Abnormal behavior in a chromosome-engineered mouse model for human 15q11-13 duplication seen in autism - Test2 - elhamkhani - TriplyDB

Abnormal behavior in a chromosome-engineered mouse model for human 15q11-13 duplication seen in autism

Abnormal behavior in a chromosome-engineered mouse model for human 15q11-13 duplication seen in autism

http://www.wikidata.org/entity/Q24599984

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Cross fostering experiments suggest that mice songs are innate.Long-lasting alterations to DNA methylation and ncRNAs could underlie the effects of fetal alcohol exposure in mice Topoisomerase inhibitors unsilence the dormant allele of Ube3a in neurons The snoRNA MBII-52 (SNORD 115) is processed into smaller RNAs and regulates alternative splicing Modifying behavioral phenotypes in Fmr1KO mice: genetic background differences reveal autistic-like responses.Striatal Circuits as a Common Node for Autism Pathophysiology Autism Spectrum Disorders and Drug Addiction: Common Pathways, Common Molecules, Distinct Disorders?The role of cerebellar circuitry alterations in the pathophysiology of autism spectrum disorders Prader-Willi, Angelman, and 15q11-q13 Duplication Syndromes Following the genes: a framework for animal modeling of psychiatric disorders CNVs: harbingers of a rare variant revolution in psychiatric genetics Behavioral and Neuroanatomical Phenotypes in Mouse Models of Autism Mice with Dab1 or Vldlr insufficiency exhibit abnormal neonatal vocalization patterns Structure and function of neonatal social communication in a genetic mouse model of autism Altered ultrasonic vocalization and impaired learning and memory in Angelman syndrome mouse model with a large maternal deletion from Ube3a to Gabrb3.PX-RICS-deficient mice mimic autism spectrum disorder in Jacobsen syndrome through impaired GABAA receptor trafficking Negative allosteric modulation of the mGluR5 receptor reduces repetitive behaviors and rescues social deficits in mouse models of autism.Single-neuron and genetic correlates of autistic behavior in macaque.Genetic aspects of autism spectrum disorders: insights from animal models Advancing the understanding of autism disease mechanisms through genetics Histopathologic characterization of the BTBR mouse model of autistic-like behavior reveals selective changes in neurodevelopmental proteins and adult hippocampal neurogenesis Mice genetically depleted of brain serotonin display social impairments, communication deficits and repetitive behaviors: possible relevance to autism The serotonin system in autism spectrum disorder: From biomarker to animal models Using mouse models of autism spectrum disorders to study the neurotoxicology of gene-environment interactions Translational Mouse Models of Autism: Advancing Toward Pharmacological Therapeutics.The disruption of Celf6, a gene identified by translational profiling of serotonergic neurons, results in autism-related behaviors.A novel protein, CHRONO, functions as a core component of the mammalian circadian clock Communication impairments in mice lacking Shank1: reduced levels of ultrasonic vocalizations and scent marking behavior.Sociability and motor functions in Shank1 mutant mice Social communication in mice--are there optimal cage conditions?JAKMIP1, a Novel Regulator of Neuronal Translation, Modulates Synaptic Function and Autistic-like Behaviors in Mouse.Absence of CNTNAP2 leads to epilepsy, neuronal migration abnormalities, and core autism-related deficits.Behavioural phenotyping assays for mouse models of autism Synaptic proteins and receptors defects in autism spectrum disorders Unraveling the genetic architecture of copy number variants associated with schizophrenia and other neuropsychiatric disorders.Paternal Aging Affects Behavior in Pax6 Mutant Mice: A Gene/Environment Interaction in Understanding Neurodevelopmental Disorders Epigenetic regulation of UBE3A and roles in human neurodevelopmental disorders.Automated three-chambered social approach task for mice Comprehensive behavioral phenotyping of a new Semaphorin 3 F mutant mouse An urgent need for experimental animal model of autism in drug development.

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Abnormal behavior in a chromosome-engineered mouse model for human 15q11-13 duplication seen in autism

http://www.wikidata.org/entity/Q24599984

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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J.CELL.2009.04.024

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Abnormal behavior in a chromos ...... -13 duplication seen in autism

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Allan Bradley

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Fumiyuki Hatanaka

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Hisashi Ohta

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Jin Nakatani

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Kazuya Iwamoto

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Kenta Yamauchi

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Kiyoshi Inoue

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Koichi Tanda

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Kota Tamada

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Makoto Okazawa

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P2860

Advances in autism genetics: on the threshold of a new neurobiology

Autism: highly heritable but not inherited

Copy-number variations associated with neuropsychiatric conditions

Genetics of austim: complex aetiology for a heterogeneous disorder

Searching for ways out of the autism maze: genetic, epigenetic and environmental clues

Autism: many genes, common pathways?

Reduced social interaction and ultrasonic communication in a mouse model of monogenic heritable autism.

A neuroligin-3 mutation implicated in autism increases inhibitory synaptic transmission in mice

Strong association of de novo copy number mutations with autism

Autism or atypical autism in maternally but not paternally derived proximal 15q duplication

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10.1016/J.CELL.2009.04.024

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Tsuyoshi Miyakawa

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26327793

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