The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
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The C. elegans VAPB homolog VPR-1 is a permissive signal for gonad development.α-Synuclein binds to the ER-mitochondria tethering protein VAPB to disrupt Ca2+ homeostasis and mitochondrial ATP productionComparison of independent screens on differentially vulnerable motor neurons reveals alpha-synuclein as a common modifier in motor neuron diseases.ER-plasma membrane contact sites contribute to autophagosome biogenesis by regulation of local PI3P synthesis.Autophagy as an Emerging Common Pathomechanism in Inherited Peripheral Neuropathies.Peroxisomal ACBD4 interacts with VAPB and promotes ER-peroxisome associations.Ca2+-associated triphasic pH changes in mitochondria during brown adipocyte activation.Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate.Oxidative stress and cellular pathologies in Parkinson's disease.Is amyotrophic lateral sclerosis/frontotemporal dementia an autophagy disease?Traditional Chinese medicine Ka-Sai-Ping suppresses the growths of gastric cancers via induction of autophagy.Protein Localization at Mitochondria-ER Contact Sites in Basal and Stress Conditions.SPLICS: a split green fluorescent protein-based contact site sensor for narrow and wide heterotypic organelle juxtaposition.Proteinopathies and OXPHOS dysfunction in neurodegenerative diseases.Lipids at membrane contact sites: cell signaling and ion transport.INF2-mediated actin polymerization at the ER stimulates mitochondrial calcium uptake, inner membrane constriction, and division.Mutations in glycyl-tRNA-synthetase impair mitochondrial metabolism in neurons.Autophagy as a common pathway in amyotrophic lateral sclerosis.Wolfram syndrome: MAMs' connection?Disruption of ER-mitochondria signalling in fronto-temporal dementia and related amyotrophic lateral sclerosis.Endoplasmic reticulum and mitochondria in diseases of motor and sensory neurons: a broken relationship?ER-mitochondria signaling in Parkinson's disease.The role of endoplasmic reticulum-mitochondria contact sites in the control of glucose homeostasis: an update.Identification of MOSPD2, a novel scaffold for endoplasmic reticulum membrane contact sites.Effectiveness of EGFR/HER2-targeted drugs is influenced by the downstream interaction shifts of PTPIP51 in HER2-amplified breast cancer cellsGlucocorticoid-mediated ER-mitochondria contacts reduce AMPA receptor and mitochondria trafficking into cell terminus via microtubule destabilization
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P2860
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
@en
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
@nl
type
label
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
@en
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
@nl
prefLabel
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
@en
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy.
@nl
P2860
P50
P1433
P1476
The ER-Mitochondria Tethering Complex VAPB-PTPIP51 Regulates Autophagy
@en
P2093
Radu Stoica
Sebastien Paillusson
P2860
P304
P356
10.1016/J.CUB.2016.12.038
P407
P577
2017-01-26T00:00:00Z