Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication - Test2 - elhamkhani - TriplyDB

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

http://www.wikidata.org/entity/Q28474967

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The Role of Gammaherpesviruses in Cancer Pathogenesis Emerging Roles of Protein Deamidation in Innate Immune Signaling Interplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the Host Recent advances on viral manipulation of NF-κB signaling pathway Herpesviral G protein-coupled receptors activate NFAT to induce tumor formation via inhibiting the SERCA calcium ATPase An Internally Translated MAVS Variant Exposes Its Amino-terminal TRAF-Binding Motifs to Deregulate Interferon Induction Modulation of NF-κB signalling by microbial pathogens The gammaherpesviruses Kaposi's sarcoma-associated herpesvirus and murine gammaherpesvirus 68 modulate the Toll-like receptor-induced proinflammatory cytokine response.Dissecting innate immune signaling in viral evasion of cytokine production.Murine gamma herpesvirus 68 hijacks MAVS and IKKβ to abrogate NFκB activation and antiviral cytokine production.Interplay between Kaposi's sarcoma-associated herpesvirus and the innate immune system.The membrane-bound transcription factor CREB3L1 is activated in response to virus infection to inhibit proliferation of virus-infected cells.Dissecting host-virus interaction in lytic replication of a model herpesvirus.Murine gammaherpesvirus 68 evades host cytokine production via replication transactivator-induced RelA degradation Infection-Induced Retrotransposon-Derived Noncoding RNAs Enhance Herpesviral Gene Expression via the NF-κB Pathway TLR-TRIF pathway enhances the expression of KSHV replication and transcription activator.IκB Kinase ε Is an NFATc1 Kinase that Inhibits T Cell Immune Response Mitochondrion: an emerging platform critical for host antiviral signaling.Evasion of adaptive and innate immune response mechanisms by γ-herpesviruses.RTA Occupancy of the Origin of Lytic Replication during Murine Gammaherpesvirus 68 Reactivation from B Cell Latency.Viral pseudo-enzymes activate RIG-I via deamidation to evade cytokine production.NF-κB activation coordinated by IKKβ and IKKε enables latent infection of Kaposi's sarcoma-associated herpesvirus.Endoplasmic reticulum stress causes EBV lytic replication.NF-κB activation by equine arteritis virus is MyD88 dependent and promotes viral replication.Genome-wide mapping of infection-induced SINE RNAs reveals a role in selective mRNA export.A Viral Deamidase Targets the Helicase Domain of RIG-I to Block RNA-Induced Activation.Host noncoding retrotransposons induced by DNA viruses: a SINE of infection?Recognizing the SINEs of Infection: Regulation of Retrotransposon Expression and Modulation of Host Cell Processes.Viral FGARAT ORF75A promotes early events in lytic infection and gammaherpesvirus pathogenesis in mice.Inhibition of murine herpesvirus-68 replication by IFN-gamma in macrophages is counteracted by the induction of SOCS1 expression

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Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

http://www.wikidata.org/entity/Q28474967

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@ast

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

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Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@nl

type

label

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@ast

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@en

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@nl

prefLabel

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@ast

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@en

Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

@nl

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JOURNAL.PPAT.1001001

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Murine gamma-herpesvirus 68 hijacks MAVS and IKKbeta to initiate lytic replication

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Haiyan Li

http://www.w3.org/2001/XMLSchema#string

Hao Feng

http://www.w3.org/2001/XMLSchema#string

Pinghui Feng

http://www.w3.org/2001/XMLSchema#string

Qinmiao Sun

http://www.w3.org/2001/XMLSchema#string

Ren Sun

http://www.w3.org/2001/XMLSchema#string

Scott A Tibbetts

http://www.w3.org/2001/XMLSchema#string

Ting-Ting Wu

http://www.w3.org/2001/XMLSchema#string

Xiaonan Dong

http://www.w3.org/2001/XMLSchema#string

P2860

Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity

IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction

IKK-1 and IKK-2: cytokine-activated IkappaB kinases essential for NF-kappaB activation

Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

Innate immune recognition of viral infection

Complete sequence and genomic analysis of murine gammaherpesvirus 68

Disruption of innate immunity due to mitochondrial targeting of a picornaviral protease precursor

An Atomic Model of the Interferon-β Enhanceosome

RIG-I-mediated antiviral responses to single-stranded RNA bearing 5'-phosphates

Pathogen recognition and innate immunity

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e1001001

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scholarly article

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4036911

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10.1371/JOURNAL.PPAT.1001001

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20686657

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2912392

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