Architectures of somatic genomic rearrangement in human cancer amplicons at sequence-level resolution - Test2 - elhamkhani - TriplyDB

Architectures of somatic genomic rearrangement in human cancer amplicons at sequence-level resolution

Architectures of somatic genomic rearrangement in human cancer amplicons at sequence-level resolution

http://www.wikidata.org/entity/Q24676904

about

Evaluation of paired-end sequencing strategies for detection of genome rearrangements in cancer Chromothripsis is a common mechanism driving genomic rearrangements in primary and metastatic colorectal cancer Combinatorics of the breakage-fusion-bridge mechanism Comprehensive long-span paired-end-tag mapping reveals characteristic patterns of structural variations in epithelial cancer genomes Massive genomic rearrangement acquired in a single catastrophic event during cancer development GLO1-A novel amplified gene in human cancer Complex landscapes of somatic rearrangement in human breast cancer genomes.A sequence-level map of chromosomal breakpoints in the MCF-7 breast cancer cell line yields insights into the evolution of a cancer genome Integrated analysis of homozygous deletions, focal amplifications, and sequence alterations in breast and colorectal cancers Identification of somatically acquired rearrangements in cancer using genome-wide massively parallel paired-end sequencing Whole-genome cancer analysis as an approach to deeper understanding of tumour biology Evolution of the cancer genome Polymerase δ replicates both strands after homologous recombination-dependent fork restart Development of personalized tumor biomarkers using massively parallel sequencing Analysis of next-generation genomic data in cancer: accomplishments and challenges The Growing Importance of CNVs: New Insights for Detection and Clinical Interpretation Distinct patterns of somatic alterations in a lymphoblastoid and a tumor genome derived from the same individual PEMer: a computational framework with simulation-based error models for inferring genomic structural variants from massive paired-end sequencing data The cancer genome The life history of 21 breast cancers The patterns and dynamics of genomic instability in metastatic pancreatic cancer Yeast genome analysis identifies chromosomal translocation, gene conversion events and several sites of Ty element insertion.Genomancy: predicting tumour response to cancer therapy based on the oracle of genetics.A sequence-based survey of the complex structural organization of tumor genomes.A microhomology-mediated break-induced replication model for the origin of human copy number variation.Disruption of chromosome 11 in canine fibrosarcomas highlights an unusual variability of CDKN2B in dogs.Fusion of nearby inverted repeats by a replication-based mechanism leads to formation of dicentric and acentric chromosomes that cause genome instability in budding yeast.Whole genome sequencing of matched tumor, adjacent non-tumor tissues and corresponding normal blood samples of hepatocellular carcinoma patients revealed dynamic changes of the mutations profiles during hepatocarcinogenesis.CNAseg--a novel framework for identification of copy number changes in cancer from second-generation sequencing data.BreakTrans: uncovering the genomic architecture of gene fusions Network-guided analysis of genes with altered somatic copy number and gene expression reveals pathways commonly perturbed in metastatic melanoma.Genomic instability in breast cancer: pathogenesis and clinical implications Long-range massively parallel mate pair sequencing detects distinct mutations and similar patterns of structural mutability in two breast cancer cell lines Genome wide proteomics of ERBB2 and EGFR and other oncogenic pathways in inflammatory breast cancer.The mutation spectrum revealed by paired genome sequences from a lung cancer patient.Expanding the computational toolbox for mining cancer genomes.What are we learning from the cancer genome?An algorithmic approach for breakage-fusion-bridge detection in tumor genomes Genome-wide detection of spontaneous chromosomal rearrangements in bacteria Detection of human tumor cells by amplicon fusion site polymerase chain reaction (AFS-PCR).

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Architectures of somatic genomic rearrangement in human cancer amplicons at sequence-level resolution

http://www.wikidata.org/entity/Q24676904

description

2007 nî lūn-bûn

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2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Architectures of somatic genom ...... s at sequence-level resolution

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Adam P Butler

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Bob Plumb

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Kirsten McLay

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Lucy Matthews

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Initial sequencing and analysis of the human genome

The Stability of Broken Ends of Chromosomes in Zea Mays.

A census of human cancer genes

End-sequence profiling: sequence-based analysis of aberrant genomes

BLAT--the BLAST-like alignment tool

Patterns of somatic mutation in human cancer genomes

SSAHA: a fast search method for large DNA databases

Oncogene amplification in solid tumors.

Amplification of oncogenes revisited: from expression profiling to clinical application.

Chromosome instability as a result of double-strand breaks near telomeres in mouse embryonic stem cells

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1296-303

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scholarly article

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4572065

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10.1101/GR.6522707

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9

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Richard Wooster

Michael Andrew Quail

P Andrew Futreal

Michael Stratton

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6164756

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1950898

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