Hypoxia attenuates the expression of E-cadherin via up-regulation of SNAIL in ovarian carcinoma cells - Test2 - elhamkhani - TriplyDB

Hypoxia attenuates the expression of E-cadherin via up-regulation of SNAIL in ovarian carcinoma cells

Hypoxia attenuates the expression of E-cadherin via up-regulation of SNAIL in ovarian carcinoma cells

http://www.wikidata.org/entity/Q24685207

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Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition Direct regulation of TWIST by HIF-1alpha promotes metastasis Upstream determinants of estrogen receptor-alpha regulation of metastatic tumor antigen 3 pathway.Role of nuclear receptors in breast cancer stem cells Hypoxia-inducible factors as molecular targets for liver diseases Epithelial to Mesenchymal Transition in a Clinical Perspective Molecular mechanisms of epithelial-mesenchymal transition Hypoxia impairs primordial germ cell migration in zebrafish (Danio rerio) embryos A novel microfluidic platform for studying mammalian cell chemotaxis in different oxygen environments under zero-flow conditions.Neuraminidase-1: a novel therapeutic target in multistage tumorigenesis Tackling Cancer Stem Cells via Inhibition of EMT Transcription Factors The cancer stem cell niche--there goes the neighborhood?A TRIP230-retinoblastoma protein complex regulates hypoxia-inducible factor-1α-mediated transcription and cancer cell invasion uPAR induces epithelial-mesenchymal transition in hypoxic breast cancer cells.Establishment and characterization of a new hypoxia-resistant cancer cell line, OCUM-12/Hypo, derived from a scirrhous gastric carcinoma.Epithelial-to-mesenchymal transition in prostate cancer: paradigm or puzzle?Hypoxia, inflammation, and the tumor microenvironment in metastatic disease Estrogen regulates Snail and Slug in the down-regulation of E-cadherin and induces metastatic potential of ovarian cancer cells through estrogen receptor alpha.The lysyl oxidases LOX and LOXL2 are necessary and sufficient to repress E-cadherin in hypoxia: insights into cellular transformation processes mediated by HIF-1 A three-dimensional in vitro model of tumor cell intravasation.The Effect of Snail1 Gene Silencing by siRNA in Metastatic Breast Cancer Cell Lines.Human prominin-1 (CD133) is detected in both neoplastic and non-neoplastic salivary gland diseases and released into saliva in a ubiquitinated form.Snail plays an oncogenic role in glioblastoma by promoting epithelial mesenchymal transition.The role of hypoxia on the acquisition of epithelial-mesenchymal transition and cancer stemness: a possible link to epigenetic regulation The role of hypoxia inducible factor-1 in hepatocellular carcinoma.Ovarian cancer development and metastasis.Hypoxia-induced Bmi1 promotes renal tubular epithelial cell-mesenchymal transition and renal fibrosis via PI3K/Akt signal Hypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells.Multilayer control of the EMT master regulators.Significance of heterogeneous Twist2 expression in human breast cancers.Redox sensor CtBP mediates hypoxia-induced tumor cell migration.Ligand-activated PPARδ modulates the migration and invasion of melanoma cells by regulating Snail expression.Implication of snail in metabolic stress-induced necrosis.Sequential molecular and cellular events during neoplastic progression: a mouse syngeneic ovarian cancer model.Hypoxic regulation of RIOK3 is a major mechanism for cancer cell invasion and metastasis Reactive oxygen species promote ovarian cancer progression via the HIF-1α/LOX/E-cadherin pathway VHL promotes E2 box-dependent E-cadherin transcription by HIF-mediated regulation of SIP1 and snail HIF-1α Promotes Epithelial-Mesenchymal Transition and Metastasis through Direct Regulation of ZEB1 in Colorectal Cancer.Prognostic significance of E-cadherin-catenin complex in epithelial ovarian cancer.Copper depletion inhibits CoCl2-induced aggressive phenotype of MCF-7 cells via downregulation of HIF-1 and inhibition of Snail/Twist-mediated epithelial-mesenchymal transition.

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P2860

Hypoxia attenuates the expression of E-cadherin via up-regulation of SNAIL in ovarian carcinoma cells

http://www.wikidata.org/entity/Q24685207

description

2003 nî lūn-bûn

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2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2003 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2003年の論文

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2003年论文

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name

Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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type

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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The American Journal of Pathology

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Hypoxia attenuates the express ...... AIL in ovarian carcinoma cells

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Akiko Horiuchi

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Cuiju Wang

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Ikuo Konishi

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Kenji Oka

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Satoshi Ohira

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Toshio Nikaido

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Tsutomu Imai

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P2860

Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

Regulation of tumor angiogenesis by p53-induced degradation of hypoxia-inducible factor 1alpha

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Overexpression of hypoxia-inducible factor 1alpha in common human cancers and their metastases

Regulation of mammalian O2 homeostasis by hypoxia-inducible factor 1

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

HIF-1 and human disease: one highly involved factor

The E-cadherin-catenin complex in tumour metastasis: structure, function and regulation

Expression of hypoxia-inducible factor 1alpha in epithelial ovarian tumors: its impact on prognosis and on response to chemotherapy

The transcription factor snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression

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