Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes - Test2 - elhamkhani - TriplyDB

Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes

Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes

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Mechanisms of Origin, Phenotypic Effects and Diagnostic Implications of Complex Chromosome Rearrangements Barcode Sequencing Screen Identifies SUB1 as a Regulator of Yeast Pheromone Inducible Genes APOBEC3A and APOBEC3B Preferentially Deaminate the Lagging Strand Template during DNA Replication Yeast Sub1 and human PC4 are G-quadruplex binding proteins that suppress genome instability at co-transcriptionally formed G4 DNA Activation induced deaminase mutational signature overlaps with CpG methylation sites in follicular lymphoma and other cancers.Mutational Strand Asymmetries in Cancer Genomes Reveal Mechanisms of DNA Damage and Repair.Strand-biased cytosine deamination at the replication fork causes cytosine to thymine mutations in Escherichia coli.MicroRNA-101 regulated transcriptional modulator SUB1 plays a role in prostate cancer.APOBEC3B cytidine deaminase targets the non-transcribed strand of tRNA genes in yeast.Functions and Malfunctions of Mammalian DNA-Cytosine Deaminases Sub1/PC4, a multifaceted factor: from transcription to genome stability.Mutational Signatures in Breast Cancer: The Problem at the DNA Level Heat shock proteins stimulate APOBEC-3-mediated cytidine deamination in the hepatitis B virus.Avoidance of APOBEC3B-induced mutation by error-free lesion bypass Living Organisms Author Their Read-Write Genomes in Evolution.DNA polymerase η mutational signatures are found in a variety of different types of cancer.Enzyme cycling contributes to efficient induction of genome mutagenesis by the cytidine deaminase APOBEC3B.Sub1 and RNAPII, until termination does them part.Recombination Is Responsible for the Increased Recovery of Drug-Resistant Mutants with Hypermutated Genomes in Resting Yeast Diploids Expressing APOBEC Deaminases.Transcribing malignancy: transcription-associated genomic instability in cancer.Mutational signatures and mutable motifs in cancer genomes.

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Disruption of Transcriptional Coactivator Sub1 Leads to Genome-Wide Re-distribution of Clustered Mutations Induced by APOBEC in Active Yeast Genes

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2015年の論文

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Disruption of Transcriptional ...... y APOBEC in Active Yeast Genes

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Disruption of Transcriptional ...... y APOBEC in Active Yeast Genes

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Disruption of Transcriptional ...... y APOBEC in Active Yeast Genes

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Disruption of Transcriptional ...... y APOBEC in Active Yeast Genes

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Alexey E Masharsky

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Alok Dhar

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Artem G Lada

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Dmitrii E Polev

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Youri I Pavlov

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P2860

AID/APOBEC cytosine deaminase induces genome-wide kataegis

The multiple personalities of Watson and Crick strands

Cancer as an evolutionary and ecological process

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Mutational heterogeneity in cancer and the search for new cancer-associated genes

Mutational processes molding the genomes of 21 breast cancers

The transcriptional landscape of the yeast genome defined by RNA sequencing

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10.1371/JOURNAL.PGEN.1005217

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Igor B. Rogozin

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