The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy. - Test2 - elhamkhani - TriplyDB

The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

http://www.wikidata.org/entity/Q33936995

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Differential involvement of trigeminal transition zone and laminated subnucleus caudalis in orofacial deep and cutaneous hyperalgesia: the effects of interleukin-10 and glial inhibitors P2X7-dependent release of interleukin-1beta and nociception in the spinal cord following lipopolysaccharide Evidence that tricyclic small molecules may possess toll-like receptor and myeloid differentiation protein 2 activity Treatment of Complex Regional Pain Syndrome (CRPS) using low dose naltrexone (LDN)Expression and distribution of Toll-like receptors 11-13 in the brain during murine neurocysticercosis Pathological and protective roles of glia in chronic pain Macrophages promote axon regeneration with concurrent neurotoxicity The "toll" of opioid-induced glial activation: improving the clinical efficacy of opioids by targeting glia Proinflammatory cytokines oppose opioid-induced acute and chronic analgesia Evidence that intrathecal morphine-3-glucuronide may cause pain enhancement via toll-like receptor 4/MD-2 and interleukin-1beta Chemokines and the pathophysiology of neuropathic pain Toll-like receptors: the swiss army knife of immunity and vaccine development Microglia in the spinal cord and neuropathic pain Rethinking inflammation: neural circuits in the regulation of immunity.Reflex principles of immunological homeostasis Controlling complexity: the clinical relevance of mouse complex genetics HMGB1 mediates endogenous TLR2 activation and brain tumor regression Evidence for a role of endocannabinoids, astrocytes and p38 phosphorylation in the resolution of postoperative pain The effects of age on pain sensitivity: preclinical studies Contemporary views on inflammatory pain mechanisms: TRPing over innate and microglial pathways International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease Neuroimmune interactions in itch: Do chronic itch, chronic pain, and chronic cough share similar mechanisms?Possible involvement of toll-like receptor 4/myeloid differentiation factor-2 activity of opioid inactive isomers causes spinal proinflammation and related behavioral consequences Non-stereoselective reversal of neuropathic pain by naloxone and naltrexone: involvement of toll-like receptor 4 (TLR4)Hemokinin-1 gene expression is upregulated in microglia activated by lipopolysaccharide through NF-κB and p38 MAPK signaling pathways TLR 2 and 4 responsiveness from isolated peripheral blood mononuclear cells from rats and humans as potential chronic pain biomarkers Inhibition of spinal microglial cathepsin S for the reversal of neuropathic pain Rapid co-release of interleukin 1beta and caspase 1 in spinal cord inflammation The biological significance of evolution in autoimmune phenomena Toll-like receptor mRNA expression is selectively increased in the colonic mucosa of two animal models relevant to irritable bowel syndrome Cellular and Molecular Mechanisms of Pain Damage-associated molecular patterns generated in osteoarthritis directly excite murine nociceptive neurons through Toll-like receptor 4.Toll-like receptor 4 contributes to chronic itch, alloknesis, and spinal astrocyte activation in male mice Differences in neural-immune gene expression response in rat spinal dorsal horn correlates with variations in electroacupuncture analgesia Mycobacteria attenuate nociceptive responses by formyl peptide receptor triggered opioid peptide release from neutrophils.Comparison of the Expression Changes after Botulinum Toxin Type A and Minocycline Administration in Lipopolysaccharide-Stimulated Rat Microglial and Astroglial Cultures.Toll-like receptor 4 mutant and null mice retain morphine-induced tolerance, hyperalgesia, and physical dependence.Down-regulation of Toll-like receptor 4 gene expression by short interfering RNA attenuates bone cancer pain in a rat model Biphalin, a Dimeric Enkephalin, Alleviates LPS-Induced Activation in Rat Primary Microglial Cultures in Opioid Receptor-Dependent and Receptor-Independent Manners.Toll-Like Receptor 4 in Paraventricular Nucleus Mediates Visceral Hypersensitivity Induced by Maternal Separation

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

http://www.wikidata.org/entity/Q33936995

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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Proceedings of the National Academy of Sciences of the United States of America

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The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy.

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Flobert Y Tanga

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Joyce A DeLeo

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Nancy Nutile-McMenemy

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Physical contact between lipopolysaccharide and toll-like receptor 4 revealed by genetic complementation

Innate immune recognition

The role of neuroinflammation and neuroimmune activation in persistent pain.

CR3: a general purpose adhesion-recognition receptor essential for innate immunity.

Complement receptor 3 (CR3): a public transducer of innate immunity signals in macrophages.

Modulation of the expression of cyclooxygenase-2 by fatty acids mediated through toll-like receptor 4-derived signaling pathways.

Activation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway.

Toll-like receptors and their signaling mechanisms.

Neuroimmune activation and neuroinflammation in chronic pain and opioid tolerance/hyperalgesia.

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