a pleiotropic effector in microbial colonization under interdisciplinary characterizationPorphyromonas gingivalis: an invasive and evasive opportunistic oral pathogenThe lysine gingipain adhesin domains from Porphyromonas gingivalis interact with erythrocytes and albumin: Structures correlate to functionMacromolecular networks and intelligence in microorganismsRole of OmpA2 surface regions of Porphyromonas gingivalis in host-pathogen interactions with oral epithelial cells.Microbial manipulation of receptor crosstalk in innate immunityPeriodontal inflammation and bone loss in aged mice.Dichotomy of gingipains action as virulence factors: from cleaving substrates with the precision of a surgeon's knife to a meat chopper-like brutal degradation of proteins.Role of Porphyromonas gingivalis phosphoserine phosphatase enzyme SerB in inflammation, immune response, and induction of alveolar bone resorption in ratsTranscriptional profiling of bone marrow stromal cells in response to Porphyromonas gingivalis secreted productsThe lipid A phosphate position determines differential host Toll-like receptor 4 responses to phylogenetically related symbiotic and pathogenic bacteria.The expression and regulation of matrix metalloproteinase-3 is critically modulated by Porphyromonas gingivalis lipopolysaccharide with heterogeneous lipid A structures in human gingival fibroblasts.α-Amylase is a potential growth inhibitor of Porphyromonas gingivalis, a periodontal pathogenic bacterium.Tetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblastsRole of complement in host-microbe homeostasis of the periodontium.Porphyromonas gingivalis regulates TREM-1 in human polymorphonuclear neutrophils via its gingipains.Interleukin-8 responses of multi-layer gingival epithelia to subgingival biofilms: role of the "red complex" speciesDistinct gene signatures in aortic tissue from ApoE-/- mice exposed to pathogens or Western diet.Polymicrobial periodontal pathogen transcriptomes in calvarial bone and soft tissue.Sialidase and sialoglycoproteases can modulate virulence in Porphyromonas gingivalis.Pathogenic microbes and community service through manipulation of innate immunityPorphyromonas gingivalis: a clonal pathogen?: Diversities in housekeeping genes and the major fimbriae gene.Immune evasion strategies of Porphyromonas gingivalis.Heritability of periodontal bone loss in mice.Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal diseasePorphyromonas gingivalis promotes Th17 inducing pathways in chronic periodontitis.Complement-targeted therapeutics in periodontitisInactivation of epidermal growth factor by Porphyromonas gingivalis as a potential mechanism for periodontal tissue damage.Insights into the virulence of oral biofilms: discoveries from proteomics.Gingipains: Critical Factors in the Development of Aspiration Pneumonia Caused by Porphyromonas gingivalis.Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.The GroEL protein of Porphyromonas gingivalis regulates atherogenic phenomena in endothelial cells mediated by upregulating toll-like receptor 4 expression.Noncanonical dendritic cell differentiation and survival driven by a bacteremic pathogen.Genetic diversity in the oral pathogen Porphyromonas gingivalis: molecular mechanisms and biological consequences.Age-related alterations in innate immune receptor expression and ability of macrophages to respond to pathogen challenge in vitro.Zebrafish as a new model to study effects of periodontal pathogens on cardiovascular diseases.Complementary Tolls in the periodontium: how periodontal bacteria modify complement and Toll-like receptor responses to prevail in the host.Blood dendritic cells: "canary in the coal mine" to predict chronic inflammatory disease?
P2860
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P2860
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
description
2009 nî lūn-bûn
@nan
2009 թուականին հրատարակուած գիտական յօդուած
@hyw
2009 թվականին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@ast
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@en
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@nl
type
label
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@ast
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@en
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@nl
prefLabel
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@ast
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@en
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@nl
P2860
P3181
P1476
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?
@en
P2093
George Hajishengallis
P2860
P304
P3181
P356
10.1016/J.MICINF.2009.03.009
P407
P577
2009-01-01T00:00:00Z