Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice. - Test2 - elhamkhani - TriplyDB

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

http://www.wikidata.org/entity/Q33929052

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MicroRNAs Regulate Mitochondrial Function in Cerebral Ischemia-Reperfusion Injury Substantial and robust changes in microRNA transcriptome support postnatal development of the hypothalamus in rat.The microRNA miR-124 suppresses seizure activity and regulates CREB1 activity Non-coding RNA in neural function, disease, and aging Kuwanon V inhibits proliferation, promotes cell survival and increases neurogenesis of neural stem cells Downregulation of blood serum microRNA 29 family in patients with Parkinson's disease Unveiling of miRNA Expression Patterns in Purkinje Cells During Development.microRNA-33 Regulates ApoE Lipidation and Amyloid-β Metabolism in the Brain MicroRNA miR-29 controls a compensatory response to limit neuronal iron accumulation during adult life and aging MicroRNA Expression in the Glaucomatous Retina Genes with de novo mutations are shared by four neuropsychiatric disorders discovered from NPdenovo database Potent Anti-seizure Effects of Locked Nucleic Acid Antagomirs Targeting miR-134 in Multiple Mouse and Rat Models of Epilepsy.Molecular basis of hair cell loss.Pharmacomodulation of microRNA Expression in Neurocognitive Diseases: Obstacles and Future Opportunities.An overview of circulating cell-free microRNAs as putative biomarkers in Alzheimer's and Parkinson's Diseases.Stress-induced changes in miRNA biogenesis and functioning.A Simple Alternative to Stereotactic Injection for Brain Specific Knockdown of miRNA.Exploring and exploiting unique properties of the hippocampal dentate gyrus for post-stroke therapy: astrocytes link ischemic resistance with neurogenic potential.The Ticking of the Epigenetic Clock: Antipsychotic Drugs in Old Age.microRNA dysregulation in polyglutamine toxicity of TATA-box binding protein is mediated through STAT1 in mouse neuronal cells MicroRNA-29c/PTEN pathway is involved in mice brain development and modulates neurite outgrowth in PC12 cells.Endothelial growth medium suppresses apoptosis of mesenchymal stem cells in vitro via decrease of miR‑29a Expression of MicroRNA-29a Regulated by Yes-Associated Protein Modulates the Neurite Outgrowth in N2a Cells.MicroRNAs in brain cholesterol metabolism and their implications for Alzheimer's disease.Noncoding RNAs in neurodegeneration.Dicer1 Ablation Impairs Responsiveness of Cerebellar Granule Neuron Precursors to Sonic Hedgehog and Disrupts Expression of Distinct Cell Cycle Regulator Genes.miR-29a differentially regulates cell survival in astrocytes from cornu ammonis 1 and dentate gyrus by targeting VDAC1.Hippocampal MicroRNAs Respond to Administration of Antidepressant Fluoxetine in Adult Mice.Identification of differentially expressed miRNAs in alopecia areata that target immune-regulatory pathways.

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P2860

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

http://www.wikidata.org/entity/Q33929052

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年學術文章

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name

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

@ast

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

@en

type

label

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

@ast

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

@en

prefLabel

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

@ast

Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

@en

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Brain-specific knockdown of miR-29 results in neuronal cell death and ataxia in mice.

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Arpita Banik

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Beena Pillai

http://www.w3.org/2001/XMLSchema#string

Manali Garg

http://www.w3.org/2001/XMLSchema#string

Mayuresh A Sarangdhar

http://www.w3.org/2001/XMLSchema#string

Mrinal Chawla

http://www.w3.org/2001/XMLSchema#string

Reema Roshan

http://www.w3.org/2001/XMLSchema#string

Shruti Shridhar

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Vijay P A L Singh

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P2860

Identification of novel targets for miR-29a using miRNA proteomics

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

MicroRNA-29 family reverts aberrant methylation in lung cancer by targeting DNA methyltransferases 3A and 3B

miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis

mir-29 regulates Mcl-1 protein expression and apoptosis

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

MicroRNAs: genomics, biogenesis, mechanism, and function

Tcl1 expression in chronic lymphocytic leukemia is regulated by miR-29 and miR-181

A microRNA component of the p53 tumour suppressor network

The bifunctional microRNA miR-9/miR-9* regulates REST and CoREST and is downregulated in Huntington's disease

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10.1261/RNA.044008.113

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8

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20

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