The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotection
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The Role of Mitochondrial Reactive Oxygen Species in Cardiovascular Injury and Protective StrategiesCell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthaseFrom ATP to PTP and Back: A Dual Function for the Mitochondrial ATP SynthaseThe EF-Hand Ca2+ Binding Protein MICU Choreographs Mitochondrial Ca2+ Dynamics in ArabidopsisIschaemic conditioning and targeting reperfusion injury: a 30 year voyage of discoveryCurrent understanding of molecular pathology and treatment of cardiomyopathy in duchenne muscular dystrophyGenetic evidence in the mouse solidifies the calcium hypothesis of myofiber death in muscular dystrophyThe valosin-containing protein is a novel mediator of mitochondrial respiration and cell survival in the heart in vivo.Mitochondria and ageing: role in heart, skeletal muscle and adipose tissue.F-ATPase of Drosophila melanogaster forms 53-picosiemen (53-pS) channels responsible for mitochondrial Ca2+-induced Ca2+ release.Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate.Reactive oxygen species trigger motoneuron death in non-cell-autonomous models of ALS through activation of c-Abl signalingVoltage-Dependent Regulation of Complex II Energized Mitochondrial Oxygen Flux.Real-Time Fluorescence Measurements of ROS and [Ca2+] in Ischemic / Reperfused Rat Hearts: Detectable Increases Occur only after Mitochondrial Pore Opening and Are Attenuated by Ischemic Preconditioning.Reperfusion injury and reactive oxygen species: The evolution of a concept.Increased Susceptibility of Gracilinanus microtarsus Liver Mitochondria to Ca²⁺-Induced Permeability Transition Is Associated with a More Oxidized State of NAD(P)HIV alters neuronal mitochondrial fission/fusion in the brain during HIV-associated neurocognitive disorders.SIRT3 Deacetylates Ceramide Synthases: IMPLICATIONS FOR MITOCHONDRIAL DYSFUNCTION AND BRAIN INJURY.Individual Cardiac Mitochondria Undergo Rare Transient Permeability Transition Pore Openings.Bacterial-excreted small volatile molecule 2-aminoacetophenone induces oxidative stress and apoptosis in murine skeletal muscle.Knockout of Cyclophilin-D Provides Partial Amelioration of Intrinsic and Synaptic Properties Altered by Mild Traumatic Brain Injury.Rhubarb Antagonizes Matrix Metalloproteinase-9-induced Vascular Endothelial PermeabilityThe methyl donor S-adenosylmethionine prevents liver hypoxia and dysregulation of mitochondrial bioenergetic function in a rat model of alcohol-induced fatty liver disease.Dynamic Monitoring of p53 Translocation to Mitochondria for the Analysis of Specific Inhibitors Using Luciferase-fragment Complementation.The c-Ring of the F1FO-ATP Synthase: Facts and Perspectives.Crosstalk of mitochondria with NADPH oxidase via reactive oxygen and nitrogen species signalling and its role for vascular function.Targeting myocardial reperfusion injuries with cyclosporine in the CIRCUS Trial - pharmacological reasons for failure.Mitochondrial redox status as a target for cardiovascular disease.Cardioprotection by H2S Donors: Nitric Oxide-Dependent and ‑Independent Mechanisms.New aspects of p66Shc in ischaemia reperfusion injury and other cardiovascular diseases.The in vivo evidence for regulated necrosis.PRKCE gene encoding protein kinase C-epsilon-Dual roles at sarcomeres and mitochondria in cardiomyocytes.The pathophysiology of acute myocardial infarction and strategies of protection beyond reperfusion: a continual challenge.Electromagnetic Fields and Stem Cell Fate: When Physics Meets Biology.Mitochondrial permeability transition in cardiac ischemia-reperfusion: whether cyclophilin D is a viable target for cardioprotection?Deduction of a calcium ion circuit affecting rooster sperm in vitro.The role of succinate and ROS in reperfusion injury - A critical appraisal.Quantification of active mitochondrial permeability transition pores using GNX-4975 inhibitor titrations provides insights into molecular identity.Sex difference in the sensitivity of cardiac mitochondrial permeability transition pore to calcium load.DS16570511 is a small-molecule inhibitor of the mitochondrial calcium uniporter.
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P2860
The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotection
description
2015 nî lūn-bûn
@nan
2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
The mitochondrial permeability ...... s a target in cardioprotection
@ast
The mitochondrial permeability ...... s a target in cardioprotection
@en
The mitochondrial permeability ...... s a target in cardioprotection
@nl
type
label
The mitochondrial permeability ...... s a target in cardioprotection
@ast
The mitochondrial permeability ...... s a target in cardioprotection
@en
The mitochondrial permeability ...... s a target in cardioprotection
@nl
prefLabel
The mitochondrial permeability ...... s a target in cardioprotection
@ast
The mitochondrial permeability ...... s a target in cardioprotection
@en
The mitochondrial permeability ...... s a target in cardioprotection
@nl
P2860
P921
P3181
P1476
The mitochondrial permeability ...... s a target in cardioprotection
@en
P2860
P3181
P356
10.1016/J.YJMCC.2014.09.023
P407
P577
2015-01-01T00:00:00Z