A genomics-based screen for yeast mutants with an altered recombination/end-joining repair ratio.
about
Modes of interaction among yeast Nej1, Lif1 and Dnl4 proteins and comparison to human XLF, XRCC4 and Lig4Inhibition of proteasomal degradation of rpn4 impairs nonhomologous end-joining repair of DNA double-strand breaks.Nej1 recruits the Srs2 helicase to DNA double-strand breaks and supports repair by a single-strand annealing-like mechanism.DOA1/UFD3 plays a role in sorting ubiquitinated membrane proteins into multivesicular bodiesA genome-wide RNAi screen reveals multiple regulators of caspase activationMultiple-pathway analysis of double-strand break repair mutations in DrosophilaThe biological functions of Naa10 - From amino-terminal acetylation to human diseaseFunctional genomic analysis reveals overlapping and distinct features of chronologically long-lived yeast populationsMethylated H3K4, a transcription-associated histone modification, is involved in the DNA damage response pathway.Srs2: the "Odd-Job Man" in DNA repair.Release of extraction-resistant mRNA in stationary phase Saccharomyces cerevisiae produces a massive increase in transcript abundance in response to stress.Differential usage of alternative pathways of double-strand break repair in Drosophila.Simultaneous screening and validation of effective zinc finger nucleases in yeastSaccharomyces cerevisiae DNA ligase IV supports imprecise end joining independently of its catalytic activity.Yeast DNA ligase IV mutations reveal a nonhomologous end joining function of BRCT1 distinct from XRCC4/Lif1 binding.Spindle Checkpoint Factors Bub1 and Bub2 Promote DNA Double-Strand Break Repair by Nonhomologous End JoiningMating-type genes and MAT switching in Saccharomyces cerevisiae.Identification of pathways controlling DNA damage induced mutation in Saccharomyces cerevisiae.Pro-recombination Role of Srs2 Protein Requires SUMO (Small Ubiquitin-like Modifier) but Is Independent of PCNA (Proliferating Cell Nuclear Antigen) Interaction.Lif1 SUMOylation and its role in non-homologous end-joining.MLH1 deficiency enhances tumor cell sensitivity to ganciclovir.Yeast Mre11 and Rad1 proteins define a Ku-independent mechanism to repair double-strand breaks lacking overlapping end sequences.Microhomology-dependent end joining and repair of transposon-induced DNA hairpins by host factors in Saccharomyces cerevisiae.Mutations of the Yku80 C terminus and Xrs2 FHA domain specifically block yeast nonhomologous end joiningThe non-homologous end-joining protein Nej1p is a target of the DNA damage checkpoint
P2860
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P2860
A genomics-based screen for yeast mutants with an altered recombination/end-joining repair ratio.
description
2002 nî lūn-bûn
@nan
2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@ast
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@en
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@nl
type
label
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@ast
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@en
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@nl
prefLabel
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@ast
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@en
A genomics-based screen for ye ...... tion/end-joining repair ratio.
@nl
P2860
P1433
P1476
A genomics-based screen for ye ...... ation/end-joining repair ratio
@en
P2093
Thomas E Wilson
P2860
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P407
P577
2002-10-01T00:00:00Z