Macrophages promote axon regeneration with concurrent neurotoxicity
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How do immune cells support and shape the brain in health, disease, and aging?Spontaneous recovery of cochlear fibrocytes after severe degeneration caused by acute energy failure.Azithromycin drives alternative macrophage activation and improves recovery and tissue sparing in contusion spinal cord injuryThe yin and yang of microgliaRegeneration-associated macrophages: a novel approach to boost intrinsic regenerative capacity for axon regenerationIdentification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cordIntravital imaging of axonal interactions with microglia and macrophages in a mouse dorsal column crush injuryMultipotent adult progenitor cells prevent macrophage-mediated axonal dieback and promote regrowth after spinal cord injury.Macrophages migrate in an activation-dependent manner to chemokines involved in neuroinflammation.Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injuryAdministration of chondroitinase ABC rostral or caudal to a spinal cord injury site promotes anatomical but not functional plasticity.Control of neuroinflammation as a therapeutic strategy for amyotrophic lateral sclerosis and other neurodegenerative disordersFormyl peptide receptors promotes neural differentiation in mouse neural stem cells by ROS generation and regulation of PI3K-AKT signalingLow-level laser facilitates alternatively activated macrophage/microglia polarization and promotes functional recovery after crush spinal cord injury in rats.Changes in NG2 cells and oligodendrocytes in a new model of intraspinal hemorrhage.IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.Involvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis.System x(c)(-) regulates microglia and macrophage glutamate excitotoxicity in vivoA toll-like receptor 9 antagonist improves bladder function and white matter sparing in spinal cord injury.Microglial Toll-like receptor 2 contributes to kainic acid-induced glial activation and hippocampal neuronal cell death.Cell Therapy From Bench to Bedside Translation in CNS Neurorestoratology Era.The effect of Lycium barbarum on spinal cord injury, particularly its relationship with M1 and M2 macrophage in ratsInosine augments the effects of a Nogo receptor blocker and of environmental enrichment to restore skilled forelimb use after stroke.Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.Wallerian degeneration: gaining perspective on inflammatory events after peripheral nerve injury.Transforming growth factor α transforms astrocytes to a growth-supportive phenotype after spinal cord injuryMicroglia: scapegoat, saboteur, or something else?A lifespan approach to neuroinflammatory and cognitive disorders: a critical role for glia.Toll-Like Receptors and Dectin-1, a C-Type Lectin Receptor, Trigger Divergent Functions in CNS Macrophages.Macrophage migration inhibitory factor (MIF) is essential for inflammatory and neuropathic pain and enhances pain in response to stressThe unusual response of serotonergic neurons after CNS Injury: lack of axonal dieback and enhanced sprouting within the inhibitory environment of the glial scarTargeting acute inflammation to complement spinal cord repair.A reassessment of a classic neuroprotective combination therapy for spinal cord injured rats: LPS/pregnenolone/indomethacinEffect of modulating macrophage phenotype on peripheral nerve repair.CCR2 overexpression promotes the efficient recruitment of retinal microglia in vitro.Low dose zymosan ameliorates both chronic and relapsing experimental autoimmune encephalomyelitis.Chronic Toxoplasma gondii infection enhances β-amyloid phagocytosis and clearance by recruited monocytes.Polarized Macrophages Have Distinct Roles in the Differentiation and Migration of Embryonic Spinal-cord-derived Neural Stem Cells After Grafting to Injured Sites of Spinal Cord.Immunohistochemical and transcriptome analyses indicate complex breakdown of axonal transport mechanisms in canine distemper leukoencephalitis.Nerve regeneration restores supraspinal control of bladder function after complete spinal cord injury.
P2860
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P2860
Macrophages promote axon regeneration with concurrent neurotoxicity
description
2009 nî lūn-bûn
@nan
2009 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մարտին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Macrophages promote axon regeneration with concurrent neurotoxicity
@ast
Macrophages promote axon regeneration with concurrent neurotoxicity
@en
Macrophages promote axon regeneration with concurrent neurotoxicity
@nl
type
label
Macrophages promote axon regeneration with concurrent neurotoxicity
@ast
Macrophages promote axon regeneration with concurrent neurotoxicity
@en
Macrophages promote axon regeneration with concurrent neurotoxicity
@nl
prefLabel
Macrophages promote axon regeneration with concurrent neurotoxicity
@ast
Macrophages promote axon regeneration with concurrent neurotoxicity
@en
Macrophages promote axon regeneration with concurrent neurotoxicity
@nl
P2093
P2860
P3181
P1476
Macrophages promote axon regeneration with concurrent neurotoxicity
@en
P2093
Daniel P Ankeny
John C Gensel
Phillip G Popovich
Satoshi Nakamura
P2860
P304
P3181
P356
10.1523/JNEUROSCI.3992-08.2009
P407
P577
2009-03-25T00:00:00Z