Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis - Test2 - elhamkhani - TriplyDB

Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis

Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis

http://www.wikidata.org/entity/Q27478369

about

Caspase-12 controls West Nile virus infection via the viral RNA receptor RIG-I.Neurotropic virus infections as the cause of immediate and delayed neuropathology Apoptosis, autophagy and unfolded protein response pathways in Arbovirus replication and pathogenesis The Interferon-Stimulated Gene Ifi27l2a Restricts West Nile Virus Infection and Pathogenesis in a Cell-Type- and Region-Specific Manner Inhibition of apoptosis prevents West Nile virus induced cell death Cell-Specific IRF-3 Responses Protect against West Nile Virus Infection by Interferon-Dependent and -Independent Mechanisms West Nile Virus Infection Activates the Unfolded Protein Response, Leading to CHOP Induction and Apoptosis Identification of Novel Small-Molecule Inhibitors of West Nile Virus Infection Differential Effects of Mutations in NS4B on West Nile Virus Replication and Inhibition of Interferon Signaling In vitro effects of selenium deficiency on West Nile virus replication and cytopathogenicity Interferon Regulatory Factor IRF-7 Induces the Antiviral Alpha Interferon Response and Protects against Lethal West Nile Virus Infection West Nile virus–induced acute flaccid paralysis is prevented by monoclonal antibody treatment when administered after infection of spinal cord neurons The structural immunology of antibody protection against West Nile virus Persistent West Nile Virus Associated with a Neurological Sequela in Hamsters Identified by Motor Unit Number Estimation The Immune Adaptor Molecule SARM Modulates Tumor Necrosis Factor Alpha Production and Microglia Activation in the Brainstem and Restricts West Nile Virus Pathogenesis Progress on the development of therapeutics against West Nile virus Apoptosis in animal models of virus-induced disease Complement Protein C1q Reduces the Stoichiometric Threshold for Antibody-Mediated Neutralization of West Nile Virus Arbovirus Infections A viral RNA structural element alters host recognition of nonself RNA Neurological suppression of diaphragm electromyographs in hamsters infected with West Nile virus.Independent regulation of reovirus membrane penetration and apoptosis by the mu1 phi domain A complement-microglial axis drives synapse loss during virus-induced memory impairment.North American encephalitic arboviruses TNF-alpha-dependent regulation of CXCR3 expression modulates neuronal survival during West Nile virus encephalitis.Exposure to the viral by-product dsRNA or Coxsackievirus B5 triggers pancreatic beta cell apoptosis via a Bim / Mcl-1 imbalance.2'-O methylation of the viral mRNA cap by West Nile virus evades ifit1-dependent and -independent mechanisms of host restriction in vivo Pro-inflammatory cytokines derived from West Nile virus (WNV)-infected SK-N-SH cells mediate neuroinflammatory markers and neuronal death Tis21 is required for adult neurogenesis in the subventricular zone and for olfactory behavior regulating cyclins, BMP4, Hes1/5 and Ids.Regulation of cell survival and death during Flavivirus infections.A CRISPR-Based Screen Identifies Genes Essential for West-Nile-Virus-Induced Cell Death.Caspase-3 activation is required for reovirus-induced encephalitis in vivo.Autoimmunity-related demyelination in infection by Japanese encephalitis virus A brain slice culture model of viral encephalitis reveals an innate CNS cytokine response profile and the therapeutic potential of caspase inhibition.West Nile virus preferentially transports along motor neuron axons after sciatic nerve injection of hamsters.Comparison of the neuropathology induced by two West Nile virus strains.The IRF-3/Bax-mediated apoptotic pathway, activated by viral cytoplasmic RNA and DNA, inhibits virus replication.West Nile virus: immunity and pathogenesis.Induction of the cellular microRNA, Hs_154, by West Nile virus contributes to virus-mediated apoptosis through repression of antiapoptotic factors.Immune clearance of attenuated rabies virus results in neuronal survival with altered gene expression.

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Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis

http://www.wikidata.org/entity/Q27478369

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Journal of Virology

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Caspase 3-Dependent Cell Death ...... f West Nile Virus Encephalitis

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Melanie A Samuel

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Protection against fatal Sindbis virus encephalitis by beclin, a novel Bcl-2-interacting protein

Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Specific involvement of caspases in the differentiation of monocytes into macrophages

Acute flaccid paralysis and West Nile virus infection

Long-term prognosis for clinical West Nile virus infection

Japanese encephalitis virus infection initiates endoplasmic reticulum stress and an unfolded protein response

Apoptosis in the mouse central nervous system in response to infection with mouse-neurovirulent dengue viruses

Antiapoptotic but not antiviral function of human bcl-2 assists establishment of Japanese encephalitis virus persistence in cultured cells

Flavivirus Activates Phosphatidylinositol 3-Kinase Signaling To Block Caspase-Dependent Apoptotic Cell Death at the Early Stage of Virus Infection

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2614-23

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scholarly article

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988160

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10.1128/JVI.02311-06

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6

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81

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Michael S. Diamond

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2007-03-01T00:00:00Z

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17192305

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West Nile virus

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1866006

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