Cancer metabolism and elevated O-GlcNAc in oncogenic signaling - Test2 - elhamkhani - TriplyDB

Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Metabolic, autophagic, and mitophagic activities in cancer initiation and progression Glycomic Approaches for the Discovery of Targets in Gastrointestinal Cancer Hallmarks of glycosylation in cancer Three Decades of Research on O-GlcNAcylation - A Major Nutrient Sensor That Regulates Signaling, Transcription and Cellular Metabolism A critical perspective of the diverse roles of O-GlcNAc transferase in chromatin Synthesis and biological evaluation of non-glucose glycoconjugated N-hydroyxindole class LDH inhibitors as anticancer agents.Mixed Lineage Leukemia 5 (MLL5) Protein Stability Is Cooperatively Regulated by O-GlcNac Transferase (OGT) and Ubiquitin Specific Protease 7 (USP7).E2F1 Transcription Factor Regulates O-linked N-acetylglucosamine (O-GlcNAc) Transferase and O-GlcNAcase Expression.Influenza A Virus-Induced Expression of a GalNAc Transferase, GALNT3, via MicroRNAs Is Required for Enhanced Viral Replication.Functional Implications of O-GlcNAcylation-dependent Phosphorylation at a Proximal Site on Keratin 18.Metabolism and epigenetics.Elevated O-GlcNAcylation promotes gastric cancer cells proliferation by modulating cell cycle related proteins and ERK 1/2 signaling.Targeting the hexosamine biosynthetic pathway and O-linked N-acetylglucosamine cycling for therapeutic and imaging capabilities in diffuse large B-cell lymphoma.Glycosylation of KEAP1 links nutrient sensing to redox stress signaling.Epithelial Mesenchymal Transition Induces Aberrant Glycosylation through Hexosamine Biosynthetic Pathway Activation.O-GlcNAcylation of the human epidermal growth factor receptor.O-GlcNAcylation and chromatin remodeling in mammals: an up-to-date overview.Hyper-O-GlcNAcylation induces cisplatin resistance via regulation of p53 and c-Myc in human lung carcinoma.Glycans as Regulatory Elements of the Insulin/IGF System: Impact in Cancer Progression.Interplay between protein glycosylation pathways in Alzheimer's disease.Glucose- and glutamine-fueled stabilization of C-Myc is required for T-cell proliferation and malignant transformation A mutant O-GlcNAcase as a probe to reveal global dynamics of protein O-GlcNAcylation during Drosophila embryonic development.Minireview series on the thirtieth anniversary of research on O-GlcNAcylation of nuclear and cytoplasmic proteins: Nutrient regulation of cellular metabolism and physiology by O-GlcNAcylation.Quantitative time-resolved chemoproteomics reveals that stable O-GlcNAc regulates box C/D snoRNP biogenesis.Inhibition of O-GlcNAcase sensitizes apoptosis and reverses bortezomib resistance in mantle cell lymphoma through modification of truncated Bid.Hyper-O-GlcNAcylation activates nuclear factor κ-light-chain-enhancer of activated B cells (NF-κB) signaling through interplay with phosphorylation and acetylation.Mitochondrial O-GlcNAc Transferase (mOGT) Regulates Mitochondrial Structure, Function, and Survival in HeLa Cells.A Conserved Splicing Silencer Dynamically Regulates O-GlcNAc Transferase Intron Retention and O-GlcNAc Homeostasis.Structures of human O-GlcNAcase and its complexes reveal a new substrate recognition mode.O-linked N-acetylglucosamine transferase enhances secretory clusterin expression via liver X receptors and sterol response element binding protein regulation in cervical cancer.Hyperglycemia and aberrant O-GlcNAcylation: contributions to tumor progression.O-Linked N-Acetylglucosamine (O-GlcNAc) Expression Levels Epigenetically Regulate Colon Cancer Tumorigenesis by Affecting the Cancer Stem Cell Compartment via Modulating Expression of Transcriptional Factor MYBL1.O-GlcNAc elevation through activation of the hexosamine biosynthetic pathway enhances cancer cell chemoresistance.The Nutrient-Sensing Hexosamine Biosynthetic Pathway as the Hub of Cancer Metabolic Rewiring.Hexosamine Biosynthetic Pathway Inhibition Leads to AML Cell Differentiation and Cell Death O-GlcNAc: A Sweetheart of the Cell Cycle and DNA Damage Response Chemical Reporters and Their Bioorthogonal Reactions for Labeling Protein -GlcNAcylation O-GlcNAcylation on Rab3A attenuates its effects on mitochondrial oxidative phosphorylation and metastasis in hepatocellular carcinoma O-GlcNAc as an Integrator of Signaling Pathways

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

http://www.wikidata.org/entity/Q26859087

description

2014 nî lūn-bûn

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2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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JBC.R114.577718

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Journal of Biological Chemistry

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Cancer metabolism and elevated O-GlcNAc in oncogenic signaling

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Keith Vosseller

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Zhiyuan Ma

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Hallmarks of Cancer: The Next Generation

Phosphofructokinase 1 glycosylation regulates cell growth and metabolism

TET2 promotes histone O-GlcNAcylation during gene transcription

TET2 and TET3 regulate GlcNAcylation and H3K4 methylation through OGT and SET1/COMPASS

Stabilization of snail by NF-kappaB is required for inflammation-induced cell migration and invasion

The glucose-responsive transcription factor ChREBP contributes to glucose-dependent anabolic synthesis and cell proliferation

Epithelial-mesenchymal transitions in development and disease

On the Origin of Cancer Cells

Adapting proteostasis for disease intervention

Matrix metalloproteinase-9 triggers the angiogenic switch during carcinogenesis

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scholarly article

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10.1074/JBC.R114.577718

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50

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2014-12-12T00:00:00Z

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