Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice.
about
Metabolism leaves its mark on the powerhouse: recent progress in post-translational modifications of lysine in mitochondriaCa2+ toxicity and mitochondrial damage in acute pancreatitis: translational overviewCell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthaseCalcium signaling in cardiac mitochondriaFrom ATP to PTP and Back: A Dual Function for the Mitochondrial ATP SynthasePostconditioning signalling in the heart: mechanisms and translatabilityCaMKIIdelta subtypes: localization and functionMelatonin: A Mitochondrial Targeting Molecule Involving Mitochondrial Protection and DynamicsThe mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotectionCaMKII induces permeability transition through Drp1 phosphorylation during chronic β-AR stimulationBax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in miceRedox regulation of mitochondrial functionMitochondrial Metabolism in Aging HeartCell biology. Metabolic control of cell deathThe Novel Extracellular Cyclophilin A (CyPA) - Inhibitor MM284 Reduces Myocardial Inflammation and Remodeling in a Mouse Model of Troponin I -Induced MyocarditisMitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes.Regulation of metabolism in individual mitochondria during excitation-contraction couplingProtein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).Radiation-induced alterations in mitochondria of the rat heart.High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities.The mitochondrial permeability transition pore is a dispensable element for mitochondrial calcium efflux.Deregulation of mitochondrial F1FO-ATP synthase via OSCP in Alzheimer's disease.Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Regulation of mitochondrial processes by protein S-nitrosylation.Monoamine oxidase inhibition prevents mitochondrial dysfunction and apoptosis in myoblasts from patients with collagen VI myopathies.Cyclophilin D modulates mitochondrial acetylomeA new twist on an old idea part 2: cyclosporine preserves normal mitochondrial but not cardiomyocyte function in mini-swine with compensated heart failureMitochondrial calcium overload is a key determinant in heart failure.Regulation of the mPTP by SIRT3-mediated deacetylation of CypD at lysine 166 suppresses age-related cardiac hypertrophy.Estrogen regulates energy metabolic pathway and upstream adenosine 5'-monophosphate-activated protein kinase and phosphatase enzyme expression in dorsal vagal complex metabolosensory neurons during glucostasis and hypoglycemia.Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes.F-ATPase of Drosophila melanogaster forms 53-picosiemen (53-pS) channels responsible for mitochondrial Ca2+-induced Ca2+ release.Bioengineering and semisynthesis of an optimized cyclophilin inhibitor for treatment of chronic viral infection.Mitochondrial reprogramming induced by CaMKIIδ mediates hypertrophy decompensationDocosahexaenoic acid supplementation alters key properties of cardiac mitochondria and modestly attenuates development of left ventricular dysfunction in pressure overload-induced heart failureGlycogen synthase kinase-3α limits ischemic injury, cardiac rupture, post-myocardial infarction remodeling and death.The permeability transition pore controls cardiac mitochondrial maturation and myocyte differentiation.Calcium and reactive oxygen species in acute pancreatitis: friend or foe?Protective role of transient pore openings in calcium handling by cardiac mitochondria.
P2860
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P2860
Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Cyclophilin D controls mitocho ...... sity for heart failure in mice
@nl
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@ast
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@en
type
label
Cyclophilin D controls mitocho ...... sity for heart failure in mice
@nl
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@ast
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@en
prefLabel
Cyclophilin D controls mitocho ...... sity for heart failure in mice
@nl
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@ast
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@en
P2093
P2860
P50
P3181
P356
P1476
Cyclophilin D controls mitocho ...... ity for heart failure in mice.
@en
P2093
Bhuvana Sakthievel
Elizabeth Murphy
Jason Karch
Joan Heller Brown
John Farber
Renee Wong
Ronald J Vagnozzi
Sanjeewa A Goonasekera
Scott Gabel
Shikha Mishra
P2860
P304
P3181
P356
10.1172/JCI43171
P407
P577
2010-09-20T00:00:00Z