PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia - Test2 - elhamkhani - TriplyDB

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

http://www.wikidata.org/entity/Q24604133

about

Mitochondrial Parkin recruitment is impaired in neurons derived from mutant PINK1 induced pluripotent stem cells The rise of mitochondria in medicine Mitochondrial Dysfunction in Alzheimer's Disease and the Rationale for Bioenergetics Based Therapies The association between PGC-1α and Alzheimer's disease Current Research Therapeutic Strategies for Alzheimer's Disease Treatment The Therapeutic Potential of the Ketogenic Diet in Treating Progressive Multiple Sclerosis Parkinson's disease, insulin resistance and novel agents of neuroprotection Mitochondrial abnormalities in Alzheimer's disease: possible targets for therapeutic intervention Calcium channel blockers and Alzheimer's disease New insights into PGC-1 coactivators: redefining their role in the regulation of mitochondrial function and beyond After the banquet: mitochondrial biogenesis, mitophagy, and cell survival The Role of the Transcription Factor Foxo3 in Hearing Maintenance: Informed Speculation on a New Player in the Cochlea Estrogenic Endocrine Disrupting Chemicals Influencing NRF1 Regulated Gene Networks in the Development of Complex Human Brain Diseases Mitochondrial dysfunction in neurodegenerative diseases Energy failure: does it contribute to neurodegeneration?PGC-1α negatively regulates extrasynaptic NMDAR activity and excitotoxicity A computational procedure for functional characterization of potential marker genes from molecular data: Alzheimer's as a case study spargel, the PGC-1α homologue, in models of Parkinson disease in Drosophila melanogaster.PGC-1α overexpression exacerbates β-amyloid and tau deposition in a transgenic mouse model of Alzheimer's disease.Cerebellar transcriptional alterations with Purkinje cell dysfunction and loss in mice lacking PGC-1α.Mitochondria in Alzheimer's Disease and Diabetes-Associated Neurodegeneration: License to Heal!Metabolic stress modulates Alzheimer's β-secretase gene transcription via SIRT1-PPARγ-PGC-1 in neurons.Mice lacking the transcriptional coactivator PGC-1α exhibit alterations in inhibitory synaptic transmission in the motor cortex.Autism-Like Behaviours and Memory Deficits Result from a Western Diet in Mice.Involvement of PGC-1α in the formation and maintenance of neuronal dendritic spines Parvalbumin deficiency and GABAergic dysfunction in mice lacking PGC-1alpha PGC-1α provides a transcriptional framework for synchronous neurotransmitter release from parvalbumin-positive interneurons.Toxicity of neurons treated with herbicides and neuroprotection by mitochondria-targeted antioxidant SS31.Differential regulation of BACE1 expression by oxidative and nitrosative signals Peroxisome proliferator-activated receptor (PPAR) γ and PPARα agonists modulate mitochondrial fusion-fission dynamics: relevance to reactive oxygen species (ROS)-related neurodegenerative disorders?Neuroprotective strategies involving ROS in Alzheimer disease.Mitochondria-targeted antioxidants protect against amyloid-beta toxicity in Alzheimer's disease neurons.Functional genomics of brain aging and Alzheimer's disease: focus on selective neuronal vulnerability.Mitochondria and antioxidant targeted therapeutic strategies for Alzheimer's disease.Upregulation of PGC-1α expression by Alzheimer's disease-associated pathway: presenilin 1/amyloid precursor protein (APP)/intracellular domain of APP Peroxisome proliferator activator receptor gamma coactivator-1alpha (PGC-1α) improves motor performance and survival in a mouse model of amyotrophic lateral sclerosis Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer's disease-relevant murine model.Ethanol suppresses PGC-1α expression by interfering with the cAMP-CREB pathway in neuronal cells.PGC-1α activity in nigral dopamine neurons determines vulnerability to α-synuclein Impaired mitochondrial biogenesis contributes to mitochondrial dysfunction in Alzheimer's disease.

P2860

Q24300095-3ED855E2-BAE2-4ED1-8260-2169B0249DF5 Q26740014-4E63190D-52E9-4C7F-911F-7F0EAC04AAFD Q26750081-5B34A733-8F7C-44B7-B704-1F0864921102 Q26751165-6BA0FEE1-3C18-45F8-BC5B-AA945C9D077F Q26765530-33A42404-283A-45D3-B79F-3FA797C525A6 Q26769966-1B8A302B-CCE8-4E42-8545-DE40EFF0F6B3 Q26853311-A5B1D7E1-4B8E-436B-8F79-B32AA6332CFE Q26865358-18B4512F-1430-4210-B4EB-F59B19915C28 Q26866552-AE03752F-FE06-4C98-B737-11382DB152F4 Q26996576-FBA44C05-53F2-413A-A388-4B16F57381EE Q27026657-242B2389-F84C-4357-B041-7DF108854404 Q28073272-CAFBEF02-6F2D-4B79-81CC-A5B017048248 Q28079898-7C36EB6D-F8D1-4E43-B351-7586825A26F9 Q28387647-D9CB302B-0EC4-4D75-A01A-15BAB2A96717 Q28389950-23C2E627-0362-4A4E-92B9-1EB045711312 Q28575407-EE458E7E-1CB1-465E-96B4-37C4F9934123 Q28741856-E2E4F702-1E82-4EAC-9976-4D628E91BE9B Q30370305-36B706D8-CF90-4B39-9B6F-D09AB881EFBB Q30574513-77164C7F-76B5-49B0-A2AB-28D007079E91 Q30613057-D44BA372-6D05-447C-A636-E072C278EA21 Q33364882-7BEDB90F-E20B-42A0-80B1-9EDA888893DF Q33579072-2D335647-FBF9-4456-A990-DD9CD8264D2D Q33799027-99EE72CE-BA06-4EF4-BF30-D0CDDA03BBD8 Q33823142-E6F36B12-B9FC-41D2-A4C2-CA530CAD4F0E Q33880898-4A10D3F5-472E-4795-8909-7849ABEFC776 Q33922914-E600E774-D609-4FEC-BCE7-0E02676A481B Q34383492-0669FE6F-B2B3-490C-B28C-BCEDAC80A5F9 Q34566188-41EA2630-A4D9-4B63-925F-C96D66BC0D3E Q34686983-C9D95EBE-48C0-473F-ADAF-0360EDBBBF12 Q34723198-167286AC-4985-4A88-A783-3B54352D24A9 Q34757939-0F3C1F63-F10E-49F8-84E4-6311491B4B40 Q34772619-E0E969EC-BE9F-48A6-9CD3-54D18820A155 Q34807482-82924FD2-4F25-4322-9C34-435424ED8CEB Q34994270-442CA63C-4749-4D13-A1BF-85E2591E603D Q35093148-5C4BFBC0-DD8F-45B9-93F5-9F86AB18CE21 Q35166545-4D67FAA7-DE94-4005-87D3-107A20F32DB3 Q35174844-68075E29-EADB-49CB-BE48-3AFAA0E6C296 Q35220528-A2BC59B4-0E6A-4575-A90B-14B5C6BAA219 Q35233106-5BAB07CE-19A1-41E7-AEA8-A0580F2F8D62 Q35660223-5005C380-3A3D-4888-869E-1379E7D08E7E

P2860

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

http://www.wikidata.org/entity/Q24604133

description

2009 nî lūn-bûn

@nan

2009 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի մարտին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@ast

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@en

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@nl

type

label

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@ast

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@en

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@nl

prefLabel

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@ast

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@en

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@nl

P1433

Q24604133-F1DBA815-7F20-462E-9A92-6F1864FE2BE5

P1476

Q24604133-CB324A9E-5E0B-4275-B0F8-D68A9711C81F

P2093

Q24604133-04D7449F-6905-400D-AF4B-ECBE397F1C96

Q24604133-55D8004D-40C3-43D7-AACD-8C9BC06320A7

Q24604133-636115D1-7A04-45BD-84F6-0762C643BA31

P2860

Q24604133-0965BCBB-95F4-4D3D-9733-BF9C7BB6CD26

Q24604133-0C2EF63B-55FD-4FCB-A9A5-BF5345D7EC74

Q24604133-0E0E9808-DD30-4AA3-9C4D-B4B7CB1BF2BD

Q24604133-114AED1F-6329-43BE-A3BF-BDC5844EC673

Q24604133-195D9C56-FD81-4ADF-8FC8-7207814D06BD

Q24604133-2A72AF63-AC76-47A3-82FE-13B1DB29B17F

Q24604133-3207BCE3-B04B-45A2-ABEF-BB6D8D82810D

Q24604133-332820BB-3E55-4F6A-BADE-12A9B2FDA8ED

Q24604133-3BCEFAB1-4140-4281-A570-E8B6642075CB

Q24604133-3C0A55BC-1EE9-42BF-B305-6090FFED8F30

P304

Q24604133-6A2188F5-17F1-41A3-9E76-CB8ACCAFB467

P31

Q24604133-568AEA41-DBF2-4BFB-A1BF-63B02B651738

P3181

Q24604133-2637F58C-EB47-4598-BBA5-4A44163A120A

P356

Q24604133-A6B46BA1-8D12-4682-805A-EEC5D43BC3F8

P407

Q24604133-F6029515-D18C-4BE9-B9A0-DC83344409F6

P433

Q24604133-DE6810BA-F1CD-46C4-B10C-12C4234B7B01

P478

Q24604133-21C08804-F263-43DF-B9B5-2C7C1C529A18

P50

Q24604133-064EB993-780F-4929-8818-2E069E034392

Q24604133-70E43A46-B8EE-43C8-9189-2B5AB22B21EE

Q24604133-BB32E15B-DC84-40AB-924A-4AEB0B79536F

Q24604133-F1C2B2DF-6E3D-4E67-9061-4F2D7106FC5E

P577

Q24604133-FD27D92F-EE65-4EA3-BF3C-8D203ACD32E3

P5875

Q24604133-3A1403C7-96AD-4AFB-BB1B-0698D93D155E

P698

Q24604133-8B838F24-9F18-4806-B8C0-5D10408BFF02

P921

Q24604133-93085B59-BC44-4D79-AE3C-B889E30E278E

Q24604133-D674715C-0025-4D92-BCF2-8A029C55397D

P932

Q24604133-92926E0B-FF77-4D93-B942-D181B424EA78

P3181

P356

ARCHNEUROL.2008.588

P1433

P1476

PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

@en

P2093

Christopher P Cardozo

http://www.w3.org/2001/XMLSchema#string

Pavel Katsel

http://www.w3.org/2001/XMLSchema#string

Weiping Qin

http://www.w3.org/2001/XMLSchema#string

P2860

Peroxisome proliferator-activated receptor-gamma coactivator 1 alpha (PGC-1 alpha): transcriptional coactivator and metabolic regulator

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

A comparison of normalization methods for high density oligonucleotide array data based on variance and bias

Exploration, normalization, and summaries of high density oligonucleotide array probe level data

The Akt-regulated forkhead transcription factor FOXO3a controls endothelial cell viability through modulation of the caspase-8 inhibitor FLIP

Control of hepatic gluconeogenesis through the transcriptional coactivator PGC-1

Nutrient control of glucose homeostasis through a complex of PGC-1alpha and SIRT1

PGC-1alpha: a key regulator of energy metabolism

Resveratrol improves mitochondrial function and protects against metabolic disease by activating SIRT1 and PGC-1alpha

Defects in adaptive energy metabolism with CNS-linked hyperactivity in PGC-1alpha null mice

P304

352-61

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P3181

2125211

http://www.w3.org/2001/XMLSchema#string

P356

10.1001/ARCHNEUROL.2008.588

http://www.w3.org/2001/XMLSchema#string

P407

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

66

http://www.w3.org/2001/XMLSchema#string

P50

Giulio Maria Pasinetti

Joseph D. Buxbaum

Vahram Haroutunian

P577

2009-03-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

24189053

http://www.w3.org/2001/XMLSchema#string

P698

19273754

http://www.w3.org/2001/XMLSchema#string

P921

Alzheimer's disease

P932

3052997

http://www.w3.org/2001/XMLSchema#string