There's Something Wrong with my MAM; the ER-Mitochondria Axis and Neurodegenerative Diseases - Test2 - elhamkhani - TriplyDB

There's Something Wrong with my MAM; the ER-Mitochondria Axis and Neurodegenerative Diseases

There's Something Wrong with my MAM; the ER-Mitochondria Axis and Neurodegenerative Diseases

http://www.wikidata.org/entity/Q26768635

about

Triad of Risk for Late Onset Alzheimer's: Mitochondrial Haplotype, APOE Genotype and Chromosomal Sex Proteomic mapping of cytosol-facing outer mitochondrial and ER membranes in living human cells by proximity biotinylation Protein Homeostasis in Amyotrophic Lateral Sclerosis: Therapeutic Opportunities?Pathogenic p62/SQSTM1 mutations impair energy metabolism through limitation of mitochondrial substrates.α-Synuclein binds to the ER-mitochondria tethering protein VAPB to disrupt Ca2+ homeostasis and mitochondrial ATP production β Cell-specific increased expression of calpastatin prevents diabetes induced by islet amyloid polypeptide toxicity.Mitochondria-associated membrane collapse is a common pathomechanism in SIGMAR1- and SOD1-linked ALS Hepatitis C Virus Replication.Emerging pathways driving early synaptic pathology in Alzheimer's disease.Orchestrating cellular signaling pathways-the cellular "conductor" protein tyrosine phosphatase interacting protein 51 (PTPIP51).Ultrastructural and biochemical basis for hepatitis C virus morphogenesis.Neurobiology of axonal transport defects in motor neuron diseases: Opportunities for translational research?Cellular Cholesterol Homeostasis in Alzheimer's Disease.NCLs and ER: A stressful relationship.Caught in the act - protein adaptation and the expanding roles of the PACS proteins in tissue homeostasis and disease.Interaction of Mitochondria with the Endoplasmic Reticulum and Plasma Membrane in Calcium Homeostasis, Lipid Trafficking and Mitochondrial Structure ALS/FTD-associated FUS activates GSK-3β to disrupt the VAPB-PTPIP51 interaction and ER-mitochondria associations.Interplay of Energetics and ER Stress Exacerbates Alzheimer's Amyloid-β (Aβ) Toxicity in Yeast.Roles of sigma-1 receptors on mitochondrial functions relevant to neurodegenerative diseases.The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.Connecting Ca2+ and lysosomes to Parkinson disease.HDAC6 inhibition reverses axonal transport defects in motor neurons derived from FUS-ALS patients.Ascorbate peroxidase proximity labeling coupled with biochemical fractionation identifies promoters of endoplasmic reticulum-mitochondrial contacts.Mitochondrial Hsp70 and the troubles of nomenclature: leaving behind tradition to gain intuitiveness and clarity.Oxidative stress and cellular pathologies in Parkinson's disease.ER-mitochondria tethering by PDZD8 regulates Ca2+ dynamics in mammalian neurons.Interorganelle Communication between Mitochondria and the Endolysosomal System.Mediating ER-mitochondrial cross-talk.The “sweet” side of ER-mitochondria contact sites.The axonal endoplasmic reticulum: One organelle-many functions in development, maintenance, and plasticity.β-Amyloid and the Pathomechanisms of Alzheimer's Disease: A Comprehensive View.Endoplasmic Reticulum-Mitochondria Communication Through Ca2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs).Over Six Decades of Discovery and Characterization of the Architecture at Mitochondria-Associated Membranes (MAMs).Interactions of Mitochondria/Metabolism and Calcium Regulation in Alzheimer's Disease: A Calcinist Point of View.13 reasons why the brain is susceptible to oxidative stress.Mitofusin-Dependent ER Stress Triggers Glial Dysfunction and Nervous System Degeneration in a Drosophila Model of Friedreich's Ataxia.A key role for MAM in mediating mitochondrial dysfunction in Alzheimer disease.Mutations in glycyl-tRNA-synthetase impair mitochondrial metabolism in neurons.Poloxamer 188 decreases membrane toxicity of mutant SOD1 and ameliorates pathology observed in SOD1 mouse model for ALS.Wolfram syndrome: MAMs' connection?

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There's Something Wrong with my MAM; the ER-Mitochondria Axis and Neurodegenerative Diseases

http://www.wikidata.org/entity/Q26768635

description

2016 nî lūn-bûn

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2016 թուականի Մարտին հրատարակուած գիտական յօդուած

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2016 թվականի մարտին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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There's Something Wrong with m ...... and Neurodegenerative Diseases

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J.TINS.2016.01.008

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Trends in Neurosciences

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There's Something Wrong with m ...... and Neurodegenerative Diseases

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Christopher C J Miller

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Dawn H W Lau

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Radu Stoica

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Creative Commons Attribution 4.0 International

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VAPB interacts with the mitochondrial protein PTPIP51 to regulate calcium homeostasis

Characterization of amyotrophic lateral sclerosis-linked P56S mutation of vesicle-associated membrane protein-associated protein B (VAPB/ALS8)

An actin-dependent step in mitochondrial fission mediated by the ER-associated formin INF2

The mechanism of Ca2+ -dependent regulation of kinesin-mediated mitochondrial motility

Mutations in the LRRK2 Roc-COR tandem domain link Parkinson's disease to Wnt signalling pathways

Fis1 and Bap31 bridge the mitochondria-ER interface to establish a platform for apoptosis induction

Autophagosomes form at ER-mitochondria contact sites

Leucine-rich repeat kinase 2 regulates tau phosphorylation through direct activation of glycogen synthase kinase-3β

An ER-mitochondria tethering complex revealed by a synthetic biology screen

MAM: more than just a housekeeper

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3151117

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10.1016/J.TINS.2016.01.008

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Patricia Gomez-Suaga

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2016-03-01T00:00:00Z

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295102839

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26899735

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neurodegeneration

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