Hypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells. - Test2 - elhamkhani - TriplyDB

Hypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells.

Hypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells.

http://www.wikidata.org/entity/Q34170239

about

The Effect of Hypoxia on Mesenchymal Stem Cell Biology HypoxamiRs and cancer: from biology to targeted therapy MicroRNA-34a: a potential therapeutic target in human cancer OxymiRs in cutaneous development, wound repair and regeneration miRNA Deregulation in Cancer Cells and the Tumor Microenvironment Regulatory Mechanisms of the Molecular Pathways in Fibrosis Induced by MicroRNAs Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition Epithelial-mesenchymal transition: An emerging target in tissue fibrosis MicroRNA-34a regulates cardiac fibrosis after myocardial infarction by targeting Smad4 MicroRNA-590 is an EMT-suppressive microRNA involved in the TGFβ signaling pathway.Paraquat poisoning induced pulmonary epithelial mesenchymal transition through Notch1 pathway.Mutual Regulation of MiR-199a-5p and HIF-1α Modulates the Warburg Effect in Hepatocellular Carcinoma.Signalling pathways involved in hypoxia-induced renal fibrosis.3'UTR polymorphisms of carbonic anhydrase IX determine the miR-34a targeting efficiency and prognosis of hepatocellular carcinoma.MicroRNA-769-3p down-regulates NDRG1 and enhances apoptosis in MCF-7 cells during reoxygenation.HypoxamiR regulation and function in ischemic cardiovascular diseases.Hypoxia-induced Bmi1 promotes renal tubular epithelial cell-mesenchymal transition and renal fibrosis via PI3K/Akt signal The miR-124-prolyl hydroxylase P4HA1-MMP1 axis plays a critical role in prostate cancer progression.Hepatitis C virus mediated changes in miRNA-449a modulates inflammatory biomarker YKL40 through components of the NOTCH signaling pathway.miR-34 - a microRNA replacement therapy is headed to the clinic.Signaling mechanisms of the epithelial-mesenchymal transition.miR-372 inhibits p62 in head and neck squamous cell carcinoma in vitro and in vivo.Snail Is a Direct Target of Hypoxia-inducible Factor 1α (HIF1α) in Hypoxia-induced Endothelial to Mesenchymal Transition of Human Coronary Endothelial Cells.Epithelium-Intrinsic MicroRNAs Contribute to Mucosal Immune Homeostasis by Promoting M-Cell Maturation.MiR-30a Inhibits the Epithelial--Mesenchymal Transition of Podocytes through Downregulation of NFATc3 Metformin inhibits gastric cancer cells metastatic traits through suppression of epithelial-mesenchymal transition in a glucose-independent manner.Macrophage migration inhibitory factor induces epithelial to mesenchymal transition, enhances tumor aggressiveness and predicts clinical outcome in resected pancreatic ductal adenocarcinoma.Dichotomy effects of Akt signaling in breast cancer.Tg737 signaling is required for hypoxia-enhanced invasion and migration of hepatoma cells.Hypoxia-regulated target genes implicated in tumor metastasis.MiR-652 inhibits acidic microenvironment-induced epithelial-mesenchymal transition of pancreatic cancer cells by targeting ZEB1.A miR-34a-SIRT6 axis in the squamous cell differentiation network Regulation of angiogenesis by hypoxia: the role of microRNA.miR-34a expression, epigenetic regulation, and function in human placental diseases.Post-transcriptional regulatory network of epithelial-to-mesenchymal and mesenchymal-to-epithelial transitions.Tumor suppressor microRNA-34a inhibits cell proliferation by targeting Notch1 in renal cell carcinoma Robustness of the hypoxic response: another job for miRNAs?Role of the endothelial-to-mesenchymal transition in renal fibrosis of chronic kidney disease.miRNA and TMPRSS2-ERG do not mind their own business in prostate cancer cells.MicroRNAs and tumor vasculature normalization: impact on anti-tumor immune response.

P2860

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P2860

Hypoxia-induced down-regulation of microRNA-34a promotes EMT by targeting the Notch signaling pathway in tubular epithelial cells.

http://www.wikidata.org/entity/Q34170239

description

2012 nî lūn-bûn

@nan

2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2012 թվականի փետրվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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name

Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

@ast

Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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type

label

Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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prefLabel

Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Hypoxia-induced down-regulatio ...... y in tubular epithelial cells.

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Chen Huang

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Hanmin Wang

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Lijuan Zhao

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Lin Xia

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Rui Du

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Shiren Sun

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Wenjuan Sun

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Yan Yu

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P2860

MiRNA-directed regulation of VEGF and other angiogenic factors under hypoxia

Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition

Interaction with factor inhibiting HIF-1 defines an additional mode of cross-coupling between the Notch and hypoxia signaling pathways

The microRNA miR-34a inhibits prostate cancer stem cells and metastasis by directly repressing CD44

A microRNA signature of hypoxia

Hypoxia attenuates the expression of E-cadherin via up-regulation of SNAIL in ovarian carcinoma cells

SIP1/ZEB2 induces EMT by repressing genes of different epithelial cell-cell junctions

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

High glucose down-regulates miR-29a to increase collagen IV production in HK-2 cells

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