miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis - Test2 - elhamkhani - TriplyDB

miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis

miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis

http://www.wikidata.org/entity/Q24647507

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MicroRNA-277 modulates the neurodegeneration caused by Fragile X premutation rCGG repeats Pumilio1 haploinsufficiency leads to SCA1-like neurodegeneration by increasing wild-type Ataxin1 levels Gene, Stem Cell, and Alternative Therapies for SCA 1 Precision medicine in spinocerebellar ataxias: treatment based on common mechanisms of disease Regulation of protein homeostasis in neurodegenerative diseases: the role of coding and non-coding genes Issues and prospects of microRNA-based biomarkers in blood and other body fluids Current understanding of the role of microRNAs in spinocerebellar ataxias Novel microRNAs differentially expressed during aging in the mouse brain MicroRNAs in Neurotoxicity Repression of alpha-synuclein expression and toxicity by microRNA-7.Prioritization of disease microRNAs through a human phenome-microRNAome network MicroRNAs in Experimental Models of Movement Disorders.Defining larger roles for "tiny" RNA molecules: role of miRNAs in neurodegeneration research.Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.Understanding microRNAs in neurodegeneration.Hydrogen peroxide responsive miR153 targets Nrf2/ARE cytoprotection in paraquat induced dopaminergic neurotoxicity.MicroRNA-7 protects against 1-methyl-4-phenylpyridinium-induced cell death by targeting RelA.Context-dependent functions of specific microRNAs in neuronal development.FOX-2 dependent splicing of ataxin-2 transcript is affected by ataxin-1 overexpression A highly expressed miR-101 isomiR is a functional silencing small RNA Genetic variations creating microRNA target sites in the FXN 3'-UTR affect frataxin expression in Friedreich ataxia.Cross-talking noncoding RNAs contribute to cell-specific neurodegeneration in SCA7 Regulation of microRNA expression by HMGA1 proteins.Reversible inhibition of PSD-95 mRNA translation by miR-125a, FMRP phosphorylation, and mGluR signaling.Distinct transduction profiles in the CNS via three injection routes of AAV9 and the application to generation of a neurodegenerative mouse model.Practical Aspects of microRNA Target Prediction.Transforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells.microRNA-101 is a potent inhibitor of autophagy MicroRNA-130a represses transcriptional activity of aquaporin 4 M1 promoter Cause or Effect: Misregulation of microRNA Pathways in Neurodegeneration.FGF-2 regulates cell proliferation, migration, and angiogenesis through an NDY1/KDM2B-miR-101-EZH2 pathway Unveiling of miRNA Expression Patterns in Purkinje Cells During Development.A let-7-to-miR-125 MicroRNA Switch Regulates Neuronal Integrity and Lifespan in Drosophila.Time is of the essence: microRNAs and age-associated neurodegeneration.TMEM106B, the risk gene for frontotemporal dementia, is regulated by the microRNA-132/212 cluster and affects progranulin pathways.Lack of miRNA Misregulation at Early Pathological Stages in Drosophila Neurodegenerative Disease Models.miR-93/miR-106b/miR-375-CIC-CRABP1: a novel regulatory axis in prostate cancer progression Wnt-5a-regulated miR-101b controls COX2 expression in hippocampal neurons Neurodegeneration the RNA way MicroRNA Biogenesis and Hedgehog-Patched Signaling Cooperate to Regulate an Important Developmental Transition in Granule Cell Development.

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miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis

http://www.wikidata.org/entity/Q24647507

description

2008 nî lūn-bûn

@nan

2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2008 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@ast

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@en

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@nl

type

label

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@ast

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@en

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@nl

prefLabel

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@ast

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@en

miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

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miR-19, miR-101 and miR-130 co ...... lly modulate SCA1 pathogenesis

@en

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Christina Thaller

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Yoontae Lee

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Nuclear localization or inclusion body formation of ataxin-2 are not necessary for SCA2 pathogenesis in mouse or human

Cerebellar neurodegeneration in the absence of microRNAs

Prediction of mammalian microRNA targets

Combinatorial microRNA target predictions

MicroRNAs: genomics, biogenesis, mechanism, and function

Opposing effects of polyglutamine expansion on native protein complexes contribute to SCA1

Trinucleotide Repeat Disorders

Ataxin-1 fusion partners alter polyQ lethality and aggregation.

Expansion of an unstable trinucleotide CAG repeat in spinocerebellar ataxia type 1.

RNAi suppresses polyglutamine-induced neurodegeneration in a model of spinocerebellar ataxia.

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1137-9

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4095656

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10.1038/NN.2183

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10

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11

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Jennifer R Gatchel

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2008-10-01T00:00:00Z

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23224790

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1032334172

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18758459

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2574629

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