Arsenic as an endocrine disruptor: arsenic disrupts retinoic acid receptor-and thyroid hormone receptor-mediated gene regulation and thyroid hormone-mediated amphibian tail metamorphosis - Test2 - elhamkhani - TriplyDB

Arsenic as an endocrine disruptor: arsenic disrupts retinoic acid receptor-and thyroid hormone receptor-mediated gene regulation and thyroid hormone-mediated amphibian tail metamorphosis

Arsenic as an endocrine disruptor: arsenic disrupts retinoic acid receptor-and thyroid hormone receptor-mediated gene regulation and thyroid hormone-mediated amphibian tail metamorphosis

http://www.wikidata.org/entity/Q28383459

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Environmental chemicals and type 2 diabetes: an updated systematic review of the epidemiologic evidence EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Role of low exposure to metals as male reproductive toxicants Effects of low-dose drinking water arsenic on mouse fetal and postnatal growth and development Permeation of roxarsone and its metabolites increases caco-2 cell proliferation A dose-response study of arsenic exposure and markers of oxidative damage in Bangladesh Environmental exposure to arsenic may reduce human semen quality: associations derived from a Chinese cross-sectional study Ranking transitive chemical-disease inferences using local network topology in the comparative toxicogenomics database Sex-specific associations of arsenic exposure with global DNA methylation and hydroxymethylation in leukocytes: results from two studies in Bangladesh Urinary metabolomics revealed arsenic internal dose-related metabolic alterations: a proof-of-concept study in a Chinese male cohort Association between arsenic exposure and thyroid function: data from NHANES 2007-2010.The Comparative Toxicogenomics Database facilitates identification and understanding of chemical-gene-disease associations: arsenic as a case study.Disruption of histone modification and CARM1 recruitment by arsenic represses transcription at glucocorticoid receptor-regulated promoters.Exposure to multiple metals from groundwater-a global crisis: geology, climate change, health effects, testing, and mitigation.The broad scope of health effects from chronic arsenic exposure: update on a worldwide public health problem.In utero exposure to low dose arsenic via drinking water impairs early life lung mechanics in mice Maternal arsenic exposure and DNA damage biomarkers, and the associations with birth outcomes in a general population from Taiwan Subchronic Exposure to Arsenic Represses the TH/TRβ1-CaMK IV Signaling Pathway in Mouse Cerebellum Prenatal arsenic exposure and drowning among children in Bangladesh.Protective effects of Vernonia amygdalina against sodium arsenite-induced genotoxicity in rat Chronic exposure to arsenic in the drinking water alters the expression of immune response genes in mouse lung.Multiple metals predict prolactin and thyrotropin (TSH) levels in men.The effects of metals as endocrine disruptors.Integrated approach to the mechanisms of thyroid toxins: electron transfer, reactive oxygen species, oxidative stress, cell signaling, receptors, and antioxidants.Genetic and epigenetic effects of environmental arsenicals.Perspectives in endocrine toxicity of heavy metals--a review.Effects of Combined Exposure to Chronic High-Fat Diet and Arsenic on Thyroid Function and Lipid Profile in Male Mouse.Monomethylated trivalent arsenic species disrupt steroid receptor interactions with their DNA response elements at non-cytotoxic cellular concentrations.Inorganic arsenite inhibits IgE receptor-mediated degranulation of mast cells.Exposure to arsenic in urban and rural areas and effects on thyroid hormones.Serum metabolomics reveals that arsenic exposure disrupted lipid and amino acid metabolism in rats: a step forward in understanding chronic arsenic toxicity.Blood biochemistry, thyroid hormones, and oxidant/antioxidant status of guinea pigs challenged with sodium arsenite or arsenic trioxide.Acute arsenic toxicity alters cytochrome P450 and soluble epoxide hydrolase and their associated arachidonic acid metabolism in C57Bl/6 mouse heart.Poultry litter incineration as a source of energy: reviewing the potential for impacts on environmental health and justice.GSTO2*N142D gene polymorphism associated with hypothyroidism in Italian patients.Mortality of centrarchid fishes in the Potomac drainage: survey results and overview of potential contributing factors.Arsenic(+3) and DNA methyltransferases, and arsenic speciation in tadpole and frog life stages of western clawed frogs (Silurana tropicalis) exposed to arsenate.Scientific report of the Endocrine Active Substances Task Force Concentration of Thyrotropic Hormone in Persons Occupationally Exposed to Lead, Cadmium and Arsenic.Thyroid hormone related gene transcription in southern sand flathead (Platycephalus bassensis) is associated with environmental mercury and arsenic exposure.

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Arsenic as an endocrine disruptor: arsenic disrupts retinoic acid receptor-and thyroid hormone receptor-mediated gene regulation and thyroid hormone-mediated amphibian tail metamorphosis

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Athena P Nomikos

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Cavus Batki

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Jean P Lariviere

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Jenna R Sherman

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Jennifer C Davey

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Joshua W Hamilton

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Manida Wungjiranirun

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P2860

Arsenic exposure is associated with decreased DNA repair in vitro and in individuals exposed to drinking water arsenic

cDNA cloning of human retinoic acid-metabolizing enzyme (hP450RAI) identifies a novel family of cytochromes P450

Cancer risks from arsenic in drinking water

Arsenic alters the function of the glucocorticoid receptor as a transcription factor

Genomic and proteomic profiling of responses to toxic metals in human lung cells.

Water arsenic exposure and children's intellectual function in Araihazar, Bangladesh

Global gene expression associated with hepatocarcinogenesis in adult male mice induced by in utero arsenic exposure.

Cytochrome P450RAI(CYP26) promoter: a distinct composite retinoic acid response element underlies the complex regulation of retinoic acid metabolism

Minireview: nuclear receptor coactivators--an update

The retinoic acid-metabolizing enzyme, CYP26A1, is essential for normal hindbrain patterning, vertebral identity, and development of posterior structures.

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