Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin - Test2 - elhamkhani - TriplyDB

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

http://www.wikidata.org/entity/Q24629286

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Adaptive regulation of the brain's antioxidant defences by neurons and astrocytes The sulfiredoxin-peroxiredoxin (Srx-Prx) axis in cell signal transduction and cancer development Nuclear heme oxygenase-1 (HO-1) modulates subcellular distribution and activation of Nrf2, impacting metabolic and anti-oxidant defenses Global transcriptome profile of Cryptococcus neoformans during exposure to hydrogen peroxide induced oxidative stress Effect of MK-801 and Clozapine on the Proteome of Cultured Human Oligodendrocytes.Nrf2 amplifies oxidative stress via induction of Klf9.Responses of cultured human keratocytes and myofibroblasts to ethyl pyruvate: a microarray analysis of gene expression Reexamination of the electrophile response element sequences and context reveals a lack of consensus in gene function.Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington's disease Redox regulation of lipopolysaccharide-mediated sulfiredoxin induction, which depends on both AP-1 and Nrf2.Transcriptomic and innate immune responses to Yersinia pestis in the lymph node during bubonic plague Reduction of cysteine sulfinic acid in eukaryotic, typical 2-Cys peroxiredoxins by sulfiredoxin.Nuclear factor E2-related factor 2 dependent overexpression of sulfiredoxin and peroxiredoxin III in human lung cancer.The subtype of GluN2 C-terminal domain determines the response to excitotoxic insults.Bardoxolone methyl decreases megalin and activates nrf2 in the kidney.Development and validation of a new in vitro assay designed to measure contact allergen-triggered oxidative stress in dendritic cells.Protective effect of carnosic acid, a pro-electrophilic compound, in models of oxidative stress and light-induced retinal degeneration Epigenetic mechanisms in stroke and epilepsy.Thioredoxin 1 protects astrocytes from oxidative stress by maintaining peroxiredoxin activity.Loss of sulfiredoxin renders mice resistant to azoxymethane/dextran sulfate sodium-induced colon carcinogenesis.Telomerase reverse transcriptase promotes the proliferation of human laryngeal carcinoma cells through activation of the activator protein 1.Recovery of NMDA receptor currents from MK-801 blockade is accelerated by Mg2+ and memantine under conditions of agonist exposure Sulforaphane enhances the ability of human retinal pigment epithelial cell against oxidative stress, and its effect on gene expression profile evaluated by microarray analysis Hepatic 3D spheroid models for the detection and study of compounds with cholestatic liability.The S-nitrosylation of glyceraldehyde-3-phosphate dehydrogenase 2 is reduced by interaction with glutathione peroxidase 3 in Saccharomyces cerevisiae.The Cryptococcus neoformans transcriptome at the site of human meningitis.Nrf2 the rescue: effects of the antioxidative/electrophilic response on the liver.Tumor promoter-induced sulfiredoxin is required for mouse skin tumorigenesis.Oxidative stress in synapse development and function.Human stem cell-derived astrocytes and their application to studying Nrf2-mediated neuroprotective pathways and therapeutics in neurodegeneration Influence of GluN2 subunit identity on NMDA receptor function.Mitochondria as a target in the therapeutic properties of curcumin.Role of sulfiredoxin in systemic diseases influenced by oxidative stress.Activation of Nrf2-regulated glutathione pathway genes by ischemic preconditioning.Gene Expression Profiling in Human Lung Cells Exposed to Isoprene-Derived Secondary Organic Aerosol.Nrf2-dependent persistent oxidative stress results in stress-induced vulnerability to depression.SMRT-mediated co-shuttling enables export of class IIa HDACs independent of their CaM kinase phosphorylation sites.THE MULTI-FACETED IMPACT OF PEROXIREDOXINS ON AGING AND DISEASE.The promising potential role of ketones in inflammatory dermatologic disease: a new frontier in treatment research.A c-Jun N-terminal kinase inhibitor, JNK-IN-8, sensitizes triple negative breast cancer cells to lapatinib.

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Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

http://www.wikidata.org/entity/Q24629286

description

2009 nî lūn-bûn

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2009 թուականի Մարտին հրատարակուած գիտական յօդուած

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2009 թվականի մարտին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@ast

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@en

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

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type

label

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@ast

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@en

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@nl

prefLabel

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@ast

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@en

Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

@nl

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Molecules and Cells

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Transcriptional regulation of the AP-1 and Nrf2 target gene sulfiredoxin

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Michael B Sporn

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Paul Baxter

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P2860

Characterization of mammalian sulfiredoxin and its reactivation of hyperoxidized peroxiredoxin through reduction of cysteine sulfinic acid in the active site to cysteine

Peroxiredoxin 2 (PRDX2), an antioxidant enzyme, is under-expressed in Down syndrome fetal brains

Structure of the sulphiredoxin–peroxiredoxin complex reveals an essential repair embrace

Reduction of Cysteine Sulfinic Acid in Peroxiredoxin by Sulfiredoxin Proceeds Directly through a Sulfinic Phosphoryl Ester Intermediate

ATP-dependent reduction of cysteine-sulphinic acid by S. cerevisiae sulphiredoxin.

Regeneration of peroxiredoxins by p53-regulated sestrins, homologs of bacterial AhpD

Sestrin 2 is not a reductase for cysteine sulfinic acid of peroxiredoxins

Structure, mechanism and regulation of peroxiredoxins

Peroxiredoxins: a historical overview and speculative preview of novel mechanisms and emerging concepts in cell signaling

Mechanistic studies of the Nrf2-Keap1 signaling pathway

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10.1007/S10059-009-0050-Y

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Thomas H. Gillingwater

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