Hypoxia-inducible factors mediate coordinated RhoA-ROCK1 expression and signaling in breast cancer cells - Test2 - elhamkhani - TriplyDB

Hypoxia-inducible factors mediate coordinated RhoA-ROCK1 expression and signaling in breast cancer cells

Hypoxia-inducible factors mediate coordinated RhoA-ROCK1 expression and signaling in breast cancer cells

http://www.wikidata.org/entity/Q24613508

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Sirtuins and Cancer: Role in the Epithelial-Mesenchymal Transition Novel Insights into the Roles of Rho Kinase in Cancer Forms, forces, and stem cell fate Statistical analysis of cell migration in 3D using the anisotropic persistent random walk model.Cellular adhesome screen identifies critical modulators of focal adhesion dynamics, cellular traction forces and cell migration behaviour.Glycolysis is the primary bioenergetic pathway for cell motility and cytoskeletal remodeling in human prostate and breast cancer cells.Effect of QSKL on MAPK and RhoA Pathways in a Rat Model of Heart Failure.Hypoxia-inducible factor-dependent signaling between triple-negative breast cancer cells and mesenchymal stem cells promotes macrophage recruitment.Hypoxia-inducible factors and RAB22A mediate formation of microvesicles that stimulate breast cancer invasion and metastasis.Estrogen-related receptor α decreases RHOA stability to induce orientated cell migration.Hypoxia and the extracellular matrix: drivers of tumour metastasis Hypoxia-inducible factor 1 mediates TAZ expression and nuclear localization to induce the breast cancer stem cell phenotype.Advancements in the development of HIF-1α-activated protein switches for use in enzyme prodrug therapy.Rho-associated kinase 1 inhibition is synthetically lethal with von Hippel-Lindau deficiency in clear cell renal cell carcinoma HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells.Expression and prognostic significance of MYL9 in esophageal squamous cell carcinoma.The hypoxic tumor microenvironment: A driving force for breast cancer progression.CKD Stimulates Muscle Protein Loss Via Rho-associated Protein Kinase 1 Activation.Induction of WNT11 by hypoxia and hypoxia-inducible factor-1α regulates cell proliferation, migration and invasion.Hypoxia promotes Rab5 activation, leading to tumor cell migration, invasion and metastasis.Inhibitors of Rho kinase (ROCK) signaling revert the malignant phenotype of breast cancer cells in 3D context Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.Hypoxia-inducible factors regulate pluripotency factor expression by ZNF217- and ALKBH5-mediated modulation of RNA methylation in breast cancer cells.Genetic variants in RhoA and ROCK1 genes are associated with the development, progression and prognosis of prostate cancer Role of miR-155 in drug resistance of breast cancer.Hypoxia regulates RhoA and Wnt/β-catenin signaling in a context-dependent way to control re-differentiation of chondrocytes.MicroRNA-340 inhibits invasion and metastasis by downregulating ROCK1 in breast cancer cells.Targeting the RhoGTPase/ROCK pathway for the treatment of VHL/HIF pathway-driven cancers.Cell type-dependent HIF1 α-mediated effects of hypoxia on proliferation, migration and metastatic potential of human tumor cells Bi-content micro-collagen chip provides contractility-based biomechanical readout for phenotypic drug screening with expanded and profiled targets.Hypoxia and the modulation of the actin cytoskeleton - emerging interrelations.PLC-β2 is modulated by low oxygen availability in breast tumor cells and plays a phenotype dependent role in their hypoxia-related malignant potential.Hypoxia Selectively Enhances Integrin α5β1 Receptor Expression in Breast Cancer to Promote Metastasis Retinoblastoma protein (Rb) links hypoxia to altered mechanical properties in cancer cells as measured by an optical tweezer.Cell-Extracellular Matrix Mechanobiology: Forceful Tools and Emerging Needs for Basic and Translational Research.Nck deficiency is associated with delayed breast carcinoma progression and reduced metastasis.The CCR2 3'UTR functions as a competing endogenous RNA to inhibit breast cancer metastasis.Directional Migration of MDA-MB-231 Cells Under O2/pH Gradients.Preventative effects of a HIF inhibitor, 17-DMAG, on partial bladder outlet obstruction-induced bladder dysfunction.Diallyl trisulfides, a natural histone deacetylase inhibitor, attenuate HIF-1α synthesis, and decreases breast cancer metastasis.

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Hypoxia-inducible factors mediate coordinated RhoA-ROCK1 expression and signaling in breast cancer cells

http://www.wikidata.org/entity/Q24613508

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2014 nî lūn-bûn

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2014年の論文

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Hypoxia-inducible factors medi ...... gnaling in breast cancer cells

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Hypoxia-inducible factors medi ...... gnaling in breast cancer cells

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Hypoxia-inducible factors medi ...... gnaling in breast cancer cells

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Hypoxia-inducible factors medi ...... gnaling in breast cancer cells

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Proceedings of the National Academy of Sciences of the United States of America

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Hypoxia-inducible factors medi ...... gnaling in breast cancer cells

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Daniele M Gilkes

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Denis Wirtz

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Lisha Xiang

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Maimon E Hubbi

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Pallavi Chaturvedi

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Sun Joo Lee

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P2860

Signaling from Rho to the actin cytoskeleton through protein kinases ROCK and LIM-kinase

Regulation of myosin phosphatase by Rho and Rho-associated kinase (Rho-kinase)

Comprehensive molecular portraits of human breast tumours

Matrix crosslinking forces tumor progression by enhancing integrin signaling

Gene expression profiling spares early breast cancer patients from adjuvant therapy: derived and validated in two population-based cohorts.

Rho GTPases: biochemistry and biology

Cell migration: integrating signals from front to back

Tensional homeostasis and the malignant phenotype

Molecular portraits of human breast tumours

Levels of hypoxia-inducible factor-1alpha independently predict prognosis in patients with lymph node negative breast carcinoma

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Gregg L. Semenza

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